肺泡上皮
- 网络alveolar epithelium;alveolar cell
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批间C.各组肺泡上皮细胞通透性间比较差异均无显著性。
There was no difference in the permeability of alveolar epithelium among ALI , LV and SI groups .
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PCNA蛋白主要在肺泡上皮细胞,支气管上皮细胞,肺泡巨噬细胞和炎症细胞表达。
PCNA was mainly expressed in alveolar epithelium cells , pulmonary macrophages and inflammatory cells .
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神经肽P物质对高体积分数氧暴露下大鼠Ⅱ型肺泡上皮细胞的调控作用
Regulative Effect of Neuropeptide Substance P on Type ⅱ Alveolar Epithelial Cells in Rat Exposed to Hyperxia
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肺泡上皮II型细胞的免疫学相关因素
The Immunological Factors of Alveolar Epithelial Type II Cell
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支气管肺泡上皮对大气中SO2、NOX反应的研究
Research on the effect of SO_2 and NO_x in the air to bronchus and alveolus epithelium
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肺结节病肺泡上皮细胞CD(44)表达的观察
A study of the expression of CD_ ( 44 ) in pneumocytes in pulmonary sarcoidosis
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超微病理改变表现为肺泡上皮细胞水肿,I型上皮细胞肿胀,n型上皮细胞微绒毛脱落,板层体排空,血管内皮细胞肿胀,肺泡隔间隙明显增厚。
Ultrastructural changes showed the edema of epithelial cells and endothelial cells , both type I and type II cell injuries .
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高浓度氧对早产鼠II型肺泡上皮细胞增殖和细胞周期的影响
Effects of hyperoxia on the proliferation and cell cycle of type II alveolar epithelial cell from premature rat
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目的:研究体外循环血清对肺泡上皮细胞的损伤作用以及N乙酰半胱氨酸的保护作用。
Objective : To explore the effect of cardiopulmonary bypass serum on apoptosis of alveolar epithelial cells and protective effect of N acetylcysteine .
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结果:在正常肺泡上皮细胞和微静脉中,bFGF和TGFβ均有一定表达。
Results : Both bFGF and TGF β gene were expressed in normal alveolar capillary bed .
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结果:随滴注液pH值的降低,通过肺泡上皮屏障的双向蛋白流明显增加。
Results : The bidirectional flux of protein across the lung epithelial barrier increased with the lowering of pH of the acid solution instilled .
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红芪能保护Ⅱ型肺泡上皮细胞,促使其恢复产生和释放PS功能,稳定板层小体的结构。
㏄ rotectiong type II alveolar cell and promoting it to release PS , and steadying Osmiophilic multilamellar body .
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电镜发现NS组Ⅱ型肺泡上皮细胞内的管髓体脱落,PS层断裂。
In group NS , the tubular myelin exfoliated into the alveolar cavity and PS layer was broken .
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结果P53和Bax的表达主要定位于各级支气管上皮和原始肺泡上皮细胞。
Results P53 and Bax were expressed throughout fetal lung development , mainly in bronchial epithelial cells and in alveolar cells .
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LPS致ALI的机制主要是直接损伤肺泡上皮和血管内皮细胞。
Main mechanism of ALI induced by LPS is that it directly damaged pulmonary alveolar epithelium and vascular endothelium .
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维甲酸下调高氧暴露下胎鼠肺成纤维细胞及Ⅱ型肺泡上皮细胞MMP-2表达
Retinoic acid down-regulates the hyperoxia-induced expression of MMP-2 in fetal rat lung fibroblasts and type ⅱ alveolar epithelial cell
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但由于在ALI发生时,肺泡上皮细胞不仅仅是单纯的结构功能损伤,还伴有严重的凋亡现象,因此,对肺泡上皮细胞在ALI时的修复涉及到细胞结构功能的修复和肺泡上皮细胞的再生。
When ALI occurred , alveolar epithelial cells appear not only structural and functional injury , but also severe apoptosis .
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COPD大鼠Ⅱ型肺泡上皮细胞凋亡水平变化及吸入糖皮质激素对其的影响
Apoptosis of Type ⅱ Alveolar Epithelial Cells in Chronic Obstructive Pulmonary Disease ( COPD ) Rats and the Effects of Inhaled Glucocorticoid
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目的观察使用牛重组碱性成纤维细胞生长因子(bFGF)支气管灌洗治疗吸入性损伤后气管粘膜及肺泡上皮的修复。
Objective To observe the repair of trachea mucous membrane and alveolar epithelium in aspiration injury by bronchoalveolar lavage with bFGF .
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免疫组化检测RSV抗原主要分布在细支气管、毛细支气管和肺泡上皮细胞胞浆内。
RSV antigen was mainly localized in cytoplasm of alveolus and bronchioles by immunohistochemistry assay .
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A、B两组肺泡上皮、肺间质及肺血管内皮细胞ICAM-1表达均呈阳性,C组肺组织ICAM-1表达阴性。
The expression of ICAM-1 was positive in the lung in group A and B but negative in group C. Pulmonary vascular endothelium was injuryed under electron microscope .
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而关于Ⅱ型肺泡上皮细胞(typeⅡalveolarepithelialcell,AEC-Ⅱ)在CLD肺上皮重塑中作用如何?肺组织纤维增生的机制等诸多问题,目前研究甚少。
And there are few investigations about the role of type II alveolar epithelial cell ( AEC - II ) in pulmonary epithelia remodeling and the interstitial fibrosis .
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结论:HT合剂可能通过保护血管内皮细胞和肺泡上皮细胞以及抑制胶原生物合成起到对肺纤维化的防治作用。
Conclusion : CHTM could through protect the vascular endothelial cells and alveoli epithelial ceils to prevent and treat pulmonary fibrosis .
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PAI-1在肺泡上皮细胞炎症因子表达和炎症细胞趋化中作用的研究
The Effect of PAI-1 on the Expression of Inflammatory Factors of Alveolar Epithelial Cells and Inflammatory Cells Chemotaxis
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且肺泡上皮ICAM-1表达与肺组织炎症积分呈正相关。
In the meantime , the increased expression of ICAM-1 in the lungs was correlated with the inflammation degree in lung tissue .
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Egr-1影响成纤维细胞增殖、向肌成纤维细胞分化,并且与肺泡上皮细胞、内皮细胞凋亡,肺泡、气道炎症以及细胞外基质形成有关。
Egr-1 is concerned with the apoptosis of alveolar epithelial cell and endothelial cell .
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而肺损伤后肺泡上皮细胞受损,故肺泡细胞CD44表达下降,从而减弱对肺泡损伤修复能力及对炎症反应的控制。
Furthermore , alveolar epithelial cell damaged after pulmonary injury , which decreased CD44 expression , so reconditioning ability decreased .
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目的:测定不同pH值条件下酸性溶液对肺泡上皮屏障的损伤;观察特布他林对急性肺损伤大鼠肺泡液体清除功能的影响。
Objective : To investigate the deleterious effect of acid solution on the alveolar epithelial barrier function and the effect of terbutaline on alveolar fluid clearance ( AFC ) in acute lung injury in rats .
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腺病毒E1A基因对吸烟所致肺泡上皮细胞IL-8表达的影响
Effect of adenovirus ElA gene on interleukin-8 expression induced by CSE in alveolar epithelial cells
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KGFR腺病毒表达载体的构建及其在LPS所致肺泡上皮细胞损伤修复中的应用
Construction of Adenovirus Expression Vector of KGFR and Its Application in LPS-Induced Alveolar Epithelial Damage Repair