合体细胞

结果：治后1－7d，在虫的皮层、合体细胞、肌束、实质组织、肠管上皮细胞和卵黄细胞等均发现超微结构变化。

RESULTS : One to 7 d after treatment , ultrastructural damage by artemether were apparent in the tegument , syncytium , musculature , parenchymal tissues , intestinal epithelial cells and vitelline cells .

结论HBV可感染胎盘合体细胞并且在其中复制，从而使胎儿在宫内被感染。

Conclusions HBV can be transmitted from mother to fetus in uterus by infecting and duplicating in the syncytial trophoblast .

组织学检查：具有合体细胞结节、血管合体膜、血管增多、朗罕氏细胞、绒毛纤维素样坏死等绒毛均高于对照组（P＜0.01）；

Histologic examination showed that the numbers of villi with syncytial cell node , vascular syncytial membrane and hyperplasia , Langerhans ′ cells , villus fibrinoid necrosis in high altitude group were all higher than those in control group ( P < 0.01 ) .

结果：胎盘郎汉斯细胞及合体细胞呈现阳性反应，而阴性对照无反应。

Results : The placental trophoblastic cells had strong positive reactions while the control groups showed negative reactions .

在合体细胞游离面有纤维蛋白样物质沉着处，基底膜往往同时增厚。

On the free surfaces of syncytial cells , fibrous deposition was sccn and the basal membrane of these cells was thickened .

局部为梭形细胞型和合体细胞型脑膜瘤形态，与横纹肌样细胞有过渡。

The fibrous and meningothelial meningioma was found in local area where the transition from spindle cells to rhabdoid cells could be revealed .

1例肺泡腔渗出物中有合体细胞形成，1例有燕麦细胞。

Oat cells were seen in exudates in alveolar spaces in one case , syncytial cell formation were seen in an another case .

本文介绍一种用吸附式电极记录合体细胞组织生物电位的改进装置。

An improved device of the adhesive electrode for recording the bioelectrical potential of animal 's syncytial tissue is described in this paper .

人胎盘绒毛膜绒毛的结构由内向外依次是绒毛基质，细胞滋养层细胞和其外层的合体细胞滋养细胞。

The bulk of the human placenta consists of branching chorionic villi with an outer syncytiotrophoblast , underlying cytotrophoblasts , and a stromal core .

30天时脐血1嵌合体细胞上升为70.0%，脐血2嵌合体细胞下降为30.0%。

Subsequently relative amounts of chimera from unit 1 went up to 70.0 % at 30 days , and that from unit 2 declined to 30.0 % .

结果（1）重度窒息组胎盘绒毛具有合体细胞结节、细胞滋养细胞增生的绒毛数显著高于正常组（P<0.05）；

Results : ( 1 ) The number of the placental villus with the syncytial knots cytotrophoblasts in SAN group was bigger than that in NN group ( P < 0.05 ) .

超微结构观察：高原胎盘绒毛的合体细胞及朗罕氏细胞增生明显，粗面内质网高度扩张，滋养叶基底膜明显增厚，可达正常的几倍至十几倍。

Ultrastructural observation in high altitude group showed significant hyperplasia of syncytial cells and Langerhans ′ cells in placenta villi , high expansion of rough surfaced endoplasmic reticulum and very thick trophoblastic basement membrane .

电镜下合体细胞微绒毛肿胀、稀少、粗面内质网普遍扩张，部分合体细胞线立体肿胀、溶酶体增加、胎儿毛细血管内皮细胞肿胀等改变

Under electron microscopy , there were swelling and decrease in number of micro-villi , and dilatation of rough endoplasmic reticulum in all syncytial cells , and swelling of mitochondria and increase of lysosomes in part of syncytial cells , and swelling of endothelial cells in some fetal capillaries

合体滋养层细胞（ST）为分化良好的细胞。

The syncytiotrophoblasts ( ST ) were well differentiated .

Leptin主要表达于胎盘合体滋养层细胞的胞浆中。

Leptin expressed on the placenta villous syncytiotrophoblast .

westernblot检测体外培养的合体滋养层细胞IDO蛋白质表达；

Western blot was applied to determinate the expression of IDO protein on cultured syncytiotrophoblast ;

结论：HCV持续感染可抑制体外培养人胎盘合体滋养层细胞包括侵袭力和激素合成、分泌功能在内的多种生物学功能。

Conclusion : HCV persistent infection suppressed the biological behavior of human placental syncytiotrophoblast in vitro .

结果：1.活化的NF一kB蛋白主要表达于细胞浆，偶见于细胞核，在合体滋养细胞、血管内皮细胞、蜕膜细胞中均可检测到。

Result : 1.Activated NF-kB was present predominantly in syncytiotrophoblastic cells , some stem villous endothelium and decidual cells .

结果表明，肿瘤性合体滋养细胞（ST）、中间型滋养细胞（IT）及细胞滋养细胞（CT）内细胞角蛋白的阳性表达率均为100%。

The results showed that the positive rate of neoplastic syncytiotrophoblast ( ST ) ; intermediate trophoblast ( IT ) and cytotrophoblast ( CT ) was 100 % respectively .

结果显示：LN在葡萄胎中的游离绒毛合体滋养细胞（LNR）呈阳性着色、LN呈阴性；

The results show : in hydatidiform moles , syncytiotrophoblast of floating villi , LN is negative ;

结论ICP患者胎盘合体滋养细胞bax基因过度表达，胎盘细胞凋亡率升高，影响胎盘功能，可能是ICP并发围生儿缺氧的病理生理基础之一。

Conclusion One of the possible mechanism involved in perinatal of ICP is apoptosis caused by overexpression of bax in syncytiotrophoblast leading placental dysfunction .

结果KiSS-1主要位于胎盘绒毛小叶的合体滋养细胞。

Results : KiSS-1 was mostly found in syncytiotrophoblast .

结论：丙型肝炎病毒持续感染时体外培养人胎盘合体滋养层细胞MMP-2和MMP-9合成分泌能力下降，为非特异性。

CONCLUSION : HCV persistent infection suppressed the synthesis and secretion of MMP-2 and MMP-9 of human placental syncytiotrophoblast in vitro , which is not specific .

结果：正常早期妊娠绒毛细胞滋养细胞增殖指数为268.7±6.7/500，且见细胞浆阳性现象，合体滋养细胞末见PCNA阳性着色；

Results : In early pregnant chorionic villi , proliferating index of cytotrophoblasts was 268.7 ± 7.6 / 500 , not only nucleus but cytoplasma were PCNA positive staining , syncytiotrophoblasts was negative .

结果bFGF和Bax在滋养细胞、上皮细胞、间充质细胞和成纤维细胞胞浆内有表达，Bcl-2在绒毛膜合体滋养细胞上有表达；

Results The expression of bFGF and Bax were located in the plasma of all types of cells including trophoblast , epithelial , mesenchymal and fibroblast , but the expression of Bcl-2 was only observed in the chorionic syncytiotrophoblast ;

实验结果说明，在兔正常妊娠中，胎盘合体滋养层细胞发生凋亡，且随妊娠的进行，凋亡细胞数量增多，胎盘细胞凋亡主要与细胞中Bax∶Bcl2的比例相关。

The results demonstrated that apoptosis occurred in syncytiotrophoblast in rabbit pregnancy and apoptotic cell death increased from early to the end of gestation period , moreover Bax to Bcl-2 ratio may be related to apoptosis in placenta .

结果各组孕产妇胎盘组织均有IDO蛋白表达，且主要表达于胎盘合体滋养细胞的胞质中，胎盘间质细胞及血管内皮细胞胞质中也可见少量表达。

Results IDO protein was expressed in the placenta , mainly in syncytiotrophoblast , of all the three groups , small amounts of IDO could be seen in vascular endothelial and interstitial cells .

结论胎盘合体滋养层细胞Syndecan-1蛋白表达减少可能是妊高征的发病机制之一。

Conclusion : It shows that the reduced expression of Syndecan-1 in placenta syncytiotrophoblast of PIH may be one of the important factors responsible for the pathogenesis of PIH .

目的：研究妊高征（PIHS）患者胎盘合体滋养细胞质膜（STCPM）中生化指标的变化及原因。

Objective : To study the changes of the biochemical indexes in syncytiotrophoblast cell plasma membranes ( STCPM ) of pregnancy induced hypertension syndrome ( PIHS ) and the cause of these changes .

人体正常组织中胎盘合体滋养层细胞、小肠和结肠粘膜上皮细胞、胆小管膜、乳腺小叶及血管内皮细胞和干细胞均能检测到BCRP的表达；

In normal human tissues , low to high expressions of BCRP in placental syncytiotrophoblasts , in the epithelium of the small intestine and colon , in the liver canalicular membrane , in ducts of the breast , in endothelium of the blood vessel and in stem cells were reported .