视神经损害

本文综合分析了近年来有关原发性青光眼视神经损害机制的研究，并以独特的视角分析了眼压对青光眼视神经损害的具体机制。

This article analyzed the recent researches on the injury mechanism of the primary glaucoma 's optic nerve and the concrete mechanism of IOP inducing glaucoma optic nerve injury from a distinct perspective .

目的比较原发性开角型青光眼（POAG）和原发性闭角型青光眼（PACG）的早期视神经损害特征和视野损害特点的差异。

Objective To compare the difference of early optic nerve damage and visual field defect between primary open angle glaucoma ( POAG ) and primary angle-closure glaucoma ( PACG ) .

与印度患者比较，中国先天性青光眼患者发病较晚，诊断较迟，角膜增大和视神经损害更严重（p＜0.01）。

Compared with the Indian patients , the Chinese patients had a later onset , a delayed diagnosis , more severe corneal changes and more severe optic nerve damages ( p0.01 ) .

建立慢性高眼压条件下视网膜损害相关基因的抑制性消减cDNA文库，可为今后青光眼视神经损害机制及视神经保护的研究奠定基础。

The subtractive differentially expressed genes cDNA library could provide valuable information in the research of optical neuro damage and protection in glaucoma .

目的：观察睫状神经营养因子（CiliaryNeurotrophicFactor，CNTF）对鼠眼急性高眼压视网膜神经节细胞（RGCs）和视神经损害的保护作用。

Objective : To observe the protective effects of ciliary Neurotrophic Factor ( CNTF ) on optic nerve and retinal ganglion cells ( RGCs ) in rat eyes with acute ocular hypertension .

结论mERG能够反映青光眼视神经损害，其确切机制仍需进一步探讨。

Conclusion mERG is useful in detecting early glaucomatous damage . However , the mechanism of mERG need to be further discussed .

结论伴随眼压升高出现的血清中HSP27抗体水平升高可能在青光眼视神经损害中具有重要作用。

Conclusion The enhanced level of anti-HSP27 antibody in serum with IOP increase in experimental rat glaucoma model might play an important role in glaucomatous optic neuropathy .

视网膜神经节细胞（Retinalganglioncells，RGCS）凋亡是青光眼视神经损害的最终共同通路。而钙拮抗药能通过多种环节阻断视网膜神经节细胞凋亡，起到视神经保护作用。

The apoptosis of retinal ganglion cells and the damage of optic nerves in glaucoma have their common passage , but antagonist drugs with calcium are able to block up the apoptosis of retinal ganglion cells by many links , thus playing a role of protecting optic nerves .

如果将BYP和CCT的检测结果结合起来分析，将对OHT患者发展为青光眼视神经损害作出更有价值的预测。

It is valuable in the prediction of the development of glaucomatous damage among ocular hypertension patients if blue-on-yellow field result and central corneal thickness are taken into account together .

经过多因素分析发现影响视力的主要因素是视神经损害、角膜损害（Haab纹）、屈光不正和屈光参差。

The major factors influencing the visual acuity were the glaucoma optic nerve damage , corneal damage ( haab striae ), refraction error and anisometropia .

一氧化氮与青光眼视神经损害

Nitric Oxide and the Deterioration of Optic Nerve in Glaucoma

原发性青光眼视神经损害的发生机制

The injury mechanism of the primary glaucoma 's optic nerve

开角型青光眼视神经损害的发生机制

The mechanism of optic nerve damage in open angle glaucoma

马兜铃酸肾病继发视神经损害一例临床分析

Clinical analysis of aristolochic acid nephropathy with optic atrophy

葛根素注射液治疗青光眼视神经损害的疗效观察

Effects of Puerarin injection on glaucoma optic nerve damage

早期青光眼视神经损害评价一致性的研究

Research on agreement among clinicians on evaluation of optic disc damage in early glaucoma

视神经损害的动物模型

Establishment of animal model of optic nerve damage

视盘血管主干位置在青光眼视神经损害诊断中的作用

The role of location of retinal vessel trunk in diagnosis of glaucomatous optic nerve

原发性开角型青光眼和闭角型青光眼早期视神经损害特征的比较

Comparison of early optic nerve damage between primary open angle glaucoma and primary angle-closure glaucoma

挫伤性视神经损害

On Optical Nerve Damage by Contusion

乙胺丁醇中毒性视神经损害

Optic nerve neuropathy by ethambutol toxicity

结论葛根素注射液对青光眼视神经损害有明显的增视作用。

Conclusion Some of the visual functions of glaucoma optic nerve damage can be improved by Puerarin injection .

青光眼是一种以视神经损害伴进行性视野缺损为主要特征的疾病，已成为全球第二大致盲眼病。

Glaucoma is a disease with the main feature of an optic nerve damage associated with progressive visual field defect .

本文从眼压、眼底、视野、青光眼视神经损害机制以及视神经保护五个方面对青光眼定义进行了阐述。

The article explains the glaucomatous definition from the five positions : intraocular pressure , fundus , visual field , the damage of glaucomatous opticus and the safeguard of opticus .

或者人为制造一块房水的回流区域（此方法主要用于急性青光眼造成的视神经损害或眼内压持续上升的情况）；

Another procedure called trabeculectomy creates an artificial drainage area ; it 's used in cases of advanced glaucoma where there is optic nerve damage and the IOP continues to soar .

术后矫正视力＜0.1者共51眼（5.1%），均为合并青光眼晚期视神经损害、黄斑变性及糖尿病性眼底病变患者。

There were 51 eyes ( 5.1 % ) with postoperative corrected vision of less than 0.1 , mostly in patients having complicating glaucoma with late optic nerve damage , macular degeneration and diabetic retinopathy .

开角型青光眼患者机体内的细胞免疫水平不同于正常人，提示免疫因素可能在视神经损害机制中起重要作用。

The alteration of the cellular immune system in patients with open-angle glaucoma supports our hypothesis that the immune system may play an important role in the initiation and sustainment of glaucomatous optic neuropathy in some patients .

视网膜缺血对兔视神经纤维损害的定量研究

Quantitate the effect of retinal ischemia on rabbit optic nerve axons

从而引起视神经的损害，也可导致失明。

By inflaming the optic nerve , it can also lead to blindness .

视神经的损害能导致视力下降，并有可能致盲。

Damage to the optic nerve may lead to decreased vision and possibly blindness .