败血症性休克
- 网络septic shock
![败血症性休克](../cache/1423790.jpg)
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心源性和败血症性休克的微循环变化
Microcirculatory alterations in cardiogenic and septic shock
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发烧、寒战和局部感染症状提示败血症性休克,特别是免疫代偿病人。
Fever , chills , and focal signs of infection suggest septic shock , particularly in immunocompromised patients .
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由于细菌毒素的作用,尤其是内毒素,败血症性休克的促炎症性反应强于其他休克。
Septic shock may be more proinflammatory than other forms because of the actions of bacterial toxins , especially endotoxin .
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败血症性休克常发生在细菌感染后,以机体免疫系统释放出大量的致炎(炎症前)细胞因子为特征。
Septic shock often follows a bacterial infection , and is characterized by the overwhelming release of pro-inflammatory cytokines by the body 's immune system .
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重度骨髓抑制易并发败血症、感染性休克等严重时可导致死亡。
Severe bone marrow suppression may predispose to sepsis , septic shock can be fatal in severe .