餐后血糖

  • 网络Postprandial blood glucose;postprandial glucose;PBG;ppg
餐后血糖餐后血糖
  1. 结论瑞易宁与格华止联用不仅能显著降低空腹和餐后血糖,而且能更有效地降低胰岛素抵抗,改善和保护患者自身β细胞功能。

    Conclusion The combination of glipizide-XL with glucophage can lower FPG and PPG , and reduce insulin resistance and improve B-cell function effectively .

  2. 结论:2型糖尿病患者常有认知功能障碍表现,该认知功能障碍特征与患者的病程和餐后血糖浓度相关。

    CONCLUSION : Type 2 diabetic patients often have cognitive dysfunction , and their cognitive dysfunction is related with their disease course and PBG .

  3. 优泌乐与优泌林R对糖尿病患者餐后血糖波动的影响

    The Impact of Humalog and Humulin R on the Blood Glucose Fluctuation in Diabetic Patients after Meal

  4. Logistic回归分析显示:脉压增大与餐后血糖显著相关,非勺型血压与胰岛素抵抗(IR)显著相关。

    The Logistic analysis showed significant correlation between increased pulse pressure and postprandial hyperglycemia , and between non-dipper hypertension and IR .

  5. 糯米、米粉对NIDDM病人和健康人餐后血糖的影响

    Postprandial response after ingestion of glutinous rice and ground rice by healthy and NIDDM subjects

  6. 血糖生成指数(GI)是反映碳水化合物餐后血糖应答水平的特征性指数。

    Glycemic index ( GI ) is an item coined to character the blood glucose response of carbohydrate .

  7. 结论低GI饮食针对餐后血糖应答,是一种灵活而有效的治疗方法。

    ( P0.01 ) Conclusion Low-GI diet for postprandial glucose response , is a flexible and effective treatment methods .

  8. 结论那格列奈可有效降低IGT个体的餐后血糖,安全性和耐受性好。

    Conclusion Nateglinide was well tolerated , safe and effective in reducing postprandial hyperglycemia in subjects with IGT .

  9. 结论IGT患者的DBP降低,脉压增大与餐后血糖水平相关。

    Conclusion The decrease in DBP and increase in PP are related to the postprandial hyperglycemia in group IGT .

  10. 目的:探讨4种干豆(白扁豆、眉豆、绿豆和赤小豆)对非胰岛素依赖型糖尿病(NIDDM)病人餐后血糖和血清C肽的影响。

    Objective : To evaluate the postprandial responses of plasma glucose and serum C peptide to four dry legumes in non insulin dependent diabetes mellitus ( NIDDM ) subjects .

  11. 骨密度与餐后血糖、HbA1C、尿糖呈负相关。

    The bone mineral density was negative correlation with postprandial blood glucose , HBA1C and harnzucker .

  12. WHO组有较高的发病年龄、BMI偏低、显著升高的高胆固醇血症和显著升高的空腹和餐后血糖。

    Meanwhile , subjects in the WHO group was characterized by lower BMI , higher hyperlipidemia , and significantly increased fasting and postprandial blood glucose level .

  13. 主要的疗效参数是阿卡波糖治疗后空腹和餐后血糖水平的变化以及HbA(IC)水平的变化。

    Main efficacy parameters were the changes in fasting and postprandial blood glucose concentrations as well as in HbA - ( 1C ) levels after acarbose treatment .

  14. 结论:苦芪方治疗糖尿病肾病有较好的临床疗效,并具有减少24h尿蛋白定量、降低尿自蛋白排泄率及改善餐后血糖和脂质代谢等作用。

    Conclusion : KF is effective for DN and can decrease 24h quantitative urinary protein , UAER and improve the blood glucose after meal and metabolism of lipid .

  15. 完善检查(标准餐后血糖、胰岛素、促胃肽、胃泌素,血脂、HbAlc、超氧化物歧化酶(SOD))。

    Finish examination ( blood glucose , insulin , ghrelin , gastrin level after standard meal , serum lipids , HbAlc and superoxide dismutase ( SOD ) .

  16. 结论预混门冬氨酸胰岛素30/70降餐后血糖和HbA1C明显优于精蛋白生物合成人胰岛素30/70。

    Conclusion Insulin aspart 30 / 70 is better than the biphasic human insulin 30 / 70 in lowering postprandial blood glucose and HbA1c .

  17. 几项有关糖尿病患者的回顾性研究揭示了FPG和餐后血糖到A1C的作用。

    Several previous studies in diabetic patients have shown the contribution of FPG and postprandial blood glucose to A1C ( 15 ) .

  18. GLP-1通过同时作用于肠道、肝脏、胰腺和大脑等多种机制改善餐后血糖。

    GLP-1 improves blood sugar after food intake through multiple effects that work in concert on the stomach , liver , pancreas and brain .

  19. 给药两周后褪黑素组患者血浆空腹血糖、餐后血糖及果糖胺水平均较安慰剂组明显下降,两组间差异具显著意义(P0.05)。

    There were significant differences in the glucose metabolic parameters , such as fasting plasma glucose , postprandial plasma glucose and fructose amine levels between melatonin treated and placebo control group respectively 2 weeks after treatment ( P 0.05 ) .

  20. 结果120mg那格列奈和50mg阿卡波糖降低餐后血糖的效果相似;

    Results The effects of 120 mg nateglinide and 50 mg acarbose on postprandial hyperglycemia in patients with type 2 diabetes were similar .

  21. 因为GLP-1以上的作用,可以降低空腹或餐后血糖,其作用已在健康人体和糖尿病病人的临床研究中得到证实,有很好的治疗糖尿病的临床应用前景。

    Its effects have been proven in the clinical trails with health volunteers and diabetics . GLP-1 may provide an additional approach for the treatment of type II diabetes .

  22. 目的:研究蒺藜皂苷(STT)对大鼠小肠α-葡萄糖苷酶的抑制作用及对餐后血糖水平的影响。

    Objective : To determine the effects of saponins from Tribulus terrestris ( STT ) on small intestinal α - glucosidase and postprandial blood glucose levels in rats .

  23. 研究表明DNJ具有通过抑制α-葡萄糖苷酶活性,进而抑制餐后血糖升高、抗病毒、抗肿瘤等多种生物学活性。

    Due to inhibiting α - glucosidase enzyme activity , it can inhibit postprandial blood sugar level and has some biological activities such as anti-virus and anti-tumor .

  24. 结论:STT抑制大鼠餐后血糖水平的升高,是由其对小肠α-葡萄糖苷酶的抑制作用实现的。

    Conclusion : It was through inhibiting the activity of α - glucosidase in small intestines that STT significantly retarded the increase in postprandial blood glucose levels in rats .

  25. HbA1c不能反映餐后血糖的漂移变化,PPGE可作为估测餐后血糖漂移程度的简易临床参数。

    HbA1c can ′ t reflect postprandial glucose excursions . PPGE can be used as a simple clinic index to evaluate the amplitude of postprandial glucose excursions .

  26. 观察移植前后胰岛素用量、空腹血糖(FBS)、餐后血糖(PBS)、糖化血红蛋白(HBA1)及肝肾功能变化。

    Fasting blood glucose ( FBS ), postprandial blood glucose ( PBS ), HBA1 , dose of insulin and function of liver and kidney were measured before and after the transplantation .

  27. 而脑梗死组急性期及恢复期餐后血糖水平均升高,IGT减低,与正常对照组有显著性差异(P<0.05)。

    In acute and convalescence of cerebral infarction group , the blood sugar becomes higher ( P < 0 . 01 ) when patients ' stomachs were on empty and after had meal ( P < 0 . 05 ) .

  28. 无症状组72例,亦按病程分为2个亚组,病程<6mo32例、病程>6mo40例,观察胰岛素强化治疗前后空腹血糖(FPG)及餐后血糖(PPG)、C肽的变化。

    The 72 cases without symptoms were divided into another two subgroups : 32 cases with course less than 6 mo and 40 cases more than 6 mo. Blood glucose and C peptide in fasting and postprandial were observed before and after insulin intensive therapy .

  29. 结果:血管内皮功能异常组的腰围、腰臀比、餐后血糖、空腹胰岛素、餐后胰岛素和HOMA-IR均显著高于血管内皮功能正常组(P<005~001)。

    Results : Waist circumference , waist-hip ratio , postprandial blood glucose , fasting insulin , postprandial insulin and HOMA-IR were significantly higher in impaired endothelial function group than in the normal one ( p < 0.05-0.001 ) .

  30. 结论糖代谢异常的高血压患者其24h血压的昼夜节律紊乱,其中以IGT患者的舒张压降低和脉压增大为著,而脉压增大与餐后血糖及血浆胰岛素水平相关。

    Conclusion The temporal rhythm of 24 h blood pressure of the hypertension patients with glycometabolism abnormality was disturbed , and the debasement of DBP and augmentation of PP in the patients with IGT were significant . PP augmentation correlated with postprandial blood sugar and plasma insulin level .