自身氧化

许多研究表明非酶糖化和葡萄糖自身氧化伴随着游离基的产生，是一个糖化氧化过程。

Many studies show that nonenzymatic glycation and glucose autoxidation which are accompanied by generation of free radicals is a process of glycation and oxidation .

而合金元素C的过渡则随着焊丝自身氧化性的增加，由增碳型过渡发展到损失型过渡。保护气氧化性的增加更容易发生增碳型过渡。

For the element C , as the oxidizability of the wire increases the transfer changed from increase to lose and as the oxidizability of the arc increases the transfer increase occurs more easily .

机理分析表明添加剂CO、CH4和H2主要是通过自身氧化过程中形成链分支反应，促进OH等活性基的生成来影响SNCR反应的。

By mechanism analysis , the effects of these additives on NOx reduction are achieved principally by promoting the production of OH and other radicals through chain reaction in their own oxidation process .

无溶剂条件下肉桂醛自身氧化还原反应的研究

Cannizzaro Reaction of the Cinnamon Aldehyde under Solvent - Free Condition

结论：左旋多巴自身氧化产生大量的自由基，促进帕金森病发展，黄芪多糖可降低左旋多巴对神经元的毒性作用，作用有时间依赖性。

CONCLUSION Free radical produced by self-oxidation of bendopa can promote development of Parkinson disease . APS can relieve the toxicity of bendopa on neurons and the effect has time dependency .

添加Fe、Cu、Ce氧化物可促进催化剂自身的氧化还原循环，提高氧化活性，降低反应温度；

Addition of Fe , Cu , Ce metal ′ s oxides to promote the redox cycle of catalysts themselves , which increases oxidation activity and lowers reaction temperature ;

软骨细胞为了保护自身免受氧化应激的损伤，提高了细胞内过氧化氢酶（CAT）和超氧化物歧化酶（SOD）的活性。

In addition , the living cells increased the activities of catalase ( CAT ) and superoxide dismutase ( SOD ) to protect them from T-2 toxin-induced oxidative stress .

二氧化钛层主要通过钛基体表面自身的氧化获得，且多数制备过程中需要经过后续高温处理，这对于基体材料的选择以及处理后二氧化钛薄膜的生物活性都有影响。

Mainly the TiO2 layer is obtained by oxidizing of titanium substrate itself . However , most of the preparation process contains heat treatment , which restricts the choice of substrate material and the bioactivity of TiO2 films .

蛋氨酸可以将全部Au3+增感剂还原为Au，其自身部分被氧化为蛋氨酸亚砜，并且在其脱质子的羟基部位与Au形成Au-O-C结构的金属配合物。

The methionine was oxidized to methionine sulfoxide , in the same time reducing Au 3 + to gold atom . The hydroxy of methionine could coordinate Au , forming Au-O-C coordinate metal complex .

高同型半胱氨酸血症能引起心脏发育异常，其自身所致的氧化应激损伤可能是重要发病环节。

The research reported that blood hyperhomocysteinemia can cause abnormal heart development , which own injury caused by oxidative stress may be an important link in the pathogenesis .

结果表明，在纯CO2与Ar+20%CO2气保护条件下，合金元素Mn的过渡均处于损失状态，并且随着焊丝自身或保护气氧化性的增加，其损失增大。

The experimental results show that the element Mn was the lose regardless of CO 2 or Ar + 20 % CO 2 shielding gas and as the oxidizability of the arc or the wire increases the lose aggravates .