发病学

  • 网络Pathogenesis
发病学发病学
  1. 目的为揭示P53抗癌基因与卵巢上皮肿瘤发病学的关系,对P53基因5-8外显子突变进行了检测。

    Objective To evaluate whether P53 gene mutation was related with the pathogenesis of ovarian malignant epithelial tumors or not .

  2. 当前,国内一些学者认识到阳虚在消渴病发病学、病机学中的重要地位,并运用温阳法治疗DM取得较好临床疗效。

    At present , some Chinese scholars have realized the importance of yang asthenia in pathogenesis and pathology of DM , and have achieved good clinical healing efficacy .

  3. 内皮功能异常在X综合征患者中的发病学意义

    The significance of endothelial dysfunction in the pathogenesis of syndrome X

  4. HIV病发病学新概念:肠淋巴组织主要病灶论

    New concepts in pathogenesis of HIV disease : hypothesis main pathogenic site of intestinal lymphoid tissue

  5. 目的:对原发性高血压(EH)患者的多相个性及其综合干预治疗前后的观察,探讨EH发病学及综合治疗的意义。

    Objective : To study the multiple personality and significance of combined intervention in patients with essential hypertension .

  6. 目的:探讨淋巴途径在二次打击致大鼠MODS的发病学作用。

    AIM : To explore the role of intestinal lymphatic pathway in MODS pathogenesis by two-hit in rats .

  7. 动脉粥样硬化(atherosclerosis,AS)是许多心、脑血管病的共同发病学基础,是引起老年人死亡的最主要原因之一。

    Atherosclerosis is the pathological basis of various cardio - and cerebrovascular diseases and accounts for the major causes of death in the elderly people .

  8. 较之猪眼小梁细胞,牛眼小梁细胞更适用于今后研究TGFβ及其受体与原发开角型青光眼发病学的关系。

    Bovine trabecular cells may be more suitable to investigate the relationship between TGF β, TGF β R and primary open angle glaucoma than porcine trabecular cells .

  9. 结论肠淋巴途径在失血性休克致大鼠肠道屏障功能下降、引起肠源性BET的发病学中具有重要作用。

    Conclusion The results demonstrate that the intestinal lymphatic pathway plays an important role after compromise of gut barrier function in carrying out BET after hemorrhagic shock .

  10. NZB/NZWF1小鼠外周血CD4~+CD8~+细胞及ICAM-1表达变化对狼疮样改变的发病学意义

    The Pathogenetic Significance of CD_4 ~ + , CD_8 ~ + Cell and ICAM-1 Expression Changes in Peripheral Blood of NZB / NZW F1 Mice Lupus-like Change

  11. 目的探讨原发性心脏肿瘤(PCT)的发病学特点、病理组织学类型及临床病理特征。

    Objective To study pathogenic characteristics , histologic types and clinical pathologic features of primary cardiac tumors ( PCT ) .

  12. 其中,脑能量代谢失衡是引起神经系统并发症的共同因素,在CPB相关脑损伤的发病学上占有十分重要的地位。

    The common factor leading to neurologic complications is the imbalance of cerebral energy metabolism , which plays a very important role in the pathogenesis of CPB-related cerebral injury .

  13. 结合其硒水平与GSH-Px活性低,组织抗氧化能力下降和膜的早期老化、氧化损伤、细胞坏死,膜破溃小泡化(基质小泡)与KBD早期化骨的机制及其病因发病学意义进行了讨论。

    The mechanism of early ossification of KBD has been discussed combined with their low level of selenium and GSH-Px , oxidative damage and vesiculation of membrane .

  14. 为探讨内皮素(ET)在肝坏死中的发病学意义,作者在四氯化碳(CCl4)肝坏死模型上观察血浆ET-ir变化及其ET抗血清(ET-Ab)的治疗作用。

    The changes in rat plasma level of endothelin-ir ( ET-ir ) during CCl4-induced hepatic necrosis and the therapeutic action of ET-antiserum ( ET Ab ) were observed .

  15. 前言细胞凋亡异常在许多人体恶性肿瘤的发病学上占有十分重要的地位。caspase-9及Bcl-2、细胞色素-C等多种癌基因产物均参与细胞凋亡的调节。

    Apoptosis malfunctions have very important in the pathogenesis of many Human malignant tumors , caspase - 9 , Cytochrome c and Bcl - 2 etc. turner gene product participate in the regulation of cell apoptosis .

  16. 目的观察结扎肠系膜淋巴管对二次打击致大鼠多器官功能障碍综合征(MODS)体液免疫功能的影响,探讨淋巴途径在MODS发病学中的意义。

    Objective To study the effects of mesenteric lymph duct ligation on humoral immunity function in multiple organ dysfunction syndrome ( MODS ) rats subjected to two-hit , and observe the meaning of lymph pathway in terms of MODS .

  17. 为探讨调节肽CGRP在休克中的发病学意义,本工作在结扎大鼠盲肠加穿孔的败血症休克模型上,观察了CGRP的变化及外源性CGRP对败血症休克过程的影响。

    In order to investigate the pathogenetic role of CGRP in septic shock , the effect of extraneous CGRP on septic shock was observed on late septic shock model produced by cecal ligature and puncture ( CLP ) in rats .

  18. 目的探讨共刺激分子B71(CD80)在血液系统恶性肿瘤发病学及免疫基因治疗中的重要作用。

    Objective To explore the significance of costimulatory molecule B7-1 ( CD80 ) and MHC molecules in the pathogenesis of hematologic malignant tumors and in the immuno-genetic therapy .

  19. 为探讨内皮素(Endothelin,ET)在败血症休克中均发病学意义,本工作在结扎大鼠盲肠并穿孔的晚期败血症模型上观察内皮素抗血清(ET-Ab)列败血症休克大鼠的治疗作用。

    In order to investigate the pathoge'netic role of endothelin in septic shock , the effect of endothelin-antiserum on septic shock was evaluated on late sepsis model ( 20h ) produced by cecal ligature and puncture ( CLP ) in rats .

  20. 着重提出两相打击学说在MODS发病学的意义,以及从液体复苏、抗氧自由基、抗内毒素治疗、抗炎性介质治疗、创面处理、营养支持、整体器官功能维护等七个方面探讨MODS的防治。

    The author emphasized the significance of the double phase attack theory in the pathogenesis of MODS and made a discussion on fluid resuscitation , anti-oxygen free redical therapy , anti-endotoxin therapy , anti-inflammatory medium therapy , wound management , nutritional support and organ function maintaining , etc.

  21. 目的探讨血清ICAM1(CD54)含量和外周血CD4+,CD8+细胞及淋巴细胞表面CD54表达变化及其在NZB/NZWF1小鼠狼疮样改变中的发病学意义。

    Objective To study the content change of ICAM-1 ( CD54 ) and the expression changes of CD_4 + and CD_8 + cell as well as CD54 of lymphocyte surface in peripheral blood of NZB / NZW F1 mice , and their pathogenetic significance to lupus-like change .

  22. 自由基在地方性甲状腺肿发病学上的可能作用

    Possible effect of free radicals in the pathogenesis of endemic goiter

  23. 血管紧张素Ⅱ在压力超负荷性心肌肥厚发病学中的意义

    Effect of angiotensin in pressure overload-induced myocardial hypertrophy in rats

  24. 丹东地区烧伤1029例发病学分析

    Morbidity Analysis of 1029 Cases of Burn in Dandong Region

  25. 实验性禽脑脊髓炎分子发病学及分子免疫机理的研究

    A Study on Molecular Pathogenesis & Molecular Immunology in Experimental Avian Encephalomyelitis

  26. 中青年复发性脑梗死的发病学研究

    The research of pathogeny of recurrence cerebral infarction in teen-agers and middle-ages

  27. 重症急性胰腺炎的临床发病学特征及预后分析

    Clinical morbidity characteristics and prognosis in severe acute pancreatitis

  28. 能量代谢基因表达改变在大鼠卒中发病学中的意义

    Changes of energy / TCA cycle genes expression in brain tissue of stroke-like episode rats

  29. 应用家兔失血性休克模型观察肾小球的超微结构改变并探讨其在发病学的上意义。

    The model of hemorrhagic shock of rabbits was established to observe ultrastructural changes of glomeruli .

  30. 克山病心肌线粒体膜氧化损伤的实验研究及其发病学意义

    Experimental study of oxidative damage in mitochondrial membrane of myocardium of keshan disease and its pathogenetic significance