线粒体基质

结果TGFβ2和bFGF主要集中于肾小管上皮细胞的线粒体基质内，在其它部位有极少量的分布。

Result TGF β 2 and bFGF were mainly concentrated in the mitochondrial matrix of renal tubular epithelial cells , but rarely found in other areas .

RS在植物细胞中的细胞核、线粒体基质空间、微体（过氧化物酶体）、叶绿体类囊体膜中的分布概率分别为：88%、36%、31.3%、28%。

The distribution probability of RS was respectively 88 % , 36 % , 31.3 % , 28 % in nucleus , mitochondrial matrix space , microbody ( peroxisome ) , chloroplast thylakoid membrane .

线粒体基质蛋白Hsp70、Hsp60的表达、纯化、鉴定及抗体制备

Expression and identification of mitochondrial heat shock protein 70 , 60 , and its antibody

结果：在所测试的细胞株中，线粒体基质内表达Dm-dNK具有较高的酶活性，转染肿瘤细胞对araC、araT及dFdC的敏感性增加。

RESULTS : The mitochondrial Dm-dNK was enzymatically active and the overexpression of the enzyme in the tested cell lines resulted in an increased sensitivity to the cytosine nucleoside analogs such as araC , araT and dFdC .

力竭性游泳对大鼠心肌线粒体基质游离Ca~（2+）及Na~+/Ca~（2+）交换的影响

Effect of Exhaustive Swimming on Myocardial Mitochondrial Matrix Free Ca2 + and Na + / Ca2 + Exchange in Rats

对于线粒体上基质的反应进行模拟，模拟本身不仅是为反应建模而是将反应的机理引入到模型中，达到人工生命与图形学的结合。

Mitochondrial matrix for the analysis of responses in this part of the mitochondrial matrix for modeling the reaction mechanism but the reaction is introduced into the model to a combination of artificial life and graphics .

大豆下胚轴线粒体在呼吸基质存在下，显著地增加了肾上腺素氧化速率，这种氧化速率能为外源SOD抑制，表明线粒体呼吸时产生分子氧的单电子还原成O2（？）

The rate of epinephrine oxidation in soybean hypocotyl , mitochondria was increased by the presence of respiratory substrates , the oxidation rate was lowered by the addition of SOD .

5h缺血骨胳肌糖原颗粒减少，线粒体肿胀、基质减少，嵴断裂，肌浆网扩张。

Skeletal muscle of 5 h ischemia showed diminished glycogen granules , swelling of mitochondria , clearing of mitochondrial matrix , breakage of mitochondrial crista and dilatation of sarcoplasmic reticulum .

近来，越来越多的证据表明存在线粒体一氧化氮合酶（mitochondrialnitricoxidesynthase，mtNOS），它与线粒体内膜的基质面结合，通过钙敏感性途径产生一氧化氮（NitricOXide，NO）。

Increasing evidences have shown the existance of a mitochondrial nitric oxide synthase ( mtNOS ), which binds to the matrix face of the mitochondrial inner membrane and produces nitric oxide ( NO ) through a Ca ~ ( 2 + ) sensitive pathway .

MPT导致线粒体膜电位下降、氧化磷酸化解耦联、ATP损耗、小分子溶质和离子在细胞质和线粒体基质间的平衡。

MPT causes dissipation of the mitochondrial membrane potential , uncoupling of oxidative phosphorylation , ATP depletion , and equilibration of small solutes and ions between the cytosol and the mitochondrial matrix .