全身性感染

  • 网络Systemic infection;sepsis
全身性感染全身性感染
  1. 全身性感染的输液治疗:晶体,胶体或白蛋白?

    Fluid infusion in sepsis : colloids , crystalloids or human albumin ?

  2. 降钙素原与全身性感染的相关性研究

    Correlative study of procalcitonin ( PCT ) and sepsis

  3. PCR方法诊断重症急性胰腺炎继发全身性感染13例研究

    PCR Diagnosis of Severe Acute Pancreatitis Complicated with Systemic Infection

  4. 全身性感染与Toll样受体2、4相关性研究进展

    The New Progress of Study on Toll Like Receptor 2,4 in Correlation with Sepsis

  5. 近年来由GAS引起的猩红热和严重全身性感染的发病率有所增长,引起了人们对该类细菌感染的更多关注。

    In recent years , there is an increase in cases of scarlet fever and severe systemic infections caused by GAS , which aroused greater concern for streptococcal infections .

  6. 目的:我们检验这样一个假设,即患有COPD的病人患动脉硬化的危险会升高,动脉硬化和骨质疏松、全身性感染有关联。

    Objectives : We tested the hypothesis that patients with COPD would have increased arterial stiffness , which would be associated with osteoporosis and systemic inflammation .

  7. 先天性CMV感染中全身性感染占16.7%,围生期全身性感染占1.8%,生后感染者无全身性感染发生;

    Generalized infection , the incidence of congenital infection and perinatal infection was 16.7 % , 1.8 % , respectively . It did not occur in postnatal infection .

  8. 大部分入院时间较晚。(2)吸入性损伤、休克、全身性感染、多脏器功能衰竭(MOF)是主要的合并伤及主要的并发症,也是主要的死亡原因。

    Inhalation injury shock , general infection and MOF are the symphysis injury and the complication , it is the major death cause too .

  9. 最近,被初步证明可能在WSSV全身性感染对虾的起始步骤中起关键作用。

    Recently , it had been proved to be likely to play a key role in the initial steps of the systemic WSSV .

  10. 结果吸入性损伤、休克、全身性感染是烧伤后导致MOF的常见诱因,烧伤合并MOF后果严重、死亡率极高。

    Results Inhalation injury shock , general infection is major cause leading to MOF . The consequences would be too ghastly to contemplate the death rate about burn with MOF .

  11. 柯萨奇病毒B3型(coxsackievirusB3,CVB3)是引起人类病毒性心肌炎、无菌性脑膜炎和新生儿全身性感染等疾病的主要病原体。

    Coxsackievirus B3 ( CVB3 ) is an important human pathogen which causes a wide spectrum of diseases , ranging from mild respiratory illness to severe myocarditis and neurological disorders .

  12. 烧伤面积大于80%、合并吸入性损伤、重度休克和全身性感染者,MOF发生率均明显高于相应对照病人,且以并发全身性感染和重度休克者最高。

    In those patients with TBSA over 80 % , inhalation injury , severe shock and systemic infections , the incidence of MOF was significantly higher . Systemic infections and severe shock took the first two places .

  13. 细菌内毒素可以在局部肠道炎症如炎性肠道疾病(IBD)、坏死性小肠结肠炎(NEC)等和全身性感染诱发的肠道炎症损伤中扮演着十分重要的角色。

    Bacterial endotoxin plays an important role in the formation of regional intestinal inflammation such as inflammatory bowel diseases ( IBD ), necrotizing enterocolitis ( NEC ), etc , as well as the formation of intestinal injury from systemic inflammation .

  14. 感染性急性肺损伤(ALI)发病机制涉及细菌、病毒、原虫等病原体对肺组织细胞的直接伤害,或全身性感染导致病原体、毒素、炎症代谢产物对肺组织的直接或间接伤害。

    Sepsis associated acute lung injury ( ALI ) and acute respiratory distress syndrome ( ARDS ) are often caused by bacteria , viruses , and other pathogens in the lungs , or as a result of sepsis associated with pathogens , toxins and metabolites of systemic inflammation .

  15. 目的研究白细胞素1(IL1)α,IL1β和IL1受体拮抗剂(IL1ra)基因多态性各等位基因及基因型在全身性感染患者中的分布频率。

    Objective To determine the allele frequencies and genotype distribution of interleukin ( IL ) - 1 α, IL-1 β and IL-1 receptor antagonist ( IL-1ra ) gene polymorphism in septic patients .

  16. 白细胞介素-1家族细胞因子基因多态性对全身性感染患者预后的影响

    Genomic polymorphism within interleukin-1 family cytokines influences the outcome of septic patients

  17. 烧伤后全身性感染防治的临床研究

    Clinical Study on the Prevention and Treatment of Systemic Infection in Burn Patients

  18. 腹腔感染和全身性感染是肠瘘治疗中的难点之一。

    Intra-abdominal infection and systemic infection are the difficulties in treatment of fistula ;

  19. 先天性感染组79例,其中全身性感染32例。

    Among the 79 cases of congenital infection group , 32 cases were general infection .

  20. 42年严重烧伤全身性感染的防治经验

    42 years ' experience on the prevention and treatment of systemic infection after severe burn

  21. 重度休克和早期全身性感染是其主要发病因素;

    Clinical course of this pattern simulated to that of ARDS and the main etiological factors included severe shock and early systemic infections ;

  22. 结论观察结果表明,大面积深度烧伤后,早期短程的广谱高效抗生素冲击治疗,既节省费用,又有效地防治了全身性感染。

    Conclusion Early use of high-potency antibiotics at a short treatment course after extensive severe burn is effective to prevent infection and reduce the cost .

  23. 致病菌大多是革兰阴性菌,通常引起腹泻、腹痛、发热甚至引起全身性感染。

    The vast majorities of intestinal infections caused by Gram-negative bacteria usually leed to diarrhea , abdominal pain , fever , even give rise to systemic infection .

  24. 结论全脾切除后自体脾组织移植能够成活并增生变大,而且能够维持一定的免疫功能,可预防严重的全身性感染发生。

    Conclusion After the whole spleen was resected the autografting of spleen can survive , get large , maintain certain immune function and prevent severe general infection .

  25. 他们的生命经常由于全身性感染而缩短。骨髓移植可被用于治疗此类疾病,但是并不总是能够成功。

    Their lifespans are usually shortened by systemic infection , and while bone marrow transplants can be used to treat the disease , they are not always successful .

  26. 本文结果提示,大面积烧伤、吸入性损伤、重度休克和全身性感染是烧伤后肺脏损害的主要发病因素,以吸入性损伤所致肺功能衰竭发生率最高。

    These findings suggested that besides the severity of burns itself , inhalation injury , severe shock and systemic infections were the main etiological factors invoking postburn lung damage , and the inhalation injury ranked the first .

  27. 另外,全身性感染患者中基因型A2/2,B2/2和RN2/2病死率明显增加(分别为80%,81%和71%)。

    In addition , genotypes A2 / 2 , B2 / 2 and RN2 / 2 were associated with a significantly higher mortality ( 80 % , 81 % and 71 % , respectively ) in septic patients .

  28. 临床观察发现,严重烧伤后患者往往出现骨骼肌组织显著性高蛋白分解代谢,如果合并全身性感染,这种代谢反应将更为剧烈,使机体陷入严重的低蛋白血症和负氮平衡。

    In clinical observation , we can find that there are obviously high protein catabolism in skeletal muscle in burn patients , and it can become more intense if combined with systemic inflammation , as result , the patients can fall into severe hypoproteinemia and negative nitrogen balance .

  29. 2例患者死亡(1例在术后16d死于全身性念珠菌感染,另1例在术后26d死于门静脉栓塞)。

    Patients died after the operation , one died of severe infection , the other died of embolism of portal vein .

  30. 前降钙素是全身炎症性感染反应的标志物

    Procalcitonin as a marker of the systemic inflammatory response to infection