小胶质细胞

小鼠出现强迫症状态与免疫系统中的小胶质细胞有关。

The mouse condition is related to immune system cells called microglia .

小胶质细胞在神经病理性疼痛中的作用

Effects of microglia on neuropathic pain

核因子-κB参与马桑内酯激活小胶质细胞化学研究

Immunocytochemical Study of Nuclear Factor - κ B in Microglial Cells Activated by Coriaria Lectone

脑出血后小胶质细胞活化与神经元DNA损伤的关系

The relationship between activation of microglia and damage of nerve cell DNA after ICH

枸杞多糖对AD模型小鼠海马齿状回神经发生和小胶质细胞的影响

Effects of LBP Treat AD Mice on Hippocampal Dentate Gyrus Neurogenesis and Microglia

与亨廷顿病严重程度相关的小胶质细胞激活：一项临床及PET研究

Microglial activation correlates with severity in Huntington disease : A clinical and PET study

因此，抑制小胶质细胞过度活化减少炎症反应对神经元的毒性是治疗PD的策略之一。

Thus , anti-inflammation has become one of the therapeutic strategies of PD .

本文就激活的小胶质细胞在AD脑内炎性反应中的双重作用予以综述。

This article reviews the dual role of microglial cells in the immune inflammatory reaction of AD.

CA能神经终末与垂体细胞和小胶质细胞之间存在着密切关系，甚至可建立突触样连接。

The close relationship even synapse-like contacts existed between the CA-ergic endings and pituicytes as well as microglial cells .

IgG刺激诱发的小胶质细胞Toll样受体4表达及细胞因子分泌

Toll-like receptor 4 expression and cytokine secretion in microglial cells induced by IgG stimulation

小胶质细胞活化与rd小鼠遗传性视网膜变性

Activation of microglia in the inherited retinal dystrophy of rd mice

抑制过度活化小胶质细胞介导的神经炎症可保护神经元，具有治疗PD的作用。

Inhibiting over-activated microglia mediated neuroinflammation may show neuroprotective effects on neurons , and achieve PD treatment effects .

抗白细胞药物对大鼠脑缺血后病灶区小胶质细胞数及血清IL-1β活性的影响

Influence of antileukocytic drugs on microglia and il-1 β after brain ischemia in rats

小胶质细胞上ATP敏感性钾通道的表达及其对神经炎性反应的调节

The Expression of ATP-sensitive Potassium Channels in Microglia and Its Regulatory Effects on Microglia-induced Neuroinflammation

⒊PD病人脑中存在小胶质细胞活化及伴随的炎性过程；

Activated microglia , which accompany an inflammatory process , are present in the nigro-striatum of the PD brain ;

目的：建立稳定高产的新生LONGEVANS大鼠视网膜小胶质细胞（retinamicrogliacellRMG）体外培养、分离的方法。

· AIM : To establish a culture method of Long Evans neonate rat retinal microglia cells ( RMGs ) in vitro .

雷公藤甲素对LPS诱导的神经炎症中星形胶质细胞和小胶质细胞激活的抑制作用

The inhibitory effects of triptolide on activation of astrocytes and microglia in neuroinflammation induced by LPS

LPS诱导小鼠脑小胶质细胞的激活及Tomatolectin的表达变化

Lipopolysaccharide induced microglia activation and change of expression of tomato lectin in microglia of mouse brain

黑质内注射脂多糖对不同年龄大鼠DA能神经元和小胶质细胞活性的影响

The effects of nigral lipopolysaccharide administration on dopaminergic dysfunction and glial cell activation in young and aged rats

4.2PC-PLC通过调节iNOS活性参与介导小胶质细胞的活化。

4.2 PC-PLC regulated the activation of microglia by mediating the activition of iNOS .

目的：探讨白藜芦醇对脂多糖（LPS）诱导的小胶质细胞激活时所分泌的一氧化氮（NO）和肿瘤坏死因子-α（TNFα-）的影响。

Objective : To observe the effects of resveratrol on LPS-induced NO and TNF - α production in activated microglia .

MTT检测G-CSF对小胶质细胞活性的影响。

MTT assay was used to evaluate the effect of G-CSF on the microglia activity .

方法：采用体外LPS诱导小胶质细胞活化模型，用ELISA法测定细胞外液中NO和MCP-1的含量。

Methods : Using LPS-induced microglia activation model and proinflammatory cytokines NO and MCP-1 were measured by ELISA .

黑质内注射脂多糖对小胶质细胞MHCⅡ表达的影响

Effects of Microinjection of LPS into Substantia Nigra on the Expression of Microglia MHC ⅱ

小胶质细胞表达B7-H1及LPS刺激对其表达影响的研究

Microglial expression of B7-H1 and its change after LPS stimulation

在国内首次应用BV一2细胞株作为体外小胶质细胞模型进行AD的炎症相关性研究，对掌握该细胞株对Ap的反应特性提供了相应的实验室资料。

As an effective model in vitro , BV-2 microglia has important value in the study of Alzheimer 's disease .

米诺环素可抑制小胶质细胞增殖及MMP-9的表达，从而可能延长UK治疗时间窗。

Minocycline can inhibit the proliferation of microglia and the expression of MMP-9 , and probably extends the therapeutic time window of UK .

目的探讨脂多糖（LPS）诱导小胶质细胞（Mic）激活规律与黑质多巴胺（DA）能神经元变性的关系。

Objective To explore the relationship between the activities of microglia induced by LPS and the degeneration of dopaminergic neurons .

局灶脑缺血恢复期半影区小胶质细胞可塑性变化和bFGF表达规律的研究

Study on the Plasticit Change of Neuroglial Cell and the Expressing Rule of bFGF During Convalescence of Focal Cerebral Ischemia Penumbra

结论：姜黄素能够有效抑制脂多糖激活的小胶质细胞iNOS蛋白的表达以及NO的产生。

Conclusion : Curcumin could decrease the iNOS expression and suppress the production of NO in activated microglia cells .