肺泡隔
- 网络alveolar septa;Alveolar septum;interalveolar septum
![肺泡隔](../cache/1191514.jpg)
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B组肺体积增大,表面淤血,病理改变以肺间质病变为主,肺组织毛细血管明显扩张、充血,肺泡隔增宽,大量白细胞渗出、聚集。
In group B , lung increased and surface congested . The pathological changes were as following : interstitial lung disease , the dilation and congestion of pulmonary capillaries , widened alveolar septum , the leakage and aggregation of a large number of white blood cells .
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BCG组于感染后第10~30天肺泡隔增宽,逐渐加重至肺大部分实变,间质内以淋巴细胞浸润为主,结核结节较少,肺内大量菌体;
The alveolar septum in BCG gradually became wide and in interstitium lymphocyte infiltration dominated , and there were less tuberculous granulomas but there were large number of Mycobacterium tuberculosis in the lungs from 10th to 30th days after challenging .
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结论VEGF及KDR水平减少与肺泡隔细胞凋亡的增加可能与肺气肿的发生相关。
Conclusion The decrease of VEGF and KDR and the increase of alveolar septal cell apoptosis may contribute to the pathogenesis of emphysema .
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其中,肺泡隔断裂与炎细胞浸润为其主要特征,这一点不同于以肺泡及间质水肿为主要表现的临床常见ALI,其原因可能是由于海水的直接损伤。
The broken alveolar septa and inflammatory cells infiltration are the main pathological features , which is different from the other ALIs whose histological feature is the edematous alveoli and lung parenchyma . It may be due to the direct damage of the seawater .
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肺病理切片显示纤维化程度降低。病理图像分析表明,治疗组与BLM组相比,有核细胞数减少,肺泡隔所占面积显著减少。
As determined by image analysis , the degree of fibrosis in the lungs of treated group was milder , the number of nucleated cells decreased and the ratio of interalveolar septum area was remarkably decreased , compared to the BLM group .
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RSV肺炎组小鼠肺间质较多淋巴细胞浸润,肺泡隔重度增宽和实变,毛细血管严重瘀血、血小板聚集和血栓形成;巨噬细胞溶酶体少,残余体堆积;
Infiltration of more lymphocytes in lung interstitium , severe thickening of alveolar inter septum , pulmonary consolidation , sludging of blood , platelet aggregation and embolism in capillaries , fewer lysosomes of macrophages associated with excessive residual products were found in the control group .
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模型动物肺组织炎性粒细胞浸润明显,弥漫性肺泡隔增厚,灶性出血和纤维蛋白渗出。
The main changes in the microscopic examination were inflammatory granulocyte infiltration , disseminated thickening of alveolar septa and focal hemorrhages .
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超微结构改变包括:①肺泡隔纤维化逐渐明显,胶原纤维增生;
The ultrastructural changes were as follows : ① Fibrosis of the alveolar septum was gradually conspicuous and collagen fibers were proliferated ;
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目的:研究家兔肺内淋巴管的微细分布,探讨肺泡隔内是否存在淋巴管。
Objective : To study the distribution of the intramural lymphatics in rabbit lung and to explore whether there are lymphatics in the alveolar septum .
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国内外的研究资料显示,ALI主要的病理特点是:肺泡及肺间质水肿,肺泡隔断裂、肺泡破裂、相互融合,大量肺泡萎陷且程度不均一。
Studies at home and abroad demonstrated that the main pathological features of ALI are edematous alveoli and lung parenchyma , broken alveolar septa , fusion of alveoli , collapse of many alveoli , and alveoli irregular in size .
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B组部分肺泡Ⅱ型上皮细胞表面微绒毛有脱落现象,肺泡隔胶原纤维组织增生,上皮细胞基底部变窄。
Rabbits of group B had the following changes : microvillus of some type ⅱ epithelial cell surface of pulmonary alveoli developed shedding , the collagen fibers tissue of pulmonary alveoli compartment proliferated , and the base of endothelial cell narrowed .
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光镜观察HE染色的肺组织切片,第7~8周实验组大鼠肺泡内被大量虫体和渗出物充满,肺Ⅱ型上皮细胞脱落,肺泡隔增厚。
HE stained slices were observed under optic microscope . In week 7-8 , the pulmonary alveoli of the experimental rats were filled with a large quantity of polypides and exudates . Type II pulmonary cells were found in exfoliation . Thickening of the interalveolar septum was noticed .
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超微病理改变表现为肺泡上皮细胞水肿,I型上皮细胞肿胀,n型上皮细胞微绒毛脱落,板层体排空,血管内皮细胞肿胀,肺泡隔间隙明显增厚。
Ultrastructural changes showed the edema of epithelial cells and endothelial cells , both type I and type II cell injuries .