上运动神经元

上运动神经元损伤所致的痉挛、僵硬和肌张力增高可以有F波放电的改变。

The increase of spasm , stiffness and muscular tension induced by upper motor neuron injury can revoke change of F-waves .

目的：探讨A型肉毒毒素（botulinumtoxintypeA，BTX-A）对上运动神经元损伤后肢体肌肉痉挛的治疗价值及其剂量影响。

Objective : To investigate the effect of botulinum toxin type A ( BTX-A ) on control of muscle spasticity and dose relationship in upper motor neuron syndrome .

结论DTI和MRS结合可以明显提高ALS上运动神经元受累的诊断敏感性。

Conclusion A combination of DTI and MRS could markedly increase the diagnostic sensitivity in upper motor neuron involvement of ALS .

目的观察大鼠上运动神经元损伤后，骨骼肌乙酰胆碱受体γ亚单位和ε亚单位mRNA的变化以及脊髓前角降钙素基因相关肽的变化。

Objective To observe the time profile of the changes of mRNA of γ and ε subunit of acetylcholine receptor ( AChR ) in skeletal muscle and the level of calcitonin gene related peptide ( CGRP ) in spinal motoneuron after upper motoneuron injury .

CMCT是从脑皮质到脊髓α前角运动神经元的传导时间，主要反映上运动神经元和脊髓前角细胞的功能。

Central motor conduction time is the conduction time from the cerebral cortex to the spinal cord anterior horn motor neurons , mainly reflect on motor neurons and spinal cord anterior horn cell function .

目的：研究肌萎缩侧索硬化症（ALS）患者经颅磁刺激运动诱发电位（TMS-MEP）改变及与临床的相关性，了解其在ALS诊断和上运动神经元（UMN）损害评价中的作用。

Objective : To investigate the transcranial magnetic stimulation motor evoked potential ( TMS-MEP ) changes in amyotrophic lateral sclerosis ( ALS ) and their correlations with clinical aspects . To assess the value of TMS-MEP in the diagnosis and upper motor neuron ( UMN ) lesion evaluations of ALS .

直肠电刺激对上运动神经元损伤后痉挛状态的疗效研究

Rectal Probe Electrical Stimulation on Spasticity of Upper Motor Neuron Lesion

机器人辅助训练对上运动神经元损伤所致上肢痉挛的疗效观察

An effect of spasticity due to upper motor neurone injury by robot-aided training

上运动神经元损伤后脊髓前角运动神经元中降钙素基因相关肽的变化

Change of calcitonin gene related peptide in spinal motoneuron after upper motoneuron injury

运动诱发电位在上运动神经元损害的诊断价值

Study on motor evoked potential in the diagnosis of lesions of upper motor neuron

卒中后痉挛与上运动神经元综合征

Post stroke spasticity and upper motor neuron syndrome

治疗组采用了含肉毒毒素的治疗。排除标准：非上运动神经元损伤后的肌张力异常，综述类文献，没有对照组的文献。

Reviews and literatures without control group or not for upper motor neuron syndrome were excluded .

其中21例系上运动神经元瘫痪，15例痊愈，6例明显好转。

15 of 21 cases of upper motor neuron paralysis were cured and 6 cases were improved .

例2和例3还同时合并下肢上运动神经元损害及认知功能障碍。

Case 2 and case 3 also developed upper motor neurons impairment at the lower limbs and cognitive impairment .

巴彬斯基征是神经病学中最为常见的体征之一，也是反映锥体束损伤最可靠的体征之一，是上运动神经元损伤后一种屈肌反射的释放。

Babinski sign is not only one of the most common signs in neurology , but also one of the most reliable signs reflecting the damage of pyramidal tract .

文章对卒中后痉挛的危害、发生机制、鉴别诊断和治疗进展，以及与上运动神经元综合征之间的关系进行了综述。

The article reviews the hazard , mechanisms , differential diagnosis , and therapeutic progress of spasticity after stroke , as well as the relationship between spasticity and upper motor neuron syndrome .

结论：肉毒毒素A对缓解上运动神经元损伤后的肢体肌肉痉挛，提高其生活自理能力及运动功能疗效显著，肉毒毒素作用的量效关系尚有待确认。

Conclusion : BTX-A has significant effect on reduction of muscle spasticity , improvement of activity of daily living and motor function , but dose-effect relationship does not reach the significant level .

通过中枢运动传导时间延长，揭示临床及亚临床的上运动神经元改变，尤其适用于发现中枢性运动神经元病变的多发性硬化亚临床改变，起到支持辅助诊断作用。

Through the prolong of central motor conducting time , motor evoked potential reveals the clinical and subclinical changes of upper motor neurons , especially the subclinical damages of multiple sclerosis and exerts an accessory role in the diagnosis .

中枢性瘫痪，又称上运动神经元性瘫痪，或称痉挛性瘫痪、硬瘫。是由于大脑皮层运动区锥体细胞及其发出的神经纤维&锥体束受损而产生。

Central paralysis , also known as upper motor neuron paralysis , or spastic paralysis , rigid paralysis , is due to the damage of the pyramidal cells in the cerebral cortex motor area and the nerve fibers from the pyramidal tract .

肌萎缩侧索硬化（amyotrophiclateralsclerosis，ALS）是一种支配随意运动的上下运动神经元选择性受累的神经系统变性疾病。

Amyotrophic lateral sclerosis ( ALS ) is a progressive neurodegenerative disease , selectively involving the upper and lower motor neurons that control voluntary movement .

临床表现主要以肌萎缩、肌无力等上下运动神经元损伤症状为主，常伴有言语不清、肢体活动不利、吞咽功能障碍甚至呼吸麻痹。

The main clinical manifestations to muscle atrophy , weakness of upper and lower motor neuron injury symptoms , often accompanied by slurred speech , physical activity , swallowing dysfunction even adverse respiratory paralysis .

肌萎缩侧索硬化（amyotrophiclateralsclerosis，ALS）是一种选择性累及上、下运动神经元的慢性进行性致死性神经系统变性疾病，其病因和发病机制尚不清楚。

Amyotrophic lateral sclerosis ( ALS ) is a fatal neurodegenerative disease characterized by progressive and selective death of upper and lower motor neuron , the etiology and pathogenesis are still unclear .

肌萎缩侧索硬化症累及部位包括上、下运动神经元以及皮质脊髓束。

Amyotrophic lateral sclerosis ( ALS ) involves upper neuron , lower neuron and corticospinal tract .

目的观察实验性自身免疫性灰质病模型中上、下运动神经元磷酸化神经丝表达的特征。

Objective To observe the expression of phosphorylated neurofilaments of the upper and lower motor neurons in experimental autoimmune gray matter disease ( EAGMD ) animal model .