咬合创伤

咬合创伤大鼠三叉神经节中PPTAmRNA表达的变化

Expression of PPTA mRNA in trigeminal ganglion in rat during occlusal trauma

结论线粒体Ca2+超载通过抑制ATP合成使咀嚼肌细胞出现能量代谢紊乱，可能是咬合创伤致咀嚼肌功能紊乱的主要作用机制。

Conclusion Ca + { 2 + } overload in mitochondria depresses ATP production , which results in energy metabolism disorder in masticatory muscle cells . It may play an important role in the mechanism that occlusal trauma results in masticatory muscle dysfunction .

X射线片显示，咬合创伤1个月组创伤侧下颌磨牙牙周膜增宽，牙槽骨有少量吸收。

As shown by X - ray films , periodontium of lower jaw molar widened and alveolar bone was absorbed a little in 1 - month trauma group .

MMP-3在咬合创伤牙周组织中的表达

Expression of MMP-3 in periodontium with traumatic occlusion

目的研究NFκB受体活化因子配体（RANKL）和骨保护蛋白（OPG））mRNA在咬合创伤大鼠牙槽骨组织中的相对表达水平，探讨RANKL和OPG在咬合创伤牙槽骨改建中的意义。

Objective To investigate the expression change of receptor activator of NF κ B ligand ( RANKL ) and osteoprotegerin ( OPG ) mRNA in rat alveolar bone and the possible mechanism of alveolar bone resorption under traumatic occlusion .

结果：（1）过度张口和咬合创伤均可导致TMJ和咀嚼肌的损伤，且均以盘周附着组织和相关的咀嚼肌反应最显著。

Results : ( 1 ) Overextended opening and traumatic occlusion resulted in some injury to TMJ and masticatory muscles . Among the injured tissues , the disc attachment and correlative masticatory muscles underwent the most obvious traumatic changes .

方法：选择10例上前牙外伤致松动的患者，应用直径为0.25mm金属结扎丝加光固化复合树脂固定，消除咬合创伤，随访观察6-24个月。

Methods To choose more than 10 cases that the patients'anterior teeth loosened traumatically , apply medical metal wire (Φ 0.25mm ) to ligate and light curing Microglass composite to splint , relieve the occlusive trauma and observe 6-24 months .

咬合创伤后牙本质形成相关细胞的形态学变化

Morphologic change of the cells related to dentinification followed occlusion trauma

神经生长因子在咬合创伤犬牙髓组织中的变化

Expression changes of NGF in traumatic occlusal pulp tissue of the dogs

咬合创伤大鼠行为学及营养状况观察

Effects of occlusal trauma on behavior and nutrition of rats

咬合创伤对颞颌关节及咀嚼肌影响的组织病理学实验研究

Effect of traumatic occlusion on histopathology of the temporomandibular joint and masticatory muscles

实验性咬合创伤去除前后大鼠磨牙髓组织的变化

A Study of Pulp during Traumatic Occlusion and after Removal in Molar of Rats

初级感觉神经元在咬合创伤致牙髓组织损害和伤害感受中的作用

Action of Primary Sensory Neurons in the Pulp Tissue Damage and Nociception by Occlusal Trauma

各种错（？）畸形造成的咬合创伤是牙周病重要的局部促进因素。

Occlusal trauma resulting from many kinds of malocclusion is a very important local promoting factor .

但是，一些特殊的情况，比如颌面部或咬合创伤都会严重的影响植体的使用寿命。

However , specific situations such as facial or occlusal trauma can negatively impact their longevity .

兔咬合创伤时咬肌损伤与线粒体Ca~（2+）超载的研究

Ca ~ ( 2 + ) overload in mitochondria and injury in rabbit 's masseter muscles induced by occlusal trauma

咬合创伤致兔咬肌线粒体钙离子和二、三磷酸腺苷含量的改变

Changes of Mitochondrial Calcium , Adenosine Triphosphate and Adenosine Diphosphate Contents Induced by Occlusal Trauma in Rabbit ′ s Masseter Muscles

目的：研究咬合创伤导致咬肌损伤的作用和线粒体Ca~（2+）超载在损伤机制中的作用。

Objective : To study the role of mitochondrial Ca2 + overload in mechanism of masticatory muscle injury induced by occlusal trauma .

结论：咬合创伤是咬肌损伤的致病因素之一，而线粒体Ca~（2+）超载则是咬肌损伤发生机制中的一个重要环节。

Conclusion : Mitochondrial Ca2 + overload may have a role in the etiology of injury of masticatory muscle induced by occlusal trauma .

本研究结果为探索中枢神经系统在咬合创伤牙周组织及牙周神经损伤修复中的调控机制提供了新的思路。

The result can provide new idea for repair mechanism of periodontium and periodontal nerve of central nervous system in occlusal trauma rats .

目的研究线粒体Ca2+超载及能量代谢在咬合创伤所致咀嚼肌功能紊乱中所起的作用。

Objective To study the role of Ca + { 2 + } overload and energy metabolism in mitochondria in masticatory muscle dysfunctional induced by occlusal trauma .

结论：细心保护脱位牙根面牙周膜、良好固定、预防感染、防止咬合创伤是再植成功的重要因素。

Conclusion : Carefully protecting periodontal membranes , keeping good fixation , preventing from infection and avoiding occlusal injuries are important factors for successful implantation of the dislocated teeth .

有研究证实咬合创伤参与了牙周炎的发生与发展，并且对牙周组织的改建产生了直接的影响。

According to the studies , trauma from occlusion can contribute to the occurrence and development of periodontitis , and make direct impact to the recreating of periodontal tissue .

对咬合创伤导致骨吸收机制的研究是目前口腔医学研究领域的一大热点，其具体分子生物学机制目前尚不清楚，有待进一步研究。

The mechanism of bone absorption caused by occlusal traumation is currently one of the hot spots in oral medicine field , the exact molecular biology mechanism is unclear , needing further study .

结论咬合创伤是牙根纵裂的主要原因，X线检查有助诊断，牙根纵裂缺乏有效的治疗方法，应以预防为主。

Conclusion Occlusal traumatism was the main causes of vertical root fracture . X-ray examination was helpful to diagnosis . Prevention methods should be taken for lacking of effective treatment on vertical root fracture of untreated teeth .

结果：老年人牙体修复后牙折与窝洞类型，根面龋，重度磨损，楔形缺损，修复频率及咬合创伤等因素有关。

Results : the senior 's tooth fracture after the tooth prosthesis is related to several terms , such as cavity type , root caries , dental abrasion , wedged shape defect , prosthesis repeatedly and traumatic occlusion , etc.

近一百年来，对牙周疾病的研究不断深入，其致病机理仍有诸多未解之谜，以往研究已经证实咬合创伤可参与牙周炎的发生与发展，并直接影响牙周组织的改建与修复。

In the last one hundred years , periodontal disease pathogenesis is still not completely solved , but existing studies have demonstrated that occlusal traumatism can aggravate the occurrence and development of periodontal disease , and even affect periodontal tissue repair .

目的探讨建立创伤咬合动物模型的有效方法，为探讨咬合创伤致口面痛机制的研究提供依据。

Objective The aim of this study was to establish an availability and credibility traumatic occlusal animal model by dogs in order to do further basic research works in traumatic occlusion field .

目前国内有关中老年人咬合状态的调查研究，主要集中在牙齿缺失情况、牙齿磨耗、咬合创伤等方面，专门描述咬合支持状态的研究报道还较少见。

At present , the domestic investigations concerned the occlusion of middle-aged and elder mainly include tooth missing 、 tooth abrasion 、 occlusal trauma and so on , but the studies on the occlusal support are also rare either in domestic or foreign .