恶性胶质瘤

  • 网络glioblastoma;gbm;glioma
恶性胶质瘤恶性胶质瘤
  1. EGFR反义RNA抑制人脑恶性胶质瘤细胞增殖并诱导凋亡的研究

    The Effect of Antisense EGFR RNA on the Proliferation and Apoptosis of Human Glioblastoma Cells

  2. MRI图像显示一环形水肿性病损,常难以与恶性胶质瘤、溃疡以及其它肿瘤(包括转移灶)相鉴别。

    MRI images often show a ring-enhancing lesion surrounded by edema , a picture indistinguishable from glioblastoma , abscess , and other tumors ( including metastasis ) .

  3. 颅内恶性胶质瘤术后早期MR影像学表现

    MR imaging in patients with intracranial postoperative malignant gliomas

  4. 恶性胶质瘤DNA拓扑异构酶的表达

    Expression of DNA topoisomerase in recurrence and primary malignant glioma

  5. 转染TNFα基因诱导恶性胶质瘤细胞凋亡的研究

    Study of Apoptosis of Malignant Glioma Cells Induced by TNF α Gene Transfection

  6. 颅内恶性胶质瘤术后脑组织正常反应与术后残存的动态增强MRI鉴别

    Dynamic enhanced MR imaging in differentiating normal tissue reaction and residual tumor after resection of intracranial malignant gliomas

  7. 分次大剂量照射对人脑恶性胶质瘤细胞BT(325)株的致死效应

    The Lethal Effects Analysis of BT_ ( 325 ) with Fractionated High Dose Irradiation

  8. p53基因增强恶性胶质瘤HSV-TK/ACV系统疗效的体外研究

    Study on Anti-Glioma Effect of Recombinant Adenovirus Mediated HSV-TK / ACV System Enhanced by wt-p53 Gene in vitro

  9. CD(44)V6、MMP-9在脑转移瘤及恶性胶质瘤中的表表及意义

    The Significance and Expression of CD_ ( 44 ) V_6 、 MMP-9 in Brain Metastases and Malignant Glioma Invasion

  10. p53基因优化恶性胶质瘤HSV-TK/GCV治疗的体外研究

    Study of Curative Effect of Recombinant Adenovirus-Mediated HSV-tk-GCV System Optimized by wt-p53 Gene Transfer on malignant gliomas in Vitro

  11. 转染白介素12的神经干细胞免疫基因治疗大鼠C6恶性胶质瘤

    Immunogene therapy of rat malignant gliomas using neural stem cells expressing IL-12

  12. bcl-2、c-myc基因表达在去甲二氢愈创木酸诱导人恶性胶质瘤细胞凋亡中的意义

    Significance of bcl-2 and c-myc gene expression in nordihydroguaiaretic acid-induced apoptosis of a human malignant glioma cell line

  13. 结论结合CT表现与病人年龄、病变部位,全面分析,能提高大脑恶性胶质瘤的诊断准确率。

    Conclusion The correct diagnosis of brain malignant glioma could be established by comprehensive consideration of the CT appearance , the patient ′ s age and the location of tumor .

  14. 针对MAGE-1基因RNAi在恶性胶质瘤SHG-44细胞中的作用研究

    Inhibition of MAGE-1 by RNAi in SHG-44 human glioma cell line

  15. 结论TNFα对恶性胶质瘤细胞具有抑制生长和诱导凋亡作用,而bcl2蛋白表达水平不受影响。

    CONCLUSION TNF α may inhibit the proliferation and induce apoptosis but do not affect bcl 2 expression of malignant glioma cells .

  16. 还发现溶解性HK活性的比例依正常脑、恶性胶质瘤、良性胶质瘤和脑膜瘤顺次升高。

    And the proportion of soluble HK activity in the normal brains , malignant gliomas , benign gliomas and meningiomas was in ascending order of level .

  17. 人参皂甙Rh2对人脑恶性胶质瘤SHG-44细胞抗肿瘤作用的蛋白质组学分析

    Proteomic analysis of ginsenoside-Rh2 on inhibition of human glioma cell line SHG-44

  18. 替尼泊苷与尼莫司汀联合治疗MGMT阴性表达的恶性胶质瘤:附18例经验

    Teniposide and Nimustine Combination Therapy for Patients with MGMT Negative Gliomas : Experience of 18 cases

  19. 恶性胶质瘤中IL-13Rα2和PCNA的表达及其与病理特征和预后的关系

    Expression of IL-13R α 2 and PCNA in malignant glioma and its relationship with pathological characteristics and prognosis

  20. 恶性胶质瘤细胞SHG-44诱导分化后的差异蛋白质组双向电泳分析

    Analysis of differential proteome of malignant glioma SHG-44 cells after induced differentiation with two-dimensional gel electrophoresis

  21. 目的研究光动力诊断(PDD)和荧光指导的肿瘤切除在脑恶性胶质瘤治疗中的作用。

    Objective To study the usefulness of the intraoperative photodynamic diagnosis ( PDD ) and fluorescence-guided resection of malignant gliomas .

  22. 结果恶性胶质瘤患者尿液EGF含量显著高于低恶度胶质瘤患者尿液EGF含量。

    Results It is found that the contents of EGF in urine of the patients with low grade gliomas were much higher than those of the patients with high grade ones .

  23. 结果各组胶质瘤均有BfgfmRNA表达,且表达水平以恶性胶质瘤为高。Ki-67标记指数在胶质瘤中升高显著。

    Results The bFGF mRNA expressed in all gliomas with different grades and its expression level was the highest in glioma grade IV. Ki-67 label index was higher in high grade gliomas than in low grade gliomas .

  24. 方法:对资料完整的48例脑恶性胶质瘤患者进行回顾性研究,其中6例手术全部切除,42例部分切除,星形细胞瘤III级32例、IV级16例。

    Methods : Total of 48 evaluable glioma patients were studied retrospectively 6 patients received grossly total resection , 42 patients subtotal resection , 32 cases were in astrocytoma grade III , 16 cases in grade IV .

  25. 随访24例患者,血浆LSA均高于正常的恶性胶质瘤患者,术后1个月LSA稍下降(P<0.01);

    The plasma LSA level in the patients with malignant glioma was significantly higher than in patients with benign tumor ( P < 0.01 ) .

  26. 人恶性胶质瘤细胞系CHG-5和SHG-44细胞骨架的比较研究

    Comparative study on cytoskeleton of human glioma cell line CHG-5 and SHG-44 in vitro

  27. 接着,科学家将含有缓慢释放BMP4的粒子直接送达植入恶性胶质瘤细胞的小鼠脑内。

    Next , the scientists delivered slow-release BMP4-containing " beads " directly into mouse brains with implanted glioblastoma cells .

  28. 背景与目的:应用甲基化敏感性APPCR法探讨去甲二氢愈创木酸(NDGA)诱导人恶性胶质瘤细胞系SHG-44细胞分化过程中基因组甲基化状态改变。

    BACKGROUND & AIM : To investigate the genomic methylation pattern in the differentiation of glioma cell induced by nordihydroguaiaretic acid ( NDGA ) with methylation-sensitive arbitrarily primed PCR ( AP_PCR ) .

  29. 血管生成抑制剂TNP-470对人恶性胶质瘤细胞系SHG44及其移植瘤生长的影响

    Inhibitory effects of angiogenesis inhibitor TNP-470 on growth of a human malignant glioma cell line SHG-44 and its xenografted tumor

  30. 作者对正常健康人73名、颅内良性肿瘤40例、恶性胶质瘤30例及脑转移瘤13例的血清TSA进行测定。

    Total sialic acid was determined in the serum in 73 normal controls , 40 patients with intracranial benign tUmor , 30 patients with malignant glioma , and 13 patients with metastatic brain tumor .