肺水肿

fèi shuǐ zhǒng
  • pulmonary edema;pneumonedema
肺水肿肺水肿
肺水肿 [fèi shuǐ zhǒng]
  • [pulmonary edema] 液体渗出物进入肺泡及肺间质

肺水肿[fèi shuǐ zhǒng]
  1. 肺水肿、肺萎陷以及换气功能障碍与肺泡表面活性物质(Pulmonarysurfactant,PS)减少有着密切关系。

    Pneumonedema , alveolar and dysfunction of gas exchange is closely related to the decrease of pulmonary surfactant ( PS ) . Pulmonary hemorrhage and pneumonedema are the significant pathological traits of pulmonary injury in blast injury .

  2. 其次为SLE肺炎5例(16.7%),尿毒症肺水肿3例(10%),弥漫性肺间质纤维化2例(6.7%),肺不张1例(3.35%)。

    There were also 5 cases of pneumonia ( 76.7 % ), 3 cases of uremia pneumonedema ( 10 % ) , 2 cases of diffuse pulmonary interstitial fibrosis ( 6.7 % ) and one case of atelectasis ( 3.3 % ) .

  3. 高原肺水肿;线粒体DNA;单核苷酸多态性;基因型;

    High altitude pulmonary edema ; Mitochondrial DNA ; Single nucleotide polymorphism ; Genotype ;

  4. 结论高原胸部火器伤后G蛋白表达异常增高,以高原移居组最为明显,这可能是高原胸部火器伤后肺水肿发生的重要环节之一。

    These changes may be one of the important links of lung edema after firearm trauma at high altitude .

  5. 左心衰CT示肺水肿、左心室扩大和胸腔积液。

    CT showed that there were pulmonary edema , left ventricular enlargement and pleural effusion , in the patients with left heart failure ;

  6. 海水淹溺肺水肿兔血浆MDA和SOD的变化

    Changes of malondialdehyde and superoxide dismutase in plasma in rabbits with pulmonary edema after seawater drowning

  7. 目的研究光气致BALB/c小鼠肺水肿模型的肺脏的DNA损伤和凋亡。

    Objective To study formation of pulmonary edema , DNA damage and apoptosis of lung cells induced by phosgene in BALB / C mice .

  8. 两组患儿X线胸片检查均呈明显肺水肿表现,存活组患儿颅脑MRI检查均提示有脑干脑炎、脑软化灶改变。

    The chest X-ray manifested pulmonary edema in both groups . Brainstem encephalitis and encephalomalacia were seen in cranial MRI detection of all survival group .

  9. A、C组同期与B组比较,肺泡炎减轻,肺水肿减轻,纤维素样渗出减少,胶原纤维沉积减少。

    Group A , C compared with group B the same period , the alveolar reduce inflammation , relieve edema , cellulose sample exudate reduce , collagen deposition decreases . 2 .

  10. 大鼠急性脊髓损伤后交感神经可能过度兴奋,刺激P物质的大量释放,参与急性脊髓损伤后肺水肿的形成过程。

    In rats with spinal cord injury , sympathetic nerve may be over excitation , which may stimulate the release of substance P to take part in the development of neurogenic pulmonary edema .

  11. 实验结果表明;HD对大鼠氯化铵引起的急性肺水肿有明显保护作用。

    The results point out that the protective effect of hemodilution on experimental pulmonary edema was significant .

  12. 血浆GMP-140变化与高原肺水肿的关系探讨

    The relationship between plasma GMP-140 levels and the occurrence of HAPE

  13. 约40%的心衰肺水肿与ARDS的鉴别诊断存在困难。

    About 40 % alveolar edema caused by congestive heart failure and ARDS are hard to distinguish .

  14. 结论AMI并休克者心肌梗死面积大,急性肺水肿、心律失常和肺炎合并症多,需主动脉内气囊反搏者多。

    Conclusion Shock patients more likely have pump failure , arrhythmia , and pneumonia and more often underwent intra-aortic balloon counterpulsation .

  15. 低氧和去铁胺习服对高原肺水肿大鼠的影响及HIF-1表达的研究

    Effect of Hypoxic Preconditioning or Desferrioxamine Preconditioning on High Altitude Pulmonary Edema and on HIF-1 Expression in Rats

  16. 方法将24只新西兰兔制作成海水淹溺性肺水肿机械通气模型,随机分成对照组(C组)和特布他林治疗组(T组)。

    Methods 24 New Zealand rabbits were infused with seawater to establish pulmonary edema models and then were randomly divided into the control group ( CG ) and the terbutaline therapy group ( TG ) .

  17. 心源性肺水肿组使用NPPV时间明显短于ARDS组,差异有显著性(P<0.05)。

    Duration of NPPV in CPE was shorter than ARDS ( P < 0.05 ) .

  18. 急性肺水肿伴急性冠状动脉综合征患者的30d预后

    Thirty day prognosis of patients with acute pulmonary oedema complicating acute coronary syndromes

  19. 目的:对34例颅脑损伤后并发中枢性肺水肿(CPE)进行临床分析并提出相应治疗措施。

    Objective : To analyse 34 cases of central pulmonary edema ( CPE ) after craniocerebral injury and put forward appropriate measure .

  20. 结果:2例顺利完成手术,1例于术后2h突发急性肺水肿死亡。

    Results : Two patients were smooth going , but one of them died from acute lung edema two hours after operation .

  21. 住院期间心脏不良事件(再发急性心肌梗死、心原性休克、急性肺水肿、发病30D内死亡)的患者发生率A组显著低于B组(0%比7.7%,P<0.01)。

    The incidence of in-hospital MACE was also significantly less in group A than in group B ( 0 vs. 7.7 % , P < 0.001 ) .

  22. 方法:利用氯气造成大鼠反应性肺水肿,利用ELISA方法测定大鼠血中E选择素浓度。

    Methods : the rat lung model of edema was prepared by being exposed to chlorine gas . double antibody sandwich quantitative ELISA method was used to measure the concentration of E selectin .

  23. 肺泡灌洗液及血清中P物质含量的变化与肺水肿轻重程度成正相关,血清CD44含量变化与肺水肿轻重程度呈负相关性。

    The degree of pulmonary edema was positive correlation with the change of substance P in serum and BALF , and was negative correlation with the change of CD44 in serum .

  24. PPV对心源性肺水肿的患者十分有益,因它可减少前负荷和后负荷。

    PPV is very helpful in patients with cardiogenic pulmonary edema , as it helps to reduce preload and afterload .

  25. 目的探讨中枢组织胺与家兔神经源性肺水肿(NPE)发生之间的相关性。

    Objective To explore the roles of central histamine in the onset of NPE .

  26. 目的研究重症心源性肺水肿伴休克患者机械通气(MV)治疗时通气压力的选择对预后的影响。

    Objective To investigate the influence of mechanical ventilation pressure on the prognosis of patients with severe cardiogenic pulmonary edema ( CPE ) and shock .

  27. 1研究背景高原肺水肿(Highaltitudepulmonaryedema,HAPE)是人体在进入高原环境后缺乏适应的情况下,发生的一种以肺动脉血管相关病变为主要特征的疾病。

    Background : High altitude pulmonary edema ( HAPE ) is a kind of disease featuring in pathological changes of pulmonary artery caused by the lacking of adaptability when people enter plateau circumstance .

  28. 据报道海水淹溺型肺水肿(pulmonaryedemaofseawaterdrowning,PE-SWD)是海水溺水死亡的主要原因,其主要病理生理学特点为低氧血症、高碳酸血症和代谢性酸中毒。

    According to what was reported the main reason for death of sea-water drowning was pulmonary edema of sea water drowning . Hypoxemia , hypercapnia and metabolic acidosis were the common pathophysiology trait .

  29. 结论肾上腺素所致急性肺水肿可以使家兔血浆MMS含量降低,其机制可能与应激反应有关。

    Conclusion Acute pulmonary edema rabbits caused by epinephrine can reduce MMS levels in the plasma , a mechanism might be associated with stress reaction .

  30. 目的评价无创通气模式持续气道正压(CPAP)和双水平气道正压(BiPAP)通气在急性心源性肺水肿治疗中的作用。

    Objective To evaluate the effects of noninvasive bi-level positive airway pressure ventilation ( BiPAP ) on acute pulmonary edema .