突触后膜

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  • postsynaptic membrane
突触后膜突触后膜
  1. 结果MG时突触后膜长度缩短,后、前膜长度比值变小,神经末端面积及其与突触后膜面积之比变小,突触后膜上的乙酰胆碱受体(AChR)减少了35.9%。

    Results The length of postsynaptic membrane decreased obviously . The ratio of the length of postsynaptic with presynaptic membrane , and that the motor nerve terminal area with postsynaptic membrane area diminished . The AChR on the postsynaptic membrane decreased by 35.9 percent as well .

  2. 随后,突触后膜开始逐渐去极化,最大去极化27.0mV。62.7min后小终板电位(mEPP)频率逐渐增高,到70.3min达到最高频率,比给药前增加了32倍。

    Hereafter postsynaptic membrane potential was depolarized gradually , and the maximum depolarization value was 27 . 0 mV . The frequency of mEPP increased markedly after 70 . 3 min MTX administered , 32 folds compared with control .

  3. S型、T型和G型突触后膜均比前膜明显增厚,呈不对称性。

    The postsynaptic membrane of S - , T - and G-types is apparently thicker than the presynaptic membrane and showed to be asymmetrical .

  4. 研究多巴胺(DA)受体对突触前膜和突触后膜Na+-K+-ATP酶活性的调节作用。

    Objective To investigate the regulation of dopamine receptors on striatal Na + , K + - ATPase in pre-and post synaptic membranes .

  5. 目的研究梭曼引起神经肌接头突触后膜持续性去极化(SD)的产生机制。

    Objective To study the mechanism underlying the sustained depolarization ( SD ) of neuromuscular junction induced by soman in isolated non-uniform stretched muscle preparation of rat diaphragm .

  6. 缺血损伤后突触后膜AMPA受体的功能变化及分子机制研究

    Study on Function Change of AMPA Receptor on Postsynaptic Membrane after Ischemic Insult and Related Molecular Mechanism

  7. OGD后突触后膜GluRs含量变化及神经元延迟性死亡研究

    Changes of GluRs on postsynaptic membrane and delayed neuron death after OGD

  8. 阿维菌素类杀虫剂主要作用于γ氨基丁酸(GABA)受体,它能促进GABA的释放,增强GABA与GABA受体的结合,使氯离子内流增加,导致突触后膜超级化。

    Avermectin insecticides can cause the release of gamma-aminobutyric acid ( GABA ) and enhance the affinity of GABA with GABA receptors , leading to chloride influx and postsynaptic hyperpolarization .

  9. 结果提示:突触后膜致密物质厚度与记忆保持的程度密切相关,而且PSD增大与出现穿孔性突触相平行。

    The results suggest that there is a correlation between the PSD and the ability of memory retention , and that the increase of the PSD is parallel with formation of perforated synapse .

  10. 腺苷是通过激活突触前膜和突触后膜腺苷A1受体而起抑制性作用,所以腺苷A1受体在促进睡眠中起重要作用。

    The adenosine exerts its inhibitory effect through activation of both pre-synaptic and post-synaptic adenosine A1 receptors ( A1R 's ) . Thus , A1 receptors play an important role in promoting sleep .

  11. 目的探时OGD损伤后神经元突触后膜GluR1~3含量变化及其在神经元延迟性细胞死亡中的作用。

    Objective To explore the changes of glutamate receptor ( GluR ) 1 ~ 3 in the postsynaptic membrane and the effects on the delayed neuron death after oxygen-glucose deprivation ( OGD ) .

  12. 肌肉特异性激酶(muscle-specifickinase,MuSK)是一种与乙酰胆碱受体相关的跨膜蛋白,在骨骼肌的发展中,MuSK是建立突触后膜时组建主要突触骨架的首要成分。

    Muscle-specific kinase ( MuSK ) is a transmembrane protein which is related with acetylcholine receptor , in the development of skeletal muscle , MuSK is a primary ingredient to establish the main synaptic skeleton of the postsynaptic membrane formation .

  13. 测量小腿三头肌肌湿重,并分别应用HE染色、Mallory三色染色的方法对失神经肌肉组织进行染色,同时应用突触后膜乙酰胆碱受体(AchR)特异性抗体对肌肉冰冻切片进行免疫荧光染色。

    Wet weight of triceps surae was measured . The denervated muscle was given HE staining and Mallory staining , while the frozen muscle biopsy tissue was given Immunofluorescence with postsynaptic membrane AchR specific antibody .

  14. 发现免疫反应阳性Vp物质主要出现在高尔基体扁平膜囊内、粗面内质网核糖体上、神经分泌粒、树突、轴突以及非对称性突触后膜内。

    It was found that Vp-like immunoreactive substance appeared mainly in the flat sacs of Golgi complex , on the ribosomes of rough endoplasmic reticulum , and in the neurosecretory granules , dendrites , axons , and in the postsynaptic membranes of the asymmetric synapses .

  15. 但是药学实验的结果却能够推论出sec-10基因突变种系的突触后膜上受体过度敏感,我们推测是由于突触后膜上离子型受体以外的其他类型受体受到了影响。

    However , the drug assay deduced the postsynaptic receptors are hypersensitive , which indicates some other kinds of receptors other than the ionotropic receptors are affected in sec-10 mutant .

  16. 电针镇痛时突触后膜厚度的变化

    The changes in the thickness of postsynaptic membrane during acupuncture analgesia

  17. 突触后膜和突触下致密小体的亚显微结构

    Submicroscopic structure of postsynaptic membrane and subsynaptic dense body

  18. 丙泊酚对中枢神经系统突触后膜受体反应性的影响及机制

    Effect and Mechanism of Propofol on Postsynaptic Receptor Reactivity of Central Nervous System

  19. 梭曼引起大鼠神经肌接头突触后膜持续性去极化的机制

    Mechanism underlying the sustained depolarization of neuromuscular junction induced by soman in rats

  20. 突触后膜致密物厚度变薄,而突触接触带的平均长度则代偿性增大。

    The average length of synaptic contact zone increase .

  21. 人参皂甙对突触后膜β受体效应影响

    Effects of Ginsenosides on the Responses Mediated by β - Receptors on the postsynaptic Membranes

  22. 多巴胺对大鼠纹状体突触前和突触后膜Na~+,K~+-ATP酶活性调节的差异

    Differential regulation of dopamine receptors on pre and postsynaptic Na + , K + ATPase in rat striatum

  23. T型终扣含有清亮球形小泡,也可见大颗粒小泡,在突触后膜下可见致密体;

    T-type contain spherical and large granular vesicles , and dense body was seen beneath the post synaptic membrane .

  24. 突触后膜上化学门控离子通道的开放比例与突触前膜电位呈正相关;

    And the opening proportion of chemical-gated ion channels on postsynaptic membrane was positively related to the potential of presynaptic membrane .

  25. 综述了丙泊酚对中枢神经系统突触后膜各类受体反应性的影响及其机制.神经元普遍存在突触前受体;

    In the present review , the authors summarized the effect and mechanism of propofol on postsynaptic receptor reactivity of central nervous system .

  26. 以上结果提示:脾气虚时神经肌肉接头传递功能将发生障碍,并且障碍可能发生在神经肌肉接头的突触后膜。

    The result indicated : Transmission function of nerve-muscle junction will be disturbed when spleen-Qi deficiency rate more obviously ( P < 0.01 ) .

  27. 突触前膜、突触后膜和突触间隙清晰,突触前终末有较多清亮的圆形囊泡,突触后膜有电子密度高的致密物质。

    Presynaptic membrane , postsynaptic membrane and synaptic cleft were clear . Presynaptic terminals had more round clearest vesicles , and postsynaptic membrane has a high density electron . 3 .

  28. 细胞移植后12周,实验组失神经肌肉表面突触素大量聚集,突触后膜形态较好,已接近正常肌肉突触后膜的形态。

    At 12 weeks after implantation , many synaptophysins gathered on the surface of denervated skeletal muscles and the form of postsynaptic membrane keep well and was similar as the one of normal membrane .

  29. 透射电镜下运动终板超微结构的变化:去神经支配后1周,突触后膜和神经末梢萎缩,初级和次级皱褶均变浅,眼肌与口肌运动终板结构相似。

    The changes of ultrastructure of MEP under electron microscope : One week after denervation , posterior member of the synapse and motor nerve ending became atrophy , and primary crease and secondary crease became shallow , and MEP structure of M.oculi and M.ocular was similar .

  30. GLU与突触后神经元膜上的受体相结合,导致钠离子和钙离子内流,引起一系列的生物化学级联反应。

    Glutamate binds to receptors on the postsynaptic neuronal membrane to activate the influx of sodium and calcium .