脑血管痉挛

提示：ET是SAH后继发脑血管痉挛（CVS）的早期重要因素；

Our results suggest that ET is an early important factor which induces CVS after SAH ;

布洛芬治疗脑血管痉挛可能与其抑制前炎症因子有关。

The possible mechanisms with treat CVS by ibuprofen maybe related to inhibiting the inflammatory cytokine .

脑血管痉挛螺旋CT血管造影临床应用

Clinical application of spiral-CT angiography to detecting cerebral vasospasm

核因子-κB在实验性迟发性脑血管痉挛中的表达变化

The activity change of NF - κ B in arterial wall of cerebral vasospasm in rabbit model

目的研究实验性家兔脑血管痉挛基底动脉核因子κB（NFκB）的活性变化。

Objective To investigate the expression of NF κ B activity in arterial wall of cerebral vasospasm in rabbit model .

大鼠蛛网膜下腔出血诱发脑血管痉挛后脑组织神经肽ymRNA的表达变化

The expression change of neuropeptide Y mRNA of cerebral tissues in rat cerebral vasospasm induced by subarachnoid hemorrhage

有无症状性脑血管痉挛根据患者的临床表现和脑血管造影、经颅多普勒超声、颅脑CT、MRI检查确定。

Patients with symptomatic cerebral vasospasm were diagnosed by clinical manifestations , cerebral panangiography , transcranial Doppler ultrasound brain CT and MRI examination .

腰池引流对破裂动脉瘤患者血浆和脑脊液ET浓度及脑血管痉挛的影响

Effect of Lumbar Cistern Drainage on Plasma and CSF Endothelin Concentrations and Cerebral Vasospasm in Patients with Ruptured Aneurysm

目的采用CT灌注成像技术，对颅内动脉瘤破裂导致蛛网膜下腔出血所产生的脑血管痉挛进行研究，分析和评价其意义和临床应用价值。

Objective The goal of the research was to investigate the application of the computed tomography perfusion imaging in the cerebral vasospasm caused by intracranial subarachnoid hemorrage .

实验性兔脑血管痉挛血清MMP-9及VEGF的变化研究

The experimental study of serum matrix MMP-9 and VEGF in rabbit with cerebral vasospasm after subarachnoid hemorrhage

脑血管痉挛后血管平滑肌细胞中c-fos基因表达及意义

Expression of c-fos gene and its role in vascular smooth-muscle cells after cerebral vasospasm

采用小脑延髓池二次注血法建立大鼠SAH后迟发性脑血管痉挛模型。

We established delayed cerebral vasospasm rat model using double-hemorrhage model .

5-HT、自由基在蛛网膜下腔出血时致脑血管痉挛的实验研究

Experimental Study of Cerebral Vasospasm Induced by 5-HT and Free Radicals in Subarachnoid Hemorrhage

PKC抑制剂对蛛网膜下腔出血性脑血管痉挛的防治作用及分子机制的实验研究

Experimental Study of Molecular Mechanism of PKC Inhibitor Effect on Subarachnoid Hemorrhagic Cerebral Vasospasm

目的：寻找一种有效的治疗脑血管痉挛（CVS）的新方法。

Objective : To find an effective method of cerebral vasospasm treatment .

氟桂利嗪治疗高血压脑血管痉挛的临床及TCD对比研究

Clinical observation and comparative study with TCD of cerebrovascular spasm of hypertension treated with Sibelium

结论：CCK-8对SAH后迟发性脑血管痉挛具有一定的预防作用。

Conclusions ; CCK-8 has the ability to prevent DCVS after SAH .

慢性脑血管痉挛的机理及EDS的作用研究

An Experimental Research on the Mechanism of Chronic Cerebral Vasospasm and the Protective Effect of Ecdysterone

方法回顾本院近5年收治的118例动脉瘤性SAH病人临床及影像学资料；评估入院时临床因素，判定脑血管痉挛程度；

Methods 118 patients with aneurysmal SAH were retrospectively reviewed with clinical and radiological data .

治疗组脑血管痉挛（CVS）发生率明显低于对照组，差异有统计学意义（P

The cerebral vasospasm ( CVS ) rate and mortality in the treatment group was lower than the control group , and showed significant difference ( P

P38信号转导通路对蛛网膜下腔出血后迟发性脑血管痉挛的影响

Effects of P38 Mitogen-Activated Protein Kinase Signal Transduction Pathway in Delayed Cerebral Vasospasm After Subarachnoid Hemorrhage

SAH后迟发性脑血管痉挛TNF-a和NF-κB表达变化及CCK-8预防作用的评价

Expression of TNF-a and NF - κ B in Delayed Cerebral Vasospasm after Subarachnoid Hemorrhage and Assessment of Cholecystokinin-8 Prevention

GM1对脑血管痉挛后神经元及血管内皮细胞的影响

The influence of GM1 on neurons and microvascular endotheliocytes after cerebrovascular spasm

动脉瘤性SAH后症状性脑血管痉挛与高凝状态相关性及防治研究

The relationship between Symptomatic Cerebral vasospasm and high coagulation state following aneurysmal SAH and its prophylactic study

结论SAH后早期应用尼莫地平对症状性脑血管痉挛具有明显的改善作用。

Conclusions Nimodipine given early after SAH may show effects on symptomatic cerebral vasospasm in rabbits remarkably .

蛛网膜下腔出血（SAH）后的脑血管痉挛（CVS）是一个多因素参与作用的复杂的病理生理过程。

Cerebral vasospasm after subarachnoid hemorrhage ( SAH ) is a complicated pathophysiological course caused by varied factors .

c-jun氨基末端激酶在蛛网膜下腔出血后症状性脑血管痉挛中作用及机制的实验研究

Experimental Study on Role of c-Jun N-terminal Kinase and Its Mechanism on Symptomatic Cerebral Vasospasm after Subarachnoid Hemorrhage

实施腰蛛网膜下腔持续引流，释放血性脑脊液，辅以抗凝及3H治疗是防治脑血管痉挛和血栓栓塞的的关键。

Continuous spinal cerebrospinal fluid drainage , anticoagulation and 3H therapy are important for the prevention and treatment of cerebral vasospasm .

Rho激酶与脑血管痉挛

Rho kinase and cerebral vasospasm

目的：评价丁咯地尔和尼莫地平对脑血管痉挛（CVS）的疗效和血流动力学的影响。

AIM : To evaluate the therapeutic effects and the hemodynamics of buflomedil and nimodipine in cerebral vascular ( CVS ) .