感染性休克

感染性休克治疗中的血糖控制及评价

Glucose control in septic shock

反义TNF（-α）寡脱氧核苷酸降低感染性休克小鼠死亡率

Antisense TNF - α oligodeoxynucleotide decreases death rate in septic shock mice

大鼠感染性休克和内毒素性休克时血浆6-酮-前列腺素F（1α）和血栓素B2的动态观察

The plasma levels of 6-keto-pGF_ ( 1 α) and TXB_2 in the rats during septic shock and endotoxic shock

结论血浆TNF水平与并发感染性休克或器官功能衰竭密切相关。

Conclusion The plasma TNF level closely correlates with the complicated infectious shock and with the organ failure .

出现感染性休克和多脏器功能不全综合征（MODS）常提示预后不良。

MODS and septic shock were the predicting of poor prognosis .

方法：通过腹腔注射LPS建立感染性休克小鼠模型。

METHODS : Septic shock mice model was induced by intraperitoneal injection of LPS .

多因素逐步Logistic回归显示，感染性休克的病死危险因素是合并器官功能衰竭数目。

In stepwise logistic regression analysis , the number of failed organ was the risk factor associated with death .

死亡原因均为合并伤伴创伤性休克或感染性休克，导致MODS。

The death causes was MODS resulted from traumatic and infections shock .

用腹腔注射脂多糖（LPS）20mg/kg制备大鼠感染性休克模型；

Septic shock was replicated by intraperitoneal injection of 20 mg / kg lipopolysaccharide ( LPS ) .

感染性休克最常见的并发症是弥散性血管内凝血（DisseminatedIntravascularCoagulation，DIC）。

One of the frequent complications of septic shock is disseminated intravascular coagulation ( DIC ) .

主要死亡原因为感染性休克、MOF、ARDS。

The main cause of death was septic shock ? MOF ? ARDS .

血浆vW因子抗原检测在儿童感染性休克中的意义

The clinical significance of von Willebrand Factor in children with septic shock

1991～2001年ICU感染性休克病死危险因素分析及临床对策

An analysis of risk factors and clinical strategies in ICU patients with septic shock during 1991 to 2001

感染性休克（SepticShock）亦称脓毒性休克、中毒性休克，是严重感染的并发症，为内科常见危重病症。

Septic shock , also named toxic shock , is a complication of severe infection and a common emergency of internal medicine .

方法检测38例不同类型的SIRS（创伤、创伤性休克、感染性休克）患者及12例健康对照组血浆TNF-a、NE、ET、ADM水平。

Methods Plasma TNF-a , NE , ET and ADM levels were measured in 38 patients with SIRS .

其主要死亡原因是感染性休克、MOF、ARDS及严重水、电解质紊乱及酸碱失衡。

The main cause of death was septic shock 、 MOF 、 ARDS and severe disturbance of water-electrolyte 、 acid-base balance .

CRRT在感染性休克致急性肾功能衰竭中的疗效评价

The efficacy of continuous renal replacement therapy in acute renal failure patients induced by septic shock

血流动力学的监测对感染性休克（SepticShock）的早期诊断、预后的判断以及治疗终点的选择、效果的观察、方案的调整均至关重要。

Hemodynamic monitoring is crucial in every step of the treatment of septic shock such as early diagnosis , prognosis evaluation , endpoint selection , effectiveness judgement and plan regulation .

研究者的研究涉及了各组病例间的BNP水平比较，13例感染性休克患者，18例早期脓毒血症，年龄匹配的18例健康对照。

The researchers'study involved a comparison of BNP levels in13 patients with septic shock , 18 with early sepsis , and18 healthy age-matched controls .

目的观察鸟苷酸环化酶抑制药亚甲蓝（MB）对感染性休克犬血液动力学和氧合功能的影响。

Objective To study the effects of methylene blue ( MB ), a guanylate cyclase inhibitor , on hemodynamics and oxygenation in dog with septic shock .

对并发消化道出血及感染性休克患者死亡前24～72h行血流动力学及氧合状态监测。

Hemodynamics and oxygenation state were supervised on patients combined with gastrointestinal hemorrhage or septic shock 24 ~ 72h before death .

本实验以庆大霉素地塞米松合用（GD）治疗活大肠杆菌诱发的犬感染性休克，观察了GD对氧自由基有关参数的影响。

Effect of combined administration of gentamycin and dexamethasone ( GD ) on oxygen free radical was observed in dogs with septic shock induced by E. coli .

本文研宄了山莨菪碱对感染性休克大鼠血浆TXB2和6-酮-PGF1a含量的影响。

The effects of anisodamine on the plasma levels of TXB2 and 6-keto-PG-F1 α in rats during septic shock were observed .

感染性休克的患者中间，随访中BNP水平下降能预测生存率，而持续高水平与死亡相关。

Among septic shock patients , a drop in BNP levels during follow-up was predictive of survival , whereas continuation of high levels correlated with death .

结论RS和HES液体复苏均有利于升高血压，提高氧输送，改善感染性休克时的血流动力学状态；但在改善内脏器官灌注上HES可能优于RS。

Conclusion Both HES and RS could improve MAP and DO2 in dogs with septic shock , but the effect of HES was better than RS on splanchnic perfusion .

目的：研究诱导性一氧化氮合酶抑制药氨基胍（AG）对感染性休克犬生化的影响。

Objective : This paper was designed to investigate the effects of Aminoguanidine ( AG ), an inducible nitric oxide synthase ( iNOS ) inhibitor , on biochemistry aspect of dogs in septic shock .

尽管抗生素和临床支持治疗取得了长足的进步，感染性休克患者的死亡率仍高达40%-60%，细菌内毒素的主要成分脂多糖（LPS）介导体内炎症免疫反应的失衡，引起广泛组织损伤是其主要原因。

One of the major reasons is that the main active component of endotoxin , lipopolysaccharide ( LPS ), mediates disturbance of immune and inflammatory response and causes extensive tissue damage .

重要并发症包括：急性心包填塞9例、Ⅰ~Ⅱ度MR10例、原有Ⅰ~Ⅱ度MR程度不同地加重者9例、脑血栓栓塞1例、感染性休克1例。

The other important complications included acute pericardial tamponade in 9 cases , brain arterial thromboembolism and infection shock in 1 case respectively .

目的由血红素氧合酶（HO）催化血红素分解代谢而形成的一氧化碳（CO）可能作为一种新的内源性介质参与感染性休克一系列病理生理过程。

Objective Endogenous carbon monoxide ( CO ), which was mainly produced through the metabolism of heme catalysed by hemeoxygenase ( HO ), was suggested to play a role in the pathophysiology of septic shock .

目的：观察连续血液净化（CBP）治疗儿童感染性休克的疗效及其免疫机制。

Objective : To observe the effects and immunological mechanism of continuous blood ( purification )( CBP ) in treatment of children with infectious shock .