代谢性酸中毒

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  • metabolic acidosis
代谢性酸中毒代谢性酸中毒
  1. 许多毒性物质可引起代谢性酸中毒。

    A number of intoxicants are associated with metabolic acidosis .

  2. 枸橼酸转运蛋白mRNA在代谢性酸中毒大鼠肾组织的表达

    Expression of citrate transporter mRNA in the kidneys of rats with metabolic acidosis

  3. 目的:探讨代谢性酸中毒对大鼠红细胞碳酸酐酶(CA)活性的影响。

    Objective : To investigate the effect of metabolic acidosis on carbonic anhydrase ( CA ) activity in rat erythrocytes .

  4. 急性代谢性酸中毒时CSFpCO2在CSF酸碱调节中的作用

    Role of CSF pCO_ 2 in CSF acid-base regulation in acute metabolic acidosis

  5. 尽管两个病例都有代谢性酸中毒合并呼吸性碱中毒,但引起的PH值变化程度是不同的。

    Although both cases have a combination of a metabolic acidosis and a respiratory alkalosis , the resultant pH in the two cases is different .

  6. 结果与对照组相比,LPS组出现明显的代谢性酸中毒、高血钾、高血钠、低血钙及高血糖等内环境紊乱状态(P<0.05)。

    Results Metabolic acidosis , hyperkalemia , hypernatremia , hypocalcemia and hyperglycemia were observed in LPS group rats ( P < 0.05 ) .

  7. 结论NICU中新生儿以代谢性酸中毒合并呼吸性酸中毒为最多见。

    Conclusion Metabolic acidosis with respiratory acidosis was most common in neonates in NICU .

  8. 第1次HBO治疗后,HBO组rCAV明显高于对照组(P<005),低氧血症和代谢性酸中毒纠正速度明显快于对照组,(P<005)。

    Hypoxia and metabolic acidosis in HBO group were corrected more rapidly than those in control group ( P < 0.05 ) .

  9. 奶牛在饲喂阴离子盐后处于代谢性酸中毒状态时,尿液pH显著降低,尿液中的钙离子和氯离子排泄量显著增加(P<0.05),其他离子的排泄情况没有显著变化(P>0.05)。

    Metabolism acidosis was induced in the prepartum cow fed anionic salts , which showed significantly low urinary pH ( P < 0.05 ), and high calcium and chlorine excretion in urine ( P < 0.05 ) .

  10. 据报道海水淹溺型肺水肿(pulmonaryedemaofseawaterdrowning,PE-SWD)是海水溺水死亡的主要原因,其主要病理生理学特点为低氧血症、高碳酸血症和代谢性酸中毒。

    According to what was reported the main reason for death of sea-water drowning was pulmonary edema of sea water drowning . Hypoxemia , hypercapnia and metabolic acidosis were the common pathophysiology trait .

  11. 结论CRF代谢性酸中毒抑制血清瘦素的分泌,血清瘦素浓度的高低与营养不良无关。

    Conclusion The metabolic acidosis inhibits the secretion of leptin in CRF patients , and the leptin seems to have no important effects on protein-malnutrition here .

  12. 严重碱血症是致死的主要原因之一。中、重度ARDS病人的酸碱失衡是以呼碱型三重酸碱失衡、呼吸性酸中毒和呼吸性酸中毒并代谢性酸中毒最常见。

    Triple disorder as respiratory alkalosis , respiratory acidosis , and respiratory acidosis combined with metabolic acidosis was very commonly encountered in moderate and severe cases of ARDS .

  13. 代谢性酸中毒犬脑脊液〔HCO-3〕与Pco2的关系

    Relationship between cerebrospinal fluid HCO - 3 concentration and cerebrospinal fluid P CO 2 in acute metabolic acidosis in dog

  14. MOF早期多为呼吸性碱中毒,并发低氧血症,随病情的加剧,可发生代谢性酸中毒,严重者可发生三重酸碱失衡。

    Ourdata showed that in the early stage of MOF respiratory alkalosis with or without hypoxemiamight be present , and later or , there might be metabolic acidosis or even triple acid-base disturbances .

  15. 结果现有条件下能够建立常温大鼠体外循环模型,较高流量(200ml·min~(-1)·kg~(-1))转流1h,转流期间保持存活,但容易出现代谢性酸中毒和动脉血压降低;

    Results It is feasible to establish the normothermic CPB model in SD rats . The rats remained alive during CPB with a high flow rate ( 200ml · min-1 · kg-1 ) .

  16. 结论HIE患儿以高AG性代谢性酸中毒为特点,并易出现混合性酸碱紊乱,重度HIE患儿易致高碳酸血症,处理HIE患儿时应改善通气与循环

    Conclusion HIE is characterized by high AG metabolic acidosis and apt to mixed acid base disturbance . Severe HIE is easy to result in hypercapnia . When HIE patients are treated , the air passage and blood circulation should be improved

  17. 探讨未测定阴离子隙(UAG)在重症感染患者代谢性酸中毒发生中的作用。方法;

    Objective : To study the effect of unaccounted anion gap ( UAG ) in metabolic acidosis during superinfection in humans .

  18. 结果与基础值相比,颈静脉泵入油酸后的10h内:①PaO2和PaCO2均降低,表现为代谢性酸中毒。

    Results Comparing with fundamental values , in 10 hours after injection of oleic acid through jugular vein , those values of PaO_2 and PaCO_2 both decreased , presenting metabolic acidosis .

  19. 结论HBO可明显提高PaO2和SaO2,有效改善低氧血症和代谢性酸中毒,减轻PE-SWD时肺组织的损伤,从而可防止PE-SWD向SW-RDS转化。

    Conclusions HBO could obviously increase PaO 2 and SaO 2 , improve effectively hypoxia and metabolic acidosis , and alleviate pulmonary tissue damage in PE SWD ; therefore , the development of SW RDS might be prevented .

  20. 代谢性酸中毒(MA)是慢性肾衰竭患者常出现的临床表现,可能与蛋白质-热能营养不良(PEM)和炎症有关,后两者合称为营养不良-炎症反应综合征(MICS)。

    Metabolic acidosis ( MA ) is a common clinical manifestation in patients with chronic renal failure ( CRF ) . and may be linked to protein-energy malnutrition ( PEM ) and inflammation , both of which are also known as malnutrition-inflammation complex syndrome ( MICS ) .

  21. 结论:重症感染患者合并代谢性酸中毒时,乳酸的增加不是导致代谢性酸中毒的唯一因素,而UAG的增加在代谢性酸中毒的发生中有重要作用。

    Conclusion : These data indicated that lactic acidosis did not entirely account for the metabolic acidosis during superinfection in humans , bet the increased UAG may play an important role in metabolic acidosis during superinfection .

  22. 支气管哮喘代谢性酸中毒的探讨乳酸菌的抗高血压作用

    METABOLIC ACIDOSIS IN ACUTE ASTHMA Antihypertensive Effect of Lactic Acid Bacteria

  23. 血液学研究显示有严重高磷血症、低钙血症和代谢性酸中毒。

    Hematologic studies revealed severe hyperphosphatemia , hypocalcemia and metabolic acidosis .

  24. 慢性代谢性酸中毒显著诱导大鼠系膜细胞的增殖

    Chronic metabolic acidosis markedly induces proliferation of mesangial cells in rats

  25. 早产儿代谢性酸中毒的临床观察与护理

    Clinical Observation and Nursing Care of Preterm Infants With Metabolic Acidosis

  26. 慢性肾衰竭患者代谢性酸中毒与营养不良-炎症反应综合征

    Metabolic acidosis and malnutrition-inflammation complex syndrome in patients with chronic renal failure

  27. 血糖水平与糖尿病代谢性酸中毒的关系分析

    The Relationship between Blood Glucose Level and Metabolic Acidosis of Diabetes Mellitus

  28. 急性口服甲醇中毒血甲醇浓度与代谢性酸中毒的关系

    The relationship between blood methanol and metabolic acidosis in oral methanol poisoning

  29. 未测定阴离子隙在重症感染患者代谢性酸中毒发生中的作用

    Effect of Unaccounted Anion Gap in Metabolic Acidosis during Superinfection in Humans

  30. 速尿在代谢性酸中毒脑脊液酸碱调节中的作用

    Effects of furosemide on cerebrospinal fluid acid-base regulation in a-cute metabolic acidosis