卵黄囊

luǎn huánɡ nánɡ
  • yolk sac
卵黄囊卵黄囊
  1. 阴道超声检测早孕卵黄囊

    Transvaginal Sonographic Detection of the Yolk Sac in the First Trimester Pregnancy

  2. 人卵黄囊的超微结构研究

    Ultrastructural Study of Human Yolk Sac

  3. 日粮中不同Mn、Zn水平对雏鸡卵黄囊中Mn、Zn转移的影响

    The Influence of Dietary Mn and Zn Levels to the Transfer of Mn and Zn From the Yolk Sac

  4. 小鼠卵黄囊间质干细胞多向分化潜能及其体外复合胶原表面改性CPC的实验研究

    Investigation of Murine Yolk Sac Multipotent Mesenchymal Stem Cells Combined with Collagen Surface-Modified CPC in Vitro

  5. MRI显示早早孕、早中期的卵黄囊、胚外体腔、羊膜囊及胎心搏动不如超声(P<0.005);

    Ultrasonography had great advantage over MRI in showing yolk sacs , amniotic sacs , extraembryonic coelom and embryocardia-beats ( P < 0.005 ) .

  6. 结论:卵黄囊干细胞具有向cFUGM分化的能力;

    Conclusion The committed differentiation from yolk sac stem cells to CFU GM is affected by many factors .

  7. 结果:在卵黄囊、原肠、背侧肠系膜和生殖嵴处有AP强阳性细胞,阳性信号位于胞质内。

    Results : Human PGCs with AP were observed in yolk sac , primitive gut , dorsal mesenteries and genital ridges .

  8. 能量水平对卵黄囊营养的吸收与利用有一定的影响,低能日粮有加速卵黄囊吸收与利用的趋势,但各采食组之间差异不显著(P>0.05)。

    Energy had a little influence on the absorption and utilization of yolk , low energy had a tendency to accelerate absorbing and utilizing the yolk nutrient , but not significantly ( P > 0.05 ) .

  9. 胶原表面改性CPC对小鼠卵黄囊间质干细胞体外生长与BMP-2表达的影响

    Effects of Col ⅰ / CPC on the growth and expression of BMP-2 of murine yolk sac mesenchymal stem cells in vitro

  10. 以AA肉仔鸡为对象,研究了日粮中添加牛磺酸对卵黄囊吸收及甲状腺激素代谢的影响。

    AA broilers were selected as animal model to determine the effects of taurine on the absorption of yolk sac and metabolism of thyroid hormone in broilers .

  11. 结果表明:氟浓度为5μg/ml时,仅对脏层卵黄囊(VYS)血管形成产生抑制作用;

    The results showed the blood circulation of visceral yolk sac vessel formation was only inhibited with exposure to 5.0 μ g / ml fluoride .

  12. 第6-7d的CAM及其血管覆盖了整个卵黄囊表面,且随着胚龄的增加而增大,毛细血管极其丰富。

    Between days 6 and 7 of incubation , the CAM and its blood vessels cover the entire surface of yolk sac , and capillary become extremely rich along with the embryo age increase .

  13. 目的研究人胚胎发育不同时期卵黄囊和胎肝内受体酪氨酸激酶(KDR)、血管内皮生长因子(VEGF)和CD34的分布及表达,为胚胎造血分化提供理论依据。

    Objective To study the expression and distribution of KDR , VEGF and CD34 in yolk sac and liver of human embryo at different development stage .

  14. 在卵黄囊息室中,SIgA阳性细胞主要分布于靠近肠绒毛基部的固有层中,在肠腺中也有少量分布。

    In the jejunum , the SIgA cells mainly were distributed between the intestinal glands and the intestinal villus lamina layer .

  15. 结果:第2组中,卵黄囊细胞出现细胞凋亡特征性的DNALadder,与正常对照组相比ERK1/2蛋白激酶活性显著下降;

    Results : There was strong characteristic apoptotic DNA ladder in yolk sac cells in embryopathic offspring from experimentally induced diabetic rats , and activities of ERK1 / 2 were dramatically decreased ( group 2 ) .

  16. 6.0μg/ml时则可致胚胎组织GSH活性和卵黄囊细胞膜脂质流动性显著降低。

    At Al 3 + concentration of 6.0 μ g / ml , the activity of GSH and the membrane lipid fluidity of visceral yolk sac decreased significantly .

  17. 对AFP增高而病理报告为良性者,应仔细检查畸胎瘤组织中有无卵黄囊组织。

    The benign teratoma as seen from pathological reports with upward AFP , should be checked very carefully to assure there were endodermal sinus tissue in teratoma tissues or not .

  18. 采用石蜡切片的方法对中国林蛙原始生殖细胞(primordialgermcells,PGCs)的发生及迁移路径进行研究,结果表明:①中国林蛙原始生殖细胞发生于卵黄囊内胚层;

    The cytogenesis and migratory path of Rana chensinensis PGCs ( primordial germ cells ) was studied with paraffin section . Result is :① PGCs originated from the endoderm of the yolk-sac .

  19. 结论氟有潜在的致畸性和胚胎毒性,胚胎组织GSH活性和卵黄囊细胞膜脂流动性降低可能在氟致胚胎发育毒性中起重要作用。

    Conclusions Fluoride could result in embryonic growth retardation and potential teratogenic toxicity . These effects might be due in part to decrease in GSH activity and membrane lipid fluidity .

  20. 目的探讨熊去氧胆酸(ursodeoxycholicacid,UDCA)的应用对胆汁淤积症(ICP)孕鼠脏层卵黄囊细胞膜脂质流动性及胎盘组织谷光甘肽(glutathione,GSH)含量的影响。

    Objective To explore the effects of ursodeoxycholic acid ( UDCA ) on the fluidity of rat visceral sac and placental glutathione ( GSH ) concentration in rats with intrahepatic cholestasis .

  21. 结果大鼠PGCs位于卵黄囊内胚层深部的间充质内,体积大,呈椭圆形或不规则形;

    Results In vivo PGCs were large with ovoid or irregular shape , located in mesemchyma under the endoderm of yolk sac .

  22. 氟浓度为10μgml时,胚胎组织GSH活性和卵黄囊细胞膜脂质流动性也显著降低(P<005)。

    GSH content in embryonic tissue and membrane lipid fluidity of visceral yolk sac cell reduced obviously at the concentration ≥ 10 μ g / ml ( P < 0.05 ) .

  23. 结果表明,氟作用16h,开始对卵黄囊血管分比和血管网形成有明显抑制作用;

    The results showed that the differentiation of blood vessels in yolk sac of 16 hrs exposed to fluoride was inhibited obviously .

  24. 水温11~13℃时,卵黄囊期需10d,有效积温110~130℃·d。

    At 11-13 ℃, it needs 10 d from hatching stage to yolk-sac absorbed stage , and the accumulative temperature of 110-130 ℃· d is necessary .

  25. ang-1和ang-2及其受体在发育第3~12周人胚胎卵黄囊血岛、主动脉-生殖腺-中肾区的时空表达。

    Blank expression of ang-1 and ang-2 and their receptors in the blood island of yolk sac and AGM region at embryonic 3 to 12 weeks .

  26. 结果发现在第3 ̄4周的人卵黄囊低表达VEGF和KDR,不表达CD34。4 ̄6周组强表达VEGF、KDR和CD34。

    It was demonstrated low expression of VEGF , KDR and null expression of CD34 at 3 ~ 4 week human yolk sac , strong expression of VEGF , KDR and CD34 at 4 ~ 6 week group .

  27. 结论卵黄囊血岛造血细胞和内皮细胞共表达CD10、CD14、CD31和FⅧ,卵黄囊血管内皮细胞表达CD34,而造血细胞不表达CD34。

    Conclusions The hematopoietic cells and endothelial cells of YS blood island co-expressed CD10 , CD14 , CD31 and factor ⅷ . Endothelial cells were CD34 positive but hematopoietic cells were negative in YS blood island .

  28. 在胚胎发育期,胎儿肝细胞和卵黄囊基因开放,表达AFP并分泌入血。出生后AFP表达迅速减少,而白蛋白合成增多。

    AFP , an oncofetal protein , is synthesized mainly in hepatocytes of fetal liver and yolk sac , and AFP production by hepatocytes is rapidly reduced from the time of birth in inverse proportion to an increased synthesis of albumin .

  29. 生物碱还可影响脏壁卵黄囊(VYS)的血液循环,抑制其生长。

    Besides , the alkaloid could effect on blood circulation and inhibit growth of the visceral yolk sac ( VYS ) .

  30. 随着胚胎的发育,HG进一步成熟,但仍以单层细胞分布于胚体和卵黄囊外表面,此时,HGCs数量急剧增多,细胞体积有所增大,分布范围更加广泛。

    With the development of the embryo , the HG with monolayered cells was still distributed on the outer surface of embryonic body and yolk sac . At the same time , the number and the volume of HGCs increased significantly , HGCs spread more widely .