复氧

人重组白细胞介素-6可减少缺氧-复氧后大鼠海马培养神经元的DNA损伤

RhIL-6 can decrease DNA damage of cultured rat hippocampal neurons after anoxia and reoxygenation

NF-κB圈套策略抗大脑皮质神经元氧糖剥夺/复氧后炎性损伤及其机制的研究

The Mechanism of NF - κ B Decoy ODNs Resisting the Inflammatory Injury in Primary Cortical Neurons after Oxygen Glucose Deprivation / Reoxygenation

雌激素在缺氧/复氧心肌细胞中对NF-κB及粘附分子表达的影响

Effect of Estrogen on Hypoxia / Reoxygenation-induced Expression of Adhesion Molecule Through NF - κ B in Cardiac Myocytes

人参皂苷Rd及其C（12）手性异构体对缺氧/再复氧后蛋白酪氨酸磷酸化的抑制作用

Inhibitory effects of ginsenoside-R_d and its C_12 isomer on tyrosine - phosphorylation induced by hypoxia / reoxygenated injury

X染色体连锁凋亡抑制蛋白对缺氧-复氧诱导的新生大鼠心肌细胞凋亡的影响

Effects of X-linked inhibitor of apoptosis protein on cultured cardiomyocytes of neonate rat apoptosis induced by hypoxia / reoxygenation

辅酶Q（10）对大鼠离体灌流心脏缺氧&复氧损伤的保护作用

Protective effect of coenzyme q_ ( 10 ) on myocardial anoxia-reoxygenation injury in isolated perfused rat heart

磷酸肌酸对大鼠心肌缺氧、复氧心肌线粒体肌酸激酶肌膜型亚基mRNA表达水平的影响

The influence of phosphocreatine on expression of mitochondrial sarcomeric creatine kinase mRNA of myocardium ischemia and reperfusion in rats

C组，缺氧预适应组（APC），内皮细胞在缺氧前遭受4个循环短时间缺氧/复氧；

Group C , hypoxia-preconditioning was performed by 4 cycles ' short anoxia and reoxygenation before prolonged anoxia .

结论海马神经元缺氧/复氧后，caspase-3样蛋白酶被激活，参与神经元损伤过程；

Conclusion caspase-3 contributes to the neuronal damage following hypoxia / reoxygenation ;

本结果表明，缺氧－复氧能使体外培养海马神经元发生DNA损伤，rhIL－6可减少缺氧－复氧后海马神经元的DNA损伤。

The results indicated that rhIL 6 can decrease DNA damage of hippocampal neurons after anoxia reoxygenation .

结果表明随缺氧和复氧时间的延长细胞内Na＋均增加。

The results indicated that [ Na + ] I rose gradually with the prolongation of hypoxia and reoxygenation .

结论：药物预适应可以对缺氧复氧损伤产生明显的拮抗作用，它可能是通过激活细胞内蛋白激酶C来发挥作用的，而各部位的心肌细胞产生药物预适应的能力没有显著性差别。

CONCLUSION : Nitrate medicine protected the cells from different parts of the heart against hypoxia and reperfusion , and this function may achieve through the activation of PKC .

目的研究短暂缺氧、血清剥夺复氧复注血清（再灌流）后有无神经细胞顿抑现象存在及可能的发生机制。Li/TK细胞3d，即可将其杀死。

Objective To identify whether there is nerve cells stunning after transient anoxia-serum deprivation / reperfusion and to explore the probable underlying mechanism of the phenomenon .

缺氧/复氧对培养SD乳大鼠心肌细胞端粒酶逆转录酶的影响

Effects of Hypoxia / reoxygenation on Telomerase Reverse Transcriptase ( TERT ) Gene of Neonatal SD Rat Cardiac Myocytes

结论低氧预适应能够提高鼠脑星形胶质细胞缺氧/复氧耐受性，可能与SOD活性增高对抗缺氧/复氧过程中产生的氧自由基对细胞的损伤有关。

Conclusion Hypoxic preconditioning can improve astrocytes ' tolerance to hypoxia / reoxygenation by increasing SOD activity to inhibit oxygen free radicals .

方法以原代培养仔鼠心肌细胞建立缺氧复氧损伤模型，应用高效液相色谱检测心肌细胞内ATP、ADP、AMP含量；

Methods A model of H / R of cultured myocardial cells was used .

缺氧2h/复氧2h；

Hypoxia 2 hours / reoxygenation 2 hours ;

EPO预处理对心肌缺氧复氧损伤保护作用的研究

Protective effects of EPO pretreatment on myocardium with hypoxia / reoxygenation injury in rats

建立大鼠脑皮质微血管内皮细胞体外培养，给予HEEL、GAG、heparinase、Sdc-1Ab、Sdc-3Ab、CAP37Ab预培养6h，然后再予以常氧环境继续培养和缺氧2h/复氧2h处理。

To established the rat brain microvascular endothelial cells culture .

BK（Ca）通道活动增强介导了缺氧/复氧诱导的培养海马神经元凋亡

Activation of BK_ ( Ca ) Channels Mediates Hippocampal Neuronal Apoptosis in Culture Induced by Hypoxia / reoxygenation

EPO预处理在心肌缺氧复氧损伤中的保护作用及其NF-κB信号机制的研究

Study of the Protective Effects of EPO Pretreatment on Myocardium with Hypoxia / reoxygenation Injury and the NF - κ B Signal Transduction Mechanism

人参皂苷Rb1与Rg1对肾小管细胞缺氧复氧损伤模型的影响

Effects of GS Rb_1 and Rg_1 on Cultured Renal Tubular Cells Injured by Hypoxia-reoxygenation

结果：EP可明显降低缺氧/复氧引起的心肌细胞凋亡，并明显增强SD乳鼠心肌细胞p-AKt/AKt比值；

Results : EP significantly decreased the apoptotic index induced by hypoxia / reoxygenation and strongly increased p-AKt / Akt level .

在培养基中加入黄嘌呤－黄嘌呤氧化酶（X－XOD）和丝裂霉素C（MMC），损伤心肌细胞以及用缺氧－复氧的方法造成心肌细胞损伤。

Damage was induced in the cardiomyocytes with Xanthine - Xanthine oxidase ( X-XOD ), mitomycin-C ( MMC ), and hypoxia-reoxygenation .

体外原代培养胎鼠海马神经元，建立缺氧/复氧诱导损伤的海马神经元凋亡模型，观察EA对损伤海马神经元的保护作用。

Rat hippocampal neurons in primary culture were used , and an apoptosis model was induced by hypoxia / reoxygenation .

PPARγ激动剂罗格列酮对缺氧/复氧大鼠心肌细胞氧化应激和凋亡的影响

PPAR γ agonist rosiglitazone alleviates hypoxia / reoxygenation-induced oxidative stress and apoptosis in rat cardiac myocytes

缺氧2h/复氧4h，每个时间点5瓶细胞。

Hypoxia 2 hours / reoxygenation 4 hours , 5 bottles at each time point .

低氧预处理对缺氧-复氧后体外培养大鼠海马神经元Fos、Jun表达和神经元凋亡的影响

Effects of hypoxic-preconditioning on Fos and Jun expression and apoptosis in cultured rat hippocampal neurons after anoxia-reoxygenation in vitro

复氧方式对慢性缺氧幼年大鼠肺组织TNF-α、IL-1β和IL-10mRNA表达的影响

Effects of reoxygenation techniques on expressions of TNF - α, IL-1 β and IL-10 mRNA in chronic hypoxia lungs of immature rat

EPO预处理可能通过NFκB活化的负反馈机制抑制缺氧复氧后心肌细胞NFκB活性的升高。

EPO pretreatment might inhibit the activation of NF - κ B after H / R through the negative feed-back mechanism of NF - κ B activation .