肺泡隔

B组肺体积增大，表面淤血，病理改变以肺间质病变为主，肺组织毛细血管明显扩张、充血，肺泡隔增宽，大量白细胞渗出、聚集。

In group B , lung increased and surface congested . The pathological changes were as following : interstitial lung disease , the dilation and congestion of pulmonary capillaries , widened alveolar septum , the leakage and aggregation of a large number of white blood cells .

BCG组于感染后第10～30天肺泡隔增宽，逐渐加重至肺大部分实变，间质内以淋巴细胞浸润为主，结核结节较少，肺内大量菌体；

The alveolar septum in BCG gradually became wide and in interstitium lymphocyte infiltration dominated , and there were less tuberculous granulomas but there were large number of Mycobacterium tuberculosis in the lungs from 10th to 30th days after challenging .

结论VEGF及KDR水平减少与肺泡隔细胞凋亡的增加可能与肺气肿的发生相关。

Conclusion The decrease of VEGF and KDR and the increase of alveolar septal cell apoptosis may contribute to the pathogenesis of emphysema .

其中，肺泡隔断裂与炎细胞浸润为其主要特征，这一点不同于以肺泡及间质水肿为主要表现的临床常见ALI，其原因可能是由于海水的直接损伤。

The broken alveolar septa and inflammatory cells infiltration are the main pathological features , which is different from the other ALIs whose histological feature is the edematous alveoli and lung parenchyma . It may be due to the direct damage of the seawater .

肺病理切片显示纤维化程度降低。病理图像分析表明，治疗组与BLM组相比，有核细胞数减少，肺泡隔所占面积显著减少。

As determined by image analysis , the degree of fibrosis in the lungs of treated group was milder , the number of nucleated cells decreased and the ratio of interalveolar septum area was remarkably decreased , compared to the BLM group .

RSV肺炎组小鼠肺间质较多淋巴细胞浸润，肺泡隔重度增宽和实变，毛细血管严重瘀血、血小板聚集和血栓形成；巨噬细胞溶酶体少，残余体堆积；

Infiltration of more lymphocytes in lung interstitium , severe thickening of alveolar inter septum , pulmonary consolidation , sludging of blood , platelet aggregation and embolism in capillaries , fewer lysosomes of macrophages associated with excessive residual products were found in the control group .

模型动物肺组织炎性粒细胞浸润明显，弥漫性肺泡隔增厚，灶性出血和纤维蛋白渗出。

The main changes in the microscopic examination were inflammatory granulocyte infiltration , disseminated thickening of alveolar septa and focal hemorrhages .

超微结构改变包括：①肺泡隔纤维化逐渐明显，胶原纤维增生；

The ultrastructural changes were as follows : ① Fibrosis of the alveolar septum was gradually conspicuous and collagen fibers were proliferated ;

目的：研究家兔肺内淋巴管的微细分布，探讨肺泡隔内是否存在淋巴管。

Objective : To study the distribution of the intramural lymphatics in rabbit lung and to explore whether there are lymphatics in the alveolar septum .

国内外的研究资料显示，ALI主要的病理特点是：肺泡及肺间质水肿，肺泡隔断裂、肺泡破裂、相互融合，大量肺泡萎陷且程度不均一。

Studies at home and abroad demonstrated that the main pathological features of ALI are edematous alveoli and lung parenchyma , broken alveolar septa , fusion of alveoli , collapse of many alveoli , and alveoli irregular in size .

B组部分肺泡Ⅱ型上皮细胞表面微绒毛有脱落现象，肺泡隔胶原纤维组织增生，上皮细胞基底部变窄。

Rabbits of group B had the following changes : microvillus of some type ⅱ epithelial cell surface of pulmonary alveoli developed shedding , the collagen fibers tissue of pulmonary alveoli compartment proliferated , and the base of endothelial cell narrowed .

光镜观察HE染色的肺组织切片，第7～8周实验组大鼠肺泡内被大量虫体和渗出物充满，肺Ⅱ型上皮细胞脱落，肺泡隔增厚。

HE stained slices were observed under optic microscope . In week 7-8 , the pulmonary alveoli of the experimental rats were filled with a large quantity of polypides and exudates . Type II pulmonary cells were found in exfoliation . Thickening of the interalveolar septum was noticed .

超微病理改变表现为肺泡上皮细胞水肿，I型上皮细胞肿胀，n型上皮细胞微绒毛脱落，板层体排空，血管内皮细胞肿胀，肺泡隔间隙明显增厚。

Ultrastructural changes showed the edema of epithelial cells and endothelial cells , both type I and type II cell injuries .