慢性胃炎

慢性胃炎患者124例（C组）分别进行内镜检查，比较3组患者胃镜下表现。

Group C. 124 patients with chronic gastritis .

慢性胃炎中幽门螺杆菌与生长抑素、胃泌素含量及D、G细胞密度的关系

Helicobacter Pylori ( Hp ) and Contents of Somatostatin and Gastrin in Chronic Gastritis

儿童过敏性紫癜反复发作与慢性胃炎及HP感染的关系探讨

Henoch-Schonlein Purpura Recurrent and Chronic Gastritis and HP Infection of Children

讨论HP感染是小儿慢性胃炎的主要病因；

Conclusion The main cause of gastritis is Hp infection in children .

目的：建立用于评价药物疗效的较理想的Hp相关性慢性胃炎大鼠模型。

Objective : To establish a better rat model of H.pylori-associated chronic gastritis .

大鼠Hp相关性慢性胃炎模型的制做

Study of Rat Model of Hp-associated Chronic Gastritis

幽门螺旋杆菌感染的慢性胃炎胃窦粘膜内NF-κB和TGF-α表达及其意义

Significance and expression of nf - κ b and tgf - α in the antral mucosa of Helicobacter pylori infected patients with chronic gastritis

慢性胃炎及消化性溃疡诱生型一氧化氮合酶表达和Hp感染的意义

Expression of inducible nitric oxide synthase and Hp infection in chronic gastritis and peptic ulcer

大量研究资料证明Hp是人慢性胃炎和消化性溃疡的特异性病原体，长期Hp感染与人胃癌和胃B细胞淋巴瘤的发生密切相关。

Many investigation data demonstrated that H.pylori is the specific pathogen responsible for human chronic gastritis and peptic ulcer .

SOD在Hp感染阳性的胃癌、胃粘膜不典型增生及慢性胃炎中表达的研究

Study of SOD expression in stomach cancer , gastric atypical hyperplasia , chronic gastritis patients with Hp

提示CP感染与慢性胃炎和消化性溃疡的发生有一定的因果关系。

CP is suggested to be etiologically associated with chronic gastritis and peptic ulceration .

幽门螺旋杆菌对慢性胃炎患者粘膜内D细胞及生长抑素mRNA表达的影响

The effect of Helicobacter pylori infection on the expression of D cell and Somatostatin mRNA in the patients with chronic gastritis

幽门螺杆菌（Hp）是一种参与多种胃、十二指肠疾病的重要人类病原体，可引起慢性胃炎和消化性溃疡，并与胃癌密切相关。

Helicobacter pylorus is a major human pathogen associated with severe gastroduodenal diseases , including ulcers and cancers .

慢性胃炎肝郁证大鼠模型表征、胃黏膜病理表现与血清NO、ET的变化趋势一致。

Chronic gastritis rat model of liver-depression syndrome characterization , gastric mucosa pathological manifestations and serum NO , ET similar changing tendency .

平胃舒胶囊能提高慢性胃炎大鼠的胃液酸度和胃蛋白酶活力，与模型组相比有差异（P0.05）。

Compared with model group , Ping-wei-shu Capsule could increase gastric acidity and stomach peptic activity of rats which having chronic gastritis ( P0.05 ) .

方法：将62例IDA伴Hp阳性慢性胃炎患者，分为A、B、C、D4组。

Method : Divided the 62 IDA patients with Hp-positive chronic gastritis into four groups ( A , B , C , D ) .

【结论】士兵慢性胃炎患者红细胞免疫功能低下，HP感染是导致其红细胞免疫功能低下的原因之一。

[ Conclusion ] The immune function of RBC is low in the patients with chronic gastritis , which is due to the infection of Hp .

pylorin阳性者，母体血VacAIgG阳性率为86%，慢性胃炎患者阳性率为66%，显著高于胃不适患者（P分别<0.005及<0.001），但二者之间无显著性差异。

The positive rate of VacA IgG suffered from gastric and duodenal ulcer , chronic gastritis was higher than that of gastric discomfort ( P < 0.005 , P < 0.001 ), and no marked difference between them .

目的：探讨慢性胃炎、胃溃疡及胃癌患者血浆内皮素（ET）水平的变化在其发病机制及治疗中的临床意义。

Objective : To explore the clinical meanings of the endothelin levels in the serums of the patients with chronic gastritis , gastric ulcer and carcinoma of stomach .

幽门螺杆菌（HelicobacterPylori，H.pylori）感染与人类慢性胃炎、消化性溃疡、胃癌的发生密切相关。

H. pylori ( Helicobacter pylori ) infection is closely associated with occurance of human chronic gastritis , peptic ulcer disease and gastric adenocarcinoma .

结论：采用H.pylon及MNNG的综合万法制备大鼠Hp相关性慢性胃炎模型，能反映Hp柏关性慢性胃炎特点，可作为探讨即相关性慢性胃炎治疗策略的研究工具。

Conclusion : The model can model - the characters of H.pylori-associated chronic gastritis , so it can be used to study effect of therapy .

方法：收集134例GERD患者与182例慢性胃炎患者的胃黏膜组织和流行病学资料。

METHODS : Gastric mucosa from 134 patients with reflux esophagitis and 182 controls with chronic gastritis were collected .

结果：Hp相关性慢性胃炎中脾胃湿热型占62.96%（P<0.05）；

Results : Among the patients with DHSS , 62.96 % were Hp associated chronic gastritis ( compared with that in the group of SAS , P < 0.05 ) .

结论在治疗HP阳性的慢性胃炎引起的IDA过程中，抗HP治疗能明显提高铁剂的治疗效果。

[ Conclusion ] It can increase the therapeutic effect of ferrous succinate when anti-HP was given in the IDA patients caused by chronic gastritis with HP positive .

PU伴随之慢性胃炎病变重，其重度炎症占7727%。

PU ( 77 27 % ) associated with severe chronic gastritis , but the chronic atrophic gastritis ( CAG ) and intestinal epithelial metaplasia were not often found .

pylori阴性的慢性胃炎胃窦黏膜COX2表达，比较根除H.pylori前后胃窦黏膜COX2表达的变化。

The expressions of COX-2 in the antral mucosa were compared before and after H.pylori eradication .

方法对31例有长期IDA病史、病因检查只发现有Hp阳性慢性胃炎的患者，进行根除Hp治疗；

Methods Eradication therapy was used on thirty one patients with a long history of iron deficiency anemia in which Hp positive gastritis was the only pathologic finding detected .

因此，控制CP感染对阻断和延长慢性胃炎癌变的发生有重要意义。

Therefore , it is very important to con - trol the CP infection for the decrease of the incidence of chronic gastritis , known as precursor of gastric cancer .

结果Hp感染蒙古沙鼠后，胃黏膜出现慢性胃炎、肠化生及异型增生改变，而对照组胃黏膜基本正常，Hp感染组肠化生及异型增生发生率明显高于对照组（P<0.05）。

Results The incidences of intestinal metaplasia and dysplasia in the gastric mucosa with H. pylori infection were significantly higher than those without H. pylori infection ( P < 0 . 05 ) .

大多数研究表明在慢性胃炎胃粘膜萎缩肠化生异型增生胃癌这一癌变模式中，H.pylori可能起着先导作用。

Many researches showed that there was the mode of " chronic gastritis-gastric atrophy intestinal metaplasia-dysplasia-gastric cancer " and H. pylori played a leading role .