神经胶质细胞

传统观念认为，神经胶质细胞在信息传递和整合过程中仅仅是一个被动的角色。

Neuroglia cell is traditionally considered to be one passive part in the synaptic transmission .

光镜下肝脏、脾脏有非典型肉芽肿形成，肺多呈间质性肺炎，脑神经胶质细胞增生；

Atypical granulomas appeared in the liver and spleen and interstitial pneumonia and hyperplasia neuroglia cell in cerebrum were observed .

脑组织TNFɑ过度表达对神经胶质细胞及脑缺血梗死灶体积的影响

Effects of overexpression of murine TNF ɑ gene on glia and infarct volume

去甲肾上腺素/烧伤血清诱导大鼠神经胶质细胞VEGF基因表达

VEGF gene expression in norepinephrine / burn serum-induced rat astrocytes

神经胶质细胞命运与Notch信号途径的决定作用

Notch signal pathway in determining glial cell fates

探讨阿魏酸对红藻氨酸诱导的AD小鼠脑神经胶质细胞活化的影响。

To study the effects of FA on the reactive astrocytes in mice cerbral neuronal cells induced by KA . 4 .

吗啡对C6神经胶质细胞瘤P53及Bcl-2蛋白质水平的影响

Effects of morphine on P53 and Bcl-2 protein levels in C6 glioma cells in rats

酒精对鼠胚胎神经胶质细胞c-fos基因表达的影响

The effect of alcohol on c-fos gene expression in rat embryo neuroglial

高压氧（02MPa）暴露第3d，组织间隙水肿消退，神经胶质细胞增生。

When the animals were exposed to HBO ( 0.2 MPa ), their intercerebral edema was absorbed and the glial cells proliferated at the 3rd d.

甲基汞对脑神经胶质细胞凋亡及c-fos表达的影响

Effect of methylmercuric chloride on apoptosis and c-fos expression of brain glial cells

小鼠神经胶质细胞培养上清IL-6水平与脑不对称性的关系

Correlation between the IL-6 levels in supernatants of glial cells of Balb / c mice and brain lateralization

神经胶质细胞在IBS内脏高敏感性中作用的展望

Advance on the study of the Role of Glial Cells in Visceral hyperalgesia of IBS

结论反义c－myc基因质粒可抑制c－myc基因的过度表达，促使肿瘤细胞凋亡，达到治疗脑神经胶质细胞瘤的目的。

Conclusion Antisense c-myc gene plasmid can inhibit the excessive expression of c-myc gene and is helpful to the treatment of glioma .

结论DM在体外可降低神经胶质细胞的存活率并短时升高胞内[Ca2+]i。

Conclusion The cell survival rate was decreased and the I concentration was temporarily increased when astrocytes were treated with DM .

Nestin和GFAP在缺氧大鼠视网膜神经胶质细胞中的表达及高氧治疗的作用

Nestin and GFAP is expressed in rat retina subjected to hypoxia and attenuated by hyperoxia treatment

培养第三天加入终浓度为5mg／ml的阿糖胞苷以抑制神经胶质细胞的过度增殖。

At the 3rd day , in order to suppress the proliferation of glial cells , cytarabine was added .

结果：Ⅱ级以上神经胶质细胞瘤PCNA表达高于Ⅰ级神经胶质细胞瘤（P<0.05）；

Result : The expression of PCNA of glial tumor grade ⅱ was higher than grade ⅰ ( P < 0.05 ) .

结果侧脑室注射AChRAb后，在MG中枢损害模型大鼠海马及大脑皮层可见神经胶质细胞凋亡。

Results The apoptotic glial cells were found in the hippocampus and cerebral cortex after intracerebroventricular administration of AChRAb .

方法：用免疫荧光法观察NE/烧伤血清刺激神经胶质细胞后24hVEGF在细胞内的分布。

METHODS : Immunofluorescence staining was used to show the distribution of VEGF in astrocytes after 24 h using norepinephrine / burn serum stimulation .

结论视网膜色素上皮细胞条件培养液促进视网膜神经胶质细胞增生，细胞间的相互作用对于PVR等的产生和发展是非常重要的。

Conclusion Interaction of RG cells and RPE cells is related to the formation and development of PVR .

结果：转小鼠TNFɑ基因大鼠与SD大鼠相比，未缺血脑组织见TNFɑ表达，且星形胶质细胞、小胶质细胞与少突神经胶质细胞呈肥大和增生性变化；

Results : TNF ɑ overexpression and hypertrophic and proliferative changes of microglia , astrocyte and oligodendrocyte in the non-ischemia brain tissue of murine TNF ɑ transgenic rats were observed .

结论在PVR中，增殖细胞来源于视网膜色素上皮细胞和神经胶质细胞。

Conclusion The proliferative cells originate from retinal pigment epithelial ( RPE ) cells and glial cells in PVR .

随着神经胶质细胞瘤分化程度的降低，肿瘤细胞内tTG表达明显增加；

Expression intensity of tTG in glioma was higher than that in benign brain tumors .

目的：研究特异性siRNA对大鼠神经胶质细胞中GLT-1基因的阻抑效果。

Objective : To study the inhibitory effect of siRNA targeting GLT-1 gene in rats glial cells .

神经胶质细胞活化在AD发生过程中显著增多，表明神经细胞受到毒害，星形胶质细胞出现胶质反应而被激活，促使GFAP大量合成。

These remarkable increased of active astrocytes in AD indicated that when the nerve cells are poison , astrocytes appear glial reaction and activated , prompted GFAP lots of synthesis .

结果VEGF及FIt-1免疫阳性细胞主要在神经元、神经胶质细胞及内皮细胞中表达。

Result VEGF gene and its receptor Flt-1 expressed in the neurons , glial cells and vascular endothelial .

EGF也可以通过支持和诱导神经元胞体及神经胶质细胞间接途径分泌神经生长因子（NGF）来促进神经细胞再生。

EGF can secret NGF to promote the regeneration of neural cells via an indirect route of sustaining and inducing the growth of neurons and glia cells .

在免疫功能正常的宿主，JC病毒主要存在于脑、髓的少数神经胶质细胞的核内。

In immunocompetent hosts , JC virus remain dormant in nuclei of rare scattered glial cells in brain and spinal cord parenchyma .

应用光镜和免疫组化法，观察缺血侧额顶叶皮质神经元、神经胶质细胞形态学变化及TNF-α、c-Myc蛋白表达。

The morphologic changes in neuron or neuroglial cells and expression of TNF - α, c-Myc protein were observed by techniques of immuno-histochemical staining and optical microscopy .

给药后1～8周光镜显示右侧DRG细胞变性、坏死、消失，神经胶质细胞增生。

Light microscopic examination revealed degeneration , necrosis and disappearance of cells and hyperplasia of gliacytes in right DRG at 1-8 weeks after injection .