缓激肽

缓激肽B1受体在卡托普利抑制心脏成纤维细胞增殖中的作用

Bradykinin b_1 receptor antagonist reduces the antiproliferative effects of captopril on cardiac fibroblasts in neonatal rats

缓激肽B2受体拮抗剂对致敏豚鼠模型咳嗽反应性的影响

The effect of bradykinin B_2 receptor antagonist on cough reactivity in a sensitized guinea pig model

残肾大鼠主动脉重塑及缓激肽B2受体

Aortic Remodeling and BKB_2 Rs mRNA Level in Rats with Remnant Kidneys

大鼠残肾模型肾脏组织学和缓激肽B2受体mRNA表达的变化

Renal Histological Changes and Down-regulation of Bradykinin B_2 Receptor mRNA in Rat Remnant Kidney Model

缓激肽B2受体在缬沙坦抑制心肌细胞肥大中的作用研究

Role of bradykinin B_2 receptor in AT_1 antagonist inhibition of Ang ⅱ - induced cardiomyocyte hypertrophy in neonatal rats

连锁分析血管紧张素转换酶及缓激肽B2受体基因多态性与血管紧张素转换酶抑制剂所致咳嗽的关系

Linkage Analysis of the Association of Genetic Polymorphisms of Angiotensin Converting Enzyme and Bradykinin B_2 Receptor with ACE Inhibitors-Induced Cough

缓激肽诱导胶质瘤细胞内cAMP反应元件结合蛋白磷酸化的实验研究

Phosphorylation of cAMP response element binding protein in glioma cells induced by bradykinin

缓激肽灌注组的NOS活性显著高于对照组。

NOS activity was significantly increased in bradykinin perfusion group comparing with control group .

ATP敏感性钾通道蛋白在缓激肽选择性开放血肿瘤屏障中的表达

The Expression of ATP-Sensitive Potassium Channel Protein in Bradykinin Induced Blood Brain Tumor Barrier Permeability Increase

缓激肽（BK）和血管紧张素II（ANGII）是参与血管紧张性调节的重要的肽。

Bradykinin and angiotensin ( Ang ) II are important peptides involved in the regulation of vascular tone .

缓激肽B2受体在大鼠C6脑胶质瘤细胞上的表达

The Expression of Bradykinin B2 Receptor in the Rat C6 Cerebral Glioma Cell

缓激肽B2受体在脑胶质瘤患者肿瘤组织中的表达及临床意义

Expression of bradykinin B2 receptor in brain glioma tissue and its clinical significance

缓激肽可抑制PDGF的作用。

The effects of PDGF were inhibited by bradykinin .

缓激肽对PDGF诱导的肾小球系膜细胞增殖的影响

Inhibition of bradykinin to PDGF-induced mesangial cells proliferation

脑肿瘤细胞的B2受体表达水平决定缓激肽诱发之血脑屏障开放的程度

The expressing level of B2 receptor mediated the selective opening of blood-tumor barrier by bradykinin

在介导这一炎症特别是神经源性炎症过程的炎性介质中，缓激肽、P物质、速激肽在哮喘的发病机理中起重要作用。

Among the mediators implicated in the genesis of this inflammatory process , bradykinin > tachykinin and substance P are considered to play important roles in the pathogenesis of asthma , especially in neurogenic inflammation .

目的探讨缓激肽（BK）对大鼠尾动脉活动产生的影响，以及BK受体在大鼠尾动脉的种类分布。

Objective To observe the influence of bradykinin on rat tail arteries .

小剂量缓激肽对大鼠脑微血管内皮细胞和C6细胞钙激活钾电流的作用

Effects of low dose bradykinin to calcium-activated potassium currents on rat 's brain microvascular endothelial cells and C6 cells

目的：在体外探讨甲硫氨酸脑啡肽（MEK）与缓激肽（BK）的相关性。

Objective : To explore and discuss the correlation between MEK and BK in vitro .

结论小剂量的缓激肽通过抑制脑微血管内皮细胞和C6细胞的钙激活性钾电流，引起细胞的去极化。

Conclusions Bradykinin can depolarize rat 's brain microvascular endothelial cells and C6 cells through the inhibition of calcium-activated potassium channels .

可完全抑制缓激肽动脉注射所引起的痛反应，其ED（50）为1.3mg/只。

Can fully inhibit the mice response induced by bradykinin ( 0.5ug/per mouse i.a. ) and the ED50 of A - ⅲ is 1.3mg/per mouse .

采用放射免疫法检测应用缓激肽前后C6胶质瘤细胞培养液中TNF-α的含量。

Detection of TNF - α concentration in the C6 culture medium , using radioimmunoassay before and after bradykinin treatment . 5 .

脑缺血后期，缓激肽B2受体能够抑制缺血导致的细胞凋亡。

At the later stage after reperfusion bradykinin B2 receptor can inhibit the cell apoptosis induced by ischemic damage . 3 .

目的通过在体兔心肌缺血再灌注模型探讨缓激肽（BK）对缺血再灌注心肌能量代谢的延迟性影响。

AIM To investigate the delayed effect of bradykinin on energy metabolism of ischemia reperfusion myocardium in rabbits .

研究提示：1、脑缺血早期，缓激肽B1和B2受体均能加重缺血后组织损伤。

At the earlier stage after reperfusion , both bradykinin B1 and B2 receptors can aggravate the tissue damage of cerebral ischemia .

缓激肽对肾系膜细胞和成纤维细胞有丝分裂的影响及与ERK途径相关性

Regulatory Effects of Bradykinin on Mitogenesis in Mesangial Cells and Renal Interstitial Fibroblasts and the Correlation with ERK1 / 2 Phosphorylation

结论缓激肽作用于血瘤屏障后，激活了HSF1并促进了hsp70蛋白的表达。

Conclusion Bradykinin activated HSF1 and induced the expression of HSP70 in tumor tissues .

褐煤腐殖酸肥料的养分缓释机理及其肥效研究缓激肽对PDGF诱导的肾小球系膜细胞增殖的影响

Nutrient Slow-release Mechanism of Humic Acid Fertilizer Extracted from Brown Coal and Its Effects ; Inhibition of bradykinin to PDGF-induced mesangial cells proliferation

Y-27632抑制缓激肽选择开放血肿瘤屏障的研究

Y-27632 inhibits the opening of blood tumor barrier caused by bradykinin

目的：研究给药时间与大鼠C6胶质瘤细胞缓激肽（bradykinin，BK）B2受体内化的关系。

OBJECTIVE : To study the relationship between drug treatment time and bradykinin B 2 receptor internalization in the rat C6 glioma cells .