胎盘异常

胎盘异常85例，占前置胎盘分娩总数的40.86%；

85 patients with abnormal placenta were detected and was 40.86 percent of the total gestational women with placenta praevia .

需行干预性治疗的产后出血的原因依次为：DIC，子宫动-静脉瘘及血管畸形，胎盘异常，子宫肌瘤及子宫裂伤。

The main causes of postpartum hemorrhage requiring operative management were disseminated intravascular coagulation ( DIC ), uterine arteriovenous fistula and malformation , placental abnormality , uterine myoma and laceration .

B-lynch缝合术治疗胎盘异常及凝血功能障碍所致产后出血4例临床分析

Clinical Analysis of Four Cases of B-Lynch Sutrue for treatment of Postpartum Haemorrhage Caused By Abnomal Placenta and Coagulopathy

目的探讨胎盘异常的超声诊断及声像图特征。

Results Anomaly of placenta was detected in31 cases with antepartal ultrasonography , and .

初孕人工流产后再孕胎盘异常67例分析

The Analysis of 67 Cases of Placental Abnormality as Re - pregnancy after Artificial abortion

目的探讨初孕人工流产对再孕胎盘异常的发生情况及影响。

Objective To study the effect of artificial abortion on placental abnormality as re - pregnancy .

孕妇外周血胎儿有核红细胞数与胎儿及胎盘异常关系

Counting of fetal nucleated erythrocytes in maternal blood and its relation with abnormal fetus and placenta

子宫出血的首位原因是各种胎盘异常，其发生与多次流产有关；

The first rea - son of uterine bleeding was some kind of abnormal placenta related to frequent abortion .

目的探讨人工流产后对未生育妇女再次妊娠分娩、胎盘异常的影响。

Objective To evaluate the influence of Induced abortion to the next childbirth and abnormal placenta to a woman who has never bear .

脐带绕颈组S/D值升高，臀位、早产、胎盘异常、羊水过少或粪染组S/D值均高于对照组。

The UmA S / D in patients with umbilical cord coiling , breech presentation , preterm labor , oligoamnios or placental abnormality was higher than that in controls .

结果1.796例孕妇中的不良妊娠结局按发生率顺位排序前五位是早产、胎膜早破、胎盘异常、低体重儿、重度子痫前期。

Among the incidences of the adverse pregnancy outcomes , premature birth , premature rupture of fetal membranes , placental abnormalities , low birth weight children and pre-eclampsia were the top five . 2 .

有人工流产史者21例（40%），胎盘异常12例（22.6%），经产妇45例（84.9%）。

Among them , 21 cases had a history of artificial abortion ( 40 % ), 12 cases were with placental abnormality ( 22.6 % ) and 45 cases were pluripara ( 84.9 % ) .

方法将我院1996年2月至1999年2月间分娩的健康初产妇中，人流的次数及初孕人工流产后距再孕的不同时间发生胎盘异常的情况进行对比分析。

Methods Among the healthy primiparas from Feb. 1996 to Feb. 1999 , the effect of numbers of artificial abortion and the duration from abortion to re - pregnancy on placental abnormality were contrast analysis .

结果显示有流产史的产妇胎盘异常、产后出血及胎儿异常发生牢明显高于无流产史组，人工流产组上述异常的发生率明显高于自然流产组。

Results showed that the incidence rates of those abnormalities were higher with statistical significance in the group with abortion history than in the group without abortion history , and in the cases with a history of induced abortion than in the cases with a history of natural abortion .

Brown和其同事还有加利福尼亚州伯克利奥克兰公共卫生学院皇家研究基金会的同行们指出：高同型半胱氨酸血症与胎盘功能异常以及妊娠合并症都有关系。

Brown of Columbia University , New York and colleagues there and at the Kaiser Foundation Division of Research , Oakland , and the Public Health Institute , Berkeley , in California .

导致PE产生的原因众说纷纭，其中多因素所致滋养细胞功能障碍而引起的胎盘功能异常和血管重铸不足可能是主要原因[1]。

The pathogenic factors leads to PE are divergent , but we currently considered that the reason leads to production of PE perhaps is placental dysfunction and insufficient vascular remodeling , which is the main reason which caused trophoblast dysfunction .

目的和方法：对5例患Budd-Chiari综合征（B-CS）妊娠28～36周孕妇的胎儿胎盘发育异常的超声显像进行研究。

Objective and Methods : : To survey the development of fetus and placenta of 28 36 weeks gestations in 5 patients suffered Budd Chiari syndrome ( B CS ) by duplex Doppler .

结论Rho/Rho激酶信号转导系统对细胞滋养细胞侵袭有重要作用，可能参与由胎盘植入异常引起的妊娠相关疾病。

ConclusionRho / Rho kinase signaling pathway is a key regulator of trophoblast cell migration , involved in placenta implantation .

这些小鼠出现了各种各样的症状，包括严重的高血压，蛋白尿和胎盘的异常。

The mice showed multiple features of the disorder , including dangerously high blood pressure , protein in the urine , and placental abnormalities .

这种相对较高的早期胚胎死亡可能与多因素影响怀孕的复杂性和在克隆胚上经常观察到的胎盘形成异常有关。

This relatively high early embryonic loss may be associated with multiple pregnancy complications or the other abnormalities of placentation frequently observed in cloned embryos .

围产期妊娠并发症.如妊娠期高血压疾病征、妊娠期肝内胆汁淤积症、妊娠期糖尿病会导致胎盘功能异常；

Perinatal complications , like pre-eelampsia ( PE ), intrahepatic cholestasis of pregnancy ( ICP ), gestational diabetes mellitus ( GDM ), may lead to abnormal placental function .

然而目前核移植成功的效率仍然很低，常出现孕期流产、存活率低，存活动物也常伴有不同程度发育缺陷和多种异常表型，并且伴有胎盘的异常发育。

Pregnancy miscarriage and low survival rate usually appear . Survival animals are usually accompanied by various degrees of developmental defects and abnormal phenotypes , and associated with abnormal development of the placenta .

氨基酸是促进胎儿生长发育的重要营养成分之一，胎盘氨基酸转运异常可影响胎儿生长发育。

Amino acids are one of the important nutrients to promote fetal growth and development . Abnormal placental amino acid transporters can affect fetal growth and development .

结果与胎儿宫内生长受限相关的临床因素为重度妊娠高血压综合征、心肺疾病、胎盘及脐带异常、双胎、羊水过少等。

Results The clinical factors of FGR include pregnancy-induced severe hypertension syndrome , heart and lung diseases , serious placenta and cord abnormality , twin pregnancy , oligohydramnios etc.

结果提示：NST是预测胎儿预后的方法之一，如果NST无反应型并伴有胎盘检测结果的异常，将会严重影响围产儿尤其是早产儿的预后。

The result showed that NST is one method of survillances to predict prognosis . If result of NST is NST NR and complicated with other abnormal results of fetal placental testing , the prognosis would be poor especial for premature baby .

本文对部分围产儿死亡的胎盘进行了研究，讨论了胎盘及其附属物异常与围产儿死亡的关系，强调在大力开展围产儿尸检的同时应提高胎盘送检率。

The authors emphasize the relationship between abnormal placenta and its appendages and perinatal death , so that the placenta of perinatal death must be examined in addition to autopsy .

结论妊娠肝内胆汁淤积症患者胎盘PLGF及Flt-1表达下降与胎盘滋养叶细胞侵入异常、胎盘血管形成异常、胎盘血管密度降低有关，可能是ICP胎儿妊娠结局不良的原因。

Conclusion : The lower expression of PLGF and its receptor Flt-1 in placenta from ICP patients is related to the abnormal invasions of trophoblast , the inadequate blood vessel formation and the decrease of placental vascular density , which could lead to fetal unhealthy pregnancy outcome .

结论：正常妊娠妇女胎盘中表达少量的TNF-α和p55TNFR，参与正常妊娠的调节。胎盘中异常增加的TNF-α与p55TNFR可能在妊高征的发病中起重要的作用。

Conclusion TNF - α and p55 TNFR expressions in small scale in normal pregnant women may participate to regulate the normal pregnancy , whereas the over expression of TNF - α and p55 TNFR in placenta may play an important role in the pathogenesis of PIH .

方法：采用改良的血管铸型方法，通过胎盘脐动、静脉灌注血管铸型材料，铸建20例正常胎盘，48例异常胎盘血管模型。

Methods : The models of normal placental vascular system of 20 cases and abnormal placental vascular system of 48 cases were established by using the modified vascular casting method which pours the casting materials into the placental arteries and veins .