水潴留

  • 网络water retention
水潴留水潴留
  1. 抗利尿激素在阿霉素肾病大鼠水潴留机制中的作用

    Antidiuretic hormone didn ′ t cause water retention in adriamycin nephrotic rats

  2. 水潴留对很多女性来说都是个问题,尤其是在经期前。

    Water retention is a problem for many women and it is often worse just before a period .

  3. 钠水潴留和肌酐水平高是高血压伴肾功能不全的血压不达标的独立危险因素。

    Excess sodium - water volume is pivotal factor in hypertension with renal insufficiency .

  4. 目的研究肝功能损害与钠水潴留的关系。

    AIMS To investigate the relationship between the liver function - al impairment and sodium and water retention .

  5. 结论:钠曲线和超滤曲线合理使用可明显减少血透急性并发症,减轻水潴留;

    Results : Reasonable use of sodium curve and ultrafiltration curve can obviously reduce the acute complications of hemodialysis .

  6. 尿毒清颗粒预防慢性肾功能衰竭病人钠水潴留临床观察

    Niao Du Qing Granules : Clinical Observation of Its Preventive Effects on Sodium and Water Retention in Chronic Renal Failure Patients

  7. 结果表明:微循环障碍、代谢产物含量升高和水潴留是尿毒症肾阳虚的病理基础;

    The results showed that the pathologic basics of uremia with deficiency of kideny-yang may be microcirculatory disturbance , increased metabolites and water retention .

  8. 本研究发现,慢性心力衰竭患者入院时钠水潴留积分与血肌酐有显著性正相关关系。

    The study found that chronic heart failure patients admitted to hospital a significant positive correlation between the sodium and water retention points and serum creatinine .

  9. 如果你吃盐太多,你就会因为水潴留而超重大约1.8公斤。

    If you have a high salt intake , you could be carrying around an extra 1.8kg ( 4lb ) in excess weight due to water retention .

  10. 此时检查急性气喘病患者的尿比重,将发现他们都处于水潴留与尿比重高的状态。

    A check of urine specific gravity at the time of admission of an acute asthmatic will invariably show a state of severe water retention with high urinary specific gravity .

  11. 水潴留可能是因为你饮水不足,或者吃盐太多,但也可能是因为引发很多经前期症状的血糖波动而导致的。

    Water retention can be caused by not drinking enough water and also too much salt but it can also be caused by the same blood sugar swings that trigger many of the pre-menstrual symptoms .

  12. 结论:高原肺心病重度水肿的发生除与右心功能不全有关外,还与严重低氧血症和高碳酸血症引起的钠水潴留有关。

    Conclusion : The occurrence of severe edema in CCP patients at high altitude is related not only to right heart dysfunction as well as to retention of sodium and water induced by severe hypoxemia and hypercapnia .

  13. 钠水潴留的大鼠均表现为腹部膨隆,尸检发现腹部大量腹水,肠管胀气,推测应均为钠水潴留引起的心衰致死。

    Rats with Sodium and water retention all showed distended abdomen , the autopsy found that the abdomen with massive ascites , intestinal flatulence , with speculating for heart failure death be caused by sodium and water retention . 4 .

  14. 糖皮质激素具有保钠排钾的作用,易导致钠水潴留,以及引起肝脏肿大和哮喘等不良反应,不同的激素引起以上不良反应的程度不同。

    Glucocorticoids have a role to save the sodium and excrete potassium , easily lead to sodium retention , as well as the adverse reactions such as liver swelling and asthma . And different hormones cause adverse reactions in varying degrees .

  15. 但由于长效激素有易导致钠水潴留的可能,若再加入碱化溶液更容易导致体内电解质及酸碱平衡的紊乱,从而引发钠水潴留或使钠水潴留的情况加重。

    However , due to long-term hormone maybe easily leading to sodium and water retention , if we join the alkalization of the solution will more easily lead to balance disorders in electrolyte and acid-base of the body , causing sodium and water retention or aggravating circumstances .

  16. 慢性肾炎患者水钠潴留与PET相关性研究

    Correlation between PET and Retention of Water and Sodium of Chronic Glomerulonephritis

  17. 为研究慢性肾炎患者水钠潴留与血浆内皮素(PET)的相关性,检测了伴或不伴水钠潴留慢性肾炎患者PET和24h尿蛋白排泄量。

    In order to study the correlation between retention of water and sodium of chronic glomerulonephritis and plasma endotheliolysin ( PET ) .

  18. 与常规蛋白组相比,酮酸组与低蛋白组KT/V、Ccr、尿量均升高(P<0.05),而酮酸组与常规蛋白组的水钠潴留发生率无统计学意义(P>0.05)。

    Levels of KT / V , Ccr and residual urine volume were significantly higher both in ketoacid group and low protein diet group ( P < 0.01 ) .

  19. 治疗3wk时两组均未见明显低钾及水钠潴留的发生。

    Neither remarkable hypopotassaemia nor water-sodium retention were found after treatment for 3 weeks .

  20. 坎离煎和开搏通均能明显改善心衰兔水钠潴留状态。

    Both of them obviously improved the status of water-sodium retention .

  21. 人体水钠潴留性疾病心钠素形态学研究

    Morphological study on atrial natriuretic peptide in patients with sodium and water retention

  22. 结论随着肾功能的恶化,低肾素型患者的比例升高提示钠离子介导的水钠潴留成为高血压的主要机理。

    Conclusion Volume expansion mediated by sodium becomes a major mechanism of hypertension in renal insufficiency .

  23. 复方甘草酸苷可迅速降低氨基转移酶水平,并减少水钠潴留和低血钾的发生。

    Compound glycyrrhizin could rapidly lower the aminopherase ( AT ) level and reduce the incidence rates of water-sodium retention and hypopotassemia .

  24. 结果:从疼痛的病因方面可以总结归纳为:外感六邪、五气过抑、气分受伤、水液潴留4个方面;

    RESULTS : It was summarized as 4 etiological factors of pain , named six exogenous pathogenic factors , retarded qi circulation in five organs , injury of qi system , retention of fluids ;

  25. 目的:探讨黄芪对阿霉素肾病大鼠水钠潴留的影响及其可能的作用机制。

    Objective : To observe the effects of astragalus on water sodium retention of adriamycin-induced nephrotic syndrome rats and explore its mechanism . Methods : Nephrotic syndrome was induced by the intravenous injection of ADR .

  26. 而实主要是湿浊瘀血贯穿疾病始终。消渴日久,伤阴耗气,阴损及阳,脾肾俱虚,血脉瘀阻,水湿潴留,浊毒内停为本病之基本病理过程。

    The basic pathologic process is injury of yin and qi , then extent that yang-qi is involved , deficiency in spleen and kidney , obstruction of blood stasis , retention of dampness , stagnation of toxic turbid in human body .

  27. 并监测患者平均动脉压、腹膜透析超滤量、残余尿量、水钠潴留情况。

    Examinations of RRF , mean arterial pressure ( MAP ), peritoneal ultrafiltration ( UF ), residual urine volume ( RUV ) and the status of water-sodium retention were performed at baseline , median and at the end of the follow-up .

  28. 心力衰竭患者水钠潴留期间,中心静脉压升高,后者传至肾静脉和肾脏,导致肾功能障碍,可能是慢性心衰患者肾功能下降的发病机制之一。

    Heart failure in patients with sodium retention , elevated central venous pressure , which spread to the renal vein and kidneys , causing renal dysfunction may be one of the pathogenesis of chronic heart failure patients with reduced renal function .

  29. 引起肝病患者水、盐潴留和腹水的原因可能不止一个。

    There are probably several factors responsible for water and salt retention and ascites in hepatic disease .

  30. 它的主要临床表现为肾功能进行性减退、水以及电解质的潴留、酸碱平衡的失调。

    Its main performance for renal progressive decline , water and electrolyte retention , acid-base disorders .