家族性偏瘫型偏头痛

家族性偏瘫型偏头痛（familialhemiplegicmigraine，FHM）中Na+/K+ATP酶α2亚基ATP1A2基因突变的发现，是偏头痛遗传学研究的重大突破。

The finding of mutations in the Na + / K + - ATPase α - subunit gene in familial hemiplegic migraine ( FHM ) is a great breakthrough in the research of migraine genetics .

文章就CSD与偏头痛神经生物学机制关第进行了综述，重点阐述了家族性偏瘫型偏头痛的基因突变以及CSD在偏头痛病理生理学机制和预防性治疗等方面的意义。

This article reviews the relationship between SCD and neurobiological mechanism of migraine , particular expounding the significance in the gene mutations of familial hemiplegic migraine , CSD in migraine pathophysiological mechanisms and their prophylactic treatment .

对家族性偏瘫型偏头痛（FHM）家系CACNA1A基因的鉴定，掀起了偏头痛分子遗传学研究的热潮。

The identification of familial hemiplegic migraine ( FHM ) calcium channel α 1A ( CACNA1A ) has set off an upsurge of research into migraine molecular genetics .