adma
- 网络非对称二甲基精氨酸;非对称性二甲基精氨酸;不对称二甲基精氨酸;不对称二甲精氨酸;美国直复营销协会
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Conclusions the present data suggest that poor collateral circulation is related to elevated levels of ADMA and ICAM-1 in patients with severe coronary artery stenosis .
结论冠状动脉严重狭窄患者冠状动脉侧支循环形成不良与高非对称性二甲基精氨酸和细胞间粘附分子1水平有关。
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The relationship between ADMA and uric acid in essential hypertension patients
高血压患者血清尿酸与ADMA的关系
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The plasma ADMA levels in essential hypertension patients were higher then the health population .
原发性高血压组患者较正常人群血浆ADMA水平升高。
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The Role Of ADMA in Cardiovascular Complication of Patients in Chronic Kidney Disease
ADMA在慢性肾脏疾病患者并发心血管疾病中的作用
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Adma : Why do the students do not respond questions ?
为什么学生不回答问题呢?
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There is abundant evidence that ADMA may be a novel risk factor for cardiovascular disease .
大量研究证明,ADMA可能是心血管疾病的一个新的危险因子。
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Clinical Research on the Relationship between DDAH Gene Polymorphisms Regulating ADMA and Chronic Kidney Disease
DDAH基因多态性调控ADMA与慢性肾脏病关系的临床研究
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Effect of ADMA on Maturation and Function of Dendritic Cells Drived from Monocyte
ADMA对单核细胞来源的树突状细胞成熟和免疫的影响
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TO investigate ADMA the role on THP-1 adhesion , migration and proliferation .
探讨ADMA对人单核细胞(THP-1)的粘附、迁移和增殖的作用。
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Furthermore , increased ADMA levels tended to contribute to increased risk of progressive diabetic kidney disease .
同时发现,血ADMA水平的升高对糖尿病肾病进展有促进作用。
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This article reviews the effects of ADMA on endothelial function and its mechanism and the research into related therapeutic procedures .
现就ADMA对内皮功能的影响和机制以及相关的治疗研究现状进行综述。
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The Study of the Levels of ADMA , Hcy and the Correlation in Patients with Vascular Cognition Impairment
VCI患者血浆ADMA、Hcy水平及相互关系的研究
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Concentration of ADMA in conditioned medium was measured by high performance liquid chromatography ( HPLC ) at 12 h.
上清液用以测定ADMA的浓度(高效液相色谱法,HPLC)。
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Role of DDAH / ADMA pathway in the protective effect of HDL on endothelial NO production
DDAH/ADMA通路在HDL促进内皮细胞NO生成中的作用
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The Effects of Trans Fatty Acids on ADMA / NOS / NO and Platelet Function in Rabbits
反式脂肪酸对兔ADMA/NOS/NO及血小板功能的影响
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In a healthy human body , the majority of ADMA is eliminated through active metabolism by DDAH .
在健康人体内,大部分ADMA通过DDAH的活性代谢而被清除。
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ADMA and threonine might be the potential bio-markers of obese .
并且ADMA和Thr可能是儿童超重与肥胖的潜在生物标记物。
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The plasma levels of ADMA were related to atherosclerosis , which could be used as indicators of RA complicated with atherosclerosis .
血浆ADMA水平与RA并发动脉粥样硬化相关,可作为检测RA并发动脉粥样硬化病情的指标之一。
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Meanwhile , many studies have suggested that ADMA has a strong correlation with the degree of atherosclerotic vascular disease among chronic renal disease patients .
同时,许多研究都表明在慢性肾功能衰竭病人中,ADMA水平与动脉粥样硬化的程度具有明显的相关性,而后者又是心血管疾病(CVD)的病理基础。
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Register-based DMAallows the processor to directly program DMA control registers to initiate aDMA transfer .
基于寄存器的DMA允许处理器直接通过修改DMA控制寄存器来启动DMA传输。
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Nisoldipine and olmesartan both can decrease plasma ADMA and ET-1 levels and improve FMD .
尼索地平和奥美沙坦均能明显降低血浆ADMA和ET-1水平并且改善FMD。
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ADMA could promote to the migration of THP-1 cell under the role of monocyte chemotactic factor ( MCP-1 ) .
ADMA能够促进在单核细胞趋化因子(MCP-1)作用下的THP-1细胞的迁移作用。
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These results suggest that in the older group of rats , the elevated level of ADMA may be a contributor to vascular endothelial dysfunction .
提示老龄大鼠血中二甲基精氨酸浓度升高可能是促进血管内皮功能不全的重要原因。
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To observe whether vascular endothelial dysfunction and oxidative stress , inflammation as well as ADMA levels elevation exist in essential hypertensive patients . 2 .
目的1.观察本研究中原发性高血压组中是否存在血管内皮功能障碍以及氧化应激、炎症反应和ADMA水平升高。
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Compared with the healthy control group , the levels of serum ADMA of PD group , CKD group and MHD group all improved significantly ( P 0.05 ) .
结果:1.与健康对照组相比较,PD组、CKD组和MHD组的血清ADMA水平均明显提高,且与对照组相比都具有统计学意义(P<0.05)。
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Objective To explore the role of endogenous nitric oxide synthase inhibitor asymmetric dimethylarginine ( ADMA ) in different factors-induced gastric mucosal injury .
目的:研究一氧化氮合酶抑制物非对称性二甲基精氨酸(ADMA)在不同因素诱发的胃黏膜损伤中的作用,并初步探讨其机制。
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Objective To observe the changes and their significance of serum asymmetric dimethylarginine ( ADMA ) and homocysteine ( Hcy ) in elderly subjects .
目的探讨离退休老干部血清不对称二甲基精氨酸(ADMA)和同型半胱氨酸(Hcy)的变化及其意义。
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Objective To investigate the protective effect of losartan against on injury induced by ox-LDL in endothelial cells and the relationship with asymmetric dimethylarginine ( ADMA ) .
目的:探讨氯沙坦对氧化型低密度脂蛋白(ox-LDL)诱导的内皮细胞损伤的保护作用及其与内源性一氧化氮合酶抑制物非对称性二甲基精氨酸(ADMA)的关系。
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One of the mechanisms leading to endothelial dysfunction is the accumulation of an endogenous inhibitor of eNOS , asymmetric dimethylarginine ( ADMA ) .
导致内皮功能紊乱的机制之一即内源性eNOS竞争性抑制剂不对称二甲基精氨酸(ADMA)的积聚。
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Conclusion : Endothelial dysfunction induced by ox-LDL is associated with the increase of ADMA concentration and reduction of DDAH activity , but not DDAH expression .
结论:ox-LDL诱导下,内皮损伤ADMA的增加与DDAH的活性减弱有关,而与DDAH的表达无关。