氧中毒

大鼠急性氧中毒后全脑细胞能量代谢的活体~（31）P磁共振谱研究

31P Nuclear magnetic resonance spectroscopy studies on global cerebral cellular energy metabolism following acute oxygen poisoning in rats

目的：探讨大鼠急性氧中毒后脑的能量代谢产物与脑细胞内pH变化的规律。

Objective : To evaluate cerebral cellular energy metabolism and pH in the rat global cerebral cellular following acute oxygen poisoning .

核因子κB在肺型氧中毒发病中的作用研究

Effect of NF - κ B on Pulmonary Oxygen Toxicity

高压氧中毒小鼠大脑皮质毛细血管ALP活性的变化

Changes of ALP activity in the mice cerebral cortex capillary after

αvβ3整合素和补体系统的参与为寻找防治肺型氧中毒的有效手段提供了新的思路。

The involvement of v β 3 integrin and complement activation provides a new area for finding therapeutic measure for pulmonary oxygen toxicity .

目的：研究心房钠尿肽（ANP）与氧中毒的相互关系。

Objective : To investigate the relation between atrial natriuretic peptide ( ANP ) and pulmonary oxygen poisoning .

经静脉注入鼠心房钠尿肽Ⅲ，观察ANP对肺型氧中毒的影响。

We injected atriopeptin ⅲ into rats veins and observed the effects of ANP on pulmonary oxygen poisoning .

结论HBO暴露导致神经元内GAD减少，可能是急性氧中毒发生的重要原因之一；

Conclusions HBO exposure could decrease GAD content in neurons , which may be an important factor correlative with the onset of acute oxygen toxicity .

急性氧中毒6、12h时血清IL-6的质量浓度均高于正常对照组，有显著或非常显著性差异（P<0.05或P<0.01）；

The serum level of IL-6 in mice at the 6 h , 12 h was higher than normal control group ( P < 0.05 or P < 0.01 ) .

结果急性氧中毒组小鼠血小板膜糖蛋白CD31和CD61的表达水平均高于正常对照组，有显著性差异（P均<0.05）；

Results The expression of CD31 , CD61 and CD62p on platelet membrane in acute oxygen toxicity group mice significantly increased compared with normal control group mice ( respectively P < 0.05 , P < 0.05 , P < 0.01 ) .

为探讨细胞因子在急性氧中毒中的作用和意义，用酶联免疫吸附法检测急性氧中毒后1、6和12h小鼠血清IL-1β、IL-6、IL-10的质量浓度。

The objective was to investigate the role and significance of cytokines in mice after acute oxygen toxicity . The serum level of IL-1 β, IL-6 and IL-10 in mice at the 1 h , 6 h and 12 h after acute oxygen toxicity was measured by enzyme-linked immuno-sorbent assay .

本文采用计算机图像定量测量方法，对高压氧中毒（HBOI）后小鼠软脑膜微血管和大脑皮质毛细血管内皮细胞碱性磷酸酶（ALP）活性进行定量检测。

The changes of meninx microvessels of mice and alkaline phosphatase ( ALP ) activity in their cerebral cortex capillary after hyperbaric oxygen intoxication ( HBOI ) wereobserved and measured by means of the computer image analyser .

心房钠尿肽对大鼠肺氧中毒的影响

Effects of atrial natriuretic peptide on pulmonary oxygen poisoning in rats

近年来药物预防急性氧中毒的研究进展

Progress in preventing acute oxygen toxicity with medicine in recent years

氧中毒大鼠蛋白酶抗蛋白酶平衡状态的实验研究

Experimental Studies on Protease Antiprotease Balance of Rats Exposed to Hyperoxia

充分发育的血管对氧中毒是不敏感的。

Fully developed blood vessels are not sensitive to oxygen toxicity .

目的：进一步探索肺氧中毒的机理。

Aim : To further investigate the mechanism of pulmonary oxygen toxicity .

急性氧中毒小鼠血小板膜糖蛋白表达的变化

Changes of platelet membrane glycoprotein expression in mice after acute oxygen toxicity

急性氧中毒小鼠血清细胞因子水平的变化

Changes of Serum Level of Cytokines in Mice after Acute Oxygen Toxicity

巨噬细胞炎症蛋白-2在肺型氧中毒炎症发生中的作用研究

Effect of Macrophage Inflammatory Protein 2 on Pulmonary Oxygen Toxicity

氧中毒方式及反应条件对F&T合成的影响

Effect of Oxygen Poisoning and Reaction Condition on Fischer-Tropsch Synthesis

谷氨酸脱羧酶在急性脑型氧中毒中的快速变化

The Rapid Changes of Glutamic Acid Decarboxylase During Convulsive Type of Oxygen Toxicity

脂质体包封超氧化物歧化酶预防急性氧中毒

Protection against Acute Oxygen Toxicity by Liposome-entrapped Superoxide Dismutase

氧中毒是氧疗的并发症。

Oxygen toxicity is a complication of oxygen therapy .

高压氧中毒对肺毛细血管细胞色素氧化酶的影响高压氧中毒小鼠肺损伤及其机理

Effects of Cytochrome Oxidase Activity in Pulmonary Capillary of Mice Under Hyperbaric Oxygen Toxicity

肺型氧中毒小鼠肺组织巨噬细胞炎症蛋白-2mRNA表达的改变

Changes of macrophage inflammatory protein-2 mRNA expression in the lung tissue of pulmonary oxygen toxicity mice

目的为探讨反应性氧中毒物质在诊断恶性肿瘤中的应用价值。

Objective To assess the applicating value of reactive oxygen toxic species in diagnosing malignant tumor .

高压氧中毒小鼠脑微血管图像分析定量研究

Study on brain microvessels of mice after hyperbaric oxygen intoxication by means of the computer image analyser

介绍了氧中毒及抗氧化剂作用机理，以及食疗抗氧化剂谷胱甘肽。

The oxidative demag and antioxidant action mechanisms in vivo , and dietary food antioxidant Glutathione are described .

但这样的直接接触，常会导致血液的含氧量过高，造成氧中毒。

But this direct contact often led to too much oxygen being absorbed and resulted in oxygen toxicity .