空泡变性

保存24h后，肝细胞出现肿胀、空泡变性及点状坏死，肝窦内皮细胞肿胀、变性，并开始向窦腔内脱落。

After 24 hours , hepatic cells swelled and occurred vacuolar degeneration and punctiform necrosis .

透射电镜观察可见SIRS大鼠心肌细胞线粒体排列紊乱，空泡变性，肌纤维水肿。

Under the electron microscope , the mitochondria in myocardium cells revealed structural disorders with vacuolar degeneration and edematous myocardial fibers in all the SIRS rats .

脂肪肝（Fattyliverdisease）是一种鱼类常见的代谢性疾病，以肝细胞出现空泡变性、脂滴沉积为主要特征。

Fatty liver is a common metabolic disease for fish .

两组相比，无统计学意义。病理学检查：B组大部分近曲小管上皮细胞严重浊肿，胞浆疏松，有空泡变性，部分近曲小管上皮细胞坏死。

On histopathology , in group B , the epithelial cells showed severe degeneration changes in proximal convoluted tubules .

结果1高眼压缺血2h后神经节纤维层开始发生空泡变性。

RESULTS ① Neuroganglion fibrous layer showed vacuolation degeneration when ocular hypertension was maintained to 2 h.

肾脏形态变化：B组6h后表现为肾小球GBM断裂、上皮细胞足突融合，肾小管上皮细胞线粒体空泡变性；C组肾损伤明显减轻。

After 6 hours , under electron microscope the renal glomeruli GBM fractured , epithelial cell foot processe obviously fused , and large quantities of mesangial cell mitochondria vacuole degenerated in group A.

50μmol浓度的UA和OA处理48小时，可见部分细胞空泡变性，细胞碎片少见。

Forty eight hours after 50 μ mol of UA or OA treatment , part of the cells degenerated with vacuoles , but fragments of cells were rarely seen .

HE染色可见药物处理6h，肝组织即呈现充血，碎片状坏死和大量炎性细胞浸润.24h肝细胞开始空泡变性、肿胀，并见大片坏死及中性粒细胞浸润。

Congestion , piecemeal necrosis and infiltration of inflammatory cells appeared at 6 h , and cellular vacuolar degeneration , engorge-ment , focal necrosis and neutrophil infiltration were observed at 24 h.

电镜下证实：GC+DENA组肝窦内枯否细胞（KC）胞浆内含有大量电子致密物或细胞空泡变性及坏死；

Under an electron microscope it was observed that the cytoplasm of KC in the GC + DENA group contained a large number of electron dense granules or KC showed vacuolar degeneration or necrosis ;

目的研究三氯化铝对学习记忆及脑神经细胞数和海马颗粒空泡变性（GVD）的影响。

Objective To study the effects of aluminum trichloride on learning memory and the numbers of cerebral neuron and granulovacuolar degeneration ( GVD ) in the hippocampus of rats .

结果证实，应用GTW或CsA后大鼠移植肾肾长明显减少，肾重明显减轻，肾小球基底膜增厚，肾小管空泡变性、坏死及间质水肿明显减轻；

It was shown that the length and the weight of kidney were markedly decreased in the rats treated with GTW . The vacuolar degeneration and necrosis in renal tubules and interstitial edema could be dramatically improved in the rats treated with GTW .

电镜下观察SD5d组CA3区锥体细胞形态不规则，结构疏松，髓鞘板层分离、线粒体肿胀、空泡变性。

In section of hippocampus from rats of SD 5 d group , form anomalous , structure rarefaction , and intercellular space augmentation of pyramidal cells and myelinic degeneration as lamellar structure , swelling and vacuolation of mitochondrion were observed .

肝水肿，狄氏间隙增宽，肝细胞空泡变性及坏死；

The liver showed edema , vacuolar degeneration and foci necrosis .

肾小管上皮细胞空泡变性，肾小球萎缩；

Vacuolar degeneration in the renal tubular epithelium and the liver cells ;

在感染过程中，被感染的神经元逐渐出现空泡变性，进而溶解、死亡。

During the infection , the neurons gradually showed vacuolation and death .

大脑皮层神经细胞有细胞空泡变性。

The neurocyte of cerebral cortex shows vacuolation degeneration .

病理形态学观察，高钼组雏鸡肝细胞呈现不同程度的颗粒变性和空泡变性。

Pathologically , hepatocytes in high Mo groups showed granular degeneration and vacuolar degeneration .

肝细胞示浊肿及空泡变性，并有灶性蜂窝状病变形成；

Liver cells showed cloudy swelling and vacuolization as well as honeycomb-like lesion formation ;

目的指示球形幽门螺杆菌致细胞空泡变性毒力的变异情况。

Aim To reval the virulence variation related to vacuolating cytotoxicity of coccoid Helicobacter pylori .

上皮的变化包括棘细胞增生，空泡变性，不全角化，过度角化和坏死等。

Epidermal changes consist of acanthosis , vacuolar degeneration , parakeratosis , hyperkeratosis and necrosis .

间变性大细胞性淋巴瘤病理切片可见癌组织大片坏死，及癌巢间大片空泡变性；

Anaplastic large cell lymphoma Pathologic observation , the tumor cells were necrosis and vacuole degeneration .

皮层下细胞存在液化坏死，皮层细胞空泡变性及坏死；

Liquefaction necrosis in subcortical cells ;

深龋组前期牙本质层变薄，成牙本质细胞空泡变性，见单层扁平的成牙本质样细胞，胞浆及核染色强阳性。

And odontoblast-like cells , which exhibited flat and aligned mono-layer , were observed and stained positive .

病理组织学观察可见脑、心脏、肾、脾、睾丸和卵巢等组织发生明显空泡变性。

Vacuolar degeneration was checked in brain , heart , kidney , spleen and ballocks or ovaries .

肾脏病理学显示肾小球病变较严重，肾小球系膜增宽，肾小管内皮细胞排列紊乱，空泡变性。

The pathology shows the glomerular destroyed , membrane widen , tubular intracellular cell disorders and vacuolization .

组织病理学变化：肝脏，肝细胞空泡变性，胆管增生。肝小叶内和汇管区有小灶性的浆细胞浸润和嗜酸性粒细胞浸润，及后期肝细胞极度肿胀；

Histopathology transformations : Liver , vacuolar degeneration and swollen in hepatocytes , small bile ducts proliferated .

结果光照后12小时，视杆细胞外节出现少量空泡变性；

Results After 12 hour light exposure , slight vesiculation was observed in the rod outer segment of the retinas .

病理观察：A组各组织结构清晰，未见水肿、空泡变性、浊肿及坏死。

Pathologic changes : In group A , the tissue structure is clear without edema , vacuolar degeneration , cloudy swelling and necrosis .

病理组织学观察实验组大鼠心肌组织可见细胞空泡变性，排列松散，结构紊乱，间质胶原纤维数量较少。

Histopathology of the hearts in LPS group shoued vacuolar degeneration , arranged in loose , structural disorder , interstitial collagen fibers decreased .

肝细胞、肾小管上皮细胞、心肌细胞颗粒变性和空泡变性；

At histopathological level , the hepatocytes , epithelial cells of renal tubule and cardiac muscle cells mainly showed granular and vacuolar degeneration .