剪接受体

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  • splicing receptor
剪接受体剪接受体
  1. 第10内含子剪接受体端杂合突变(INS10-2A>G),由父亲遗传而来。

    A heterozygous splicing muta-tion of INS 10 - 2 A > G at the acceptor site of intron 10 was originated from her father .

  2. 结论脐血瘦素浓度降低和胎盘瘦素、OBR受体表达低下与胎儿生长受限密切相关,脐血瘦素可能通过与不同剪接体的受体结合实现对胎儿生长发育的调控作用。

    Leptin in cord blood may have an important role in regulate fetal growth and development through different OB R.

  3. 结果显示,正常、炎症和恶性胃组织同时表达正常和异位剪接的CCK-B受体基因,且两种受体基因的表达量与胃癌的分化和转移无关。

    The results showed that human gastric tissues from normal , inflammatory and malignant specimens simultaneously expressed the normal and aberrant splicing cholecystokinin-B / gastrin receptor genes and the expression levels of the two receptor genes were not correlated with the differentiation and metastases of gastric cancers .

  4. 选择性剪接在Toll样受体4信号转导通路中的作用

    The role of alternative splicing in the Toll-like receptor 4 signal transduction

  5. 此对上游引物和下游引物分别位于剪接供体之前和剪接受体之后。

    In this primer pairs , splice donor and splice acceptor were located at its downstream and upstream respectively .