脑水肿

目的：了解MK-801对大鼠感染性脑水肿是否具有保护作用及可能的作用机制。

Objective : To investigate the protective effect and its possible mechanism of action infective encephaledema of rats .

脑脊液铁离子含量与急性颅脑损伤脑水肿关系的研究

Correlation between content of Fe in cerebrospinal fluid and encephaledema

实验性脑水肿CT值和脑组织含水量的关系

Relationship between CT value and brain tissue water content in brain edema

并根据CT值可将脑水肿分为轻、中、重度。

Based on CT value , brain edema is classified as mild , moderate and severe .

核因子-κB活性在大鼠脑水肿的变化及热休克反应对其影响的研究

NF - κ B activity and effects of heat shock response on brain tissue in rats with infection brain edema

本文对40例经CT扫描、手术及病理证实的颅内脑膜瘤与脑水肿的关系作了回顾性分析。

This paper presents a retrospective analysis of 40 patients with intracranial meningioma diagnostically confirmed by CT scan , operation and pathological studies .

MMP-9高表达是致脑水肿的关键物质。

MMP-9 were the key substance which induced the brain edema .

ATP酶变化在1,2二氯乙烷致脑水肿过程中的作用研究

Study on changes of activity of ATPase in brain edema caused by 1,2-dichloroethane

脑水肿的SPECT·rCBF显像和X线CT的比较分析

Comparision and analysis of SPECT · rCBF research with X-ray CT on brain edema

应用干-湿重法观察脑水肿规律、HE染色观察血肿周围炎性细胞浸润，免疫组化方法观察NF-κB表达。

Brain edema law was observed by dry-wet method , inflammatory cells infiltrating round hematoma by HE staining , and NF - κ B expression by immunohistochemistry .

目的研究急性高原病和（或）高原性脑水肿的颅脑CT表现及其病理学基础。

Purpose To investigate the relationship between brain CT manifestations and pathologic changes of acute mountain sickness and / or high altitude cerebral edema in cats .

颅内再出血，脑水肿，脑肿胀时ICP可呈阶梯式反跳。

Rebleeding , brain edema and cerebral swelling could increasing ICP by ladder way .

高压氧对外伤性脑水肿家兔线粒体ATP酶活性的影响

Effect of hyperbaric oxygenation on cerebral mitochondria and ATPase activity in traumatic cerebral edema of rabbits

测量损伤24h后损伤区域脑水肿程度。

Brain edema severity was measured at 24 h.

目的用MRI影像学方法检测抗脑水肿药对急性脑梗死后缺血性脑水肿的治疗效果。

Objective To investigate the efficacy of drugs used to treat brain edema in a rat acute cerebral infarction model by MRI image analysis .

结果照射后2h出现急性放射性脑水肿病理改变，照射后3d达高峰；

Results Acute radiation induced cerebral edema was observed morphologically 2 hours after irradiation and peaked on day 3 after irradiation .

热休克蛋白70对感染性脑水肿核转录因子-κB及其抑制蛋白的影响

Effect of heat shock protein 70 on nuclear factor - κ B / inhibitor protein - α of nuclear factor - κ B in brain edema induced by infection in rat

结论严重烧伤早期脑水肿模型兼有血管源性和细胞毒性脑水肿的MRI及病理组织学特征。

Conclusion The model of the brain edema after severe burn had the feature of both vasogenic edema and cellular edema in the MRI and pathology .

结果显示，早期应用大剂量维生素C治疗急性颅脑伤，能有效地控制脑水肿的发展，减轻继发性脑损伤。

The results showed that acute brain injury should he treated with large dose vitamin C at the early stage which could control the development of cerebral edema and secondary brain inJury .

七叶总皂苷及A、B、C、D组分均具有抗炎、抗脑水肿和增强静脉张力的作用，且都呈现出剂量依赖性。

All Escin and its elements A 、 B 、 C 、 D could inhibit inflammation , decrease brain edema and increase the tension of the vein effects in a dose-depended way .

出现症状性放射性脑水肿6例，均为AVM患者。

Brain edema was found in 6 patients with AVM .

按再灌注不同时间即6h、24h、3d三个时间点采集标本。并通过干湿重法测脑水肿变化隋况；

According to pours into the different time is again 6h , 24h , the 3d three time gathering specimen .

结论HA中性大孔树脂血浆吸附治疗肝性脑病、脑水肿、全身性炎性反应综合征等方面具有较好的应用价值，且具有安全性高的特点。

Conclusions : Plasma adsorption technique with HA microporous resin can be applied safely as therapeutic management for HE , brain oedema and SIRS .

结论MMP-9及TIMP-1可能参与了感染性脑水肿的发生和发展。

Conclusion Both MMP-9 and TIMP-1 might play roles in forming and developing infectious brain edema of rats .

结果：黄芩甙能明显减轻脑水肿程度（P<0.01），降低脑组织铁含量和MDA浓度（P<0.01），提高SOD活性（P<0.01）。

Results : The baicalin could reduce the infective brain edema , and lower iron content , MDA level , and improve the activity of SOD .

急性颅脑损伤ANP，AngⅡ变化及对创伤性脑水肿的作用机理研究

A Study of Atrial Natriuretic Peptide , Angiotensin ⅱ Changes and Mechanism to Traumatic Brain Edema during Acute Head Injury

结论：谷氨酸有加重创伤性脑水肿的作用，谷氨酸使Na＋和Ca2＋内流增加可能是其加重脑水肿的主要病理生理机理。

The increases of Na ￣ + and Ca ￣ 2 + influx may be the main pathophysiological mechanism of glutamate in exacerbating brain edema post-injury .

结论：HSP70、iNOS、TNF-α及IL-β参与了外伤性脑水肿的病理过程。

Conclusion : HSP 70 , iNOS , TNF - α and IL-1 β are involved in contusion cerebral edema .

结论：MK-801对大鼠感染性脑水肿有保护作用。

Conclusions : MK-801 acts the part of protection on infective encephaledema of rats .

目的：探讨连续性血液滤过（CHF）治疗心肺复苏后对血液动力学不稳定的脑水肿患者的疗效。

Objective : To investigate the effect of continuous hemofiltration ( CHF ) on outcome of cerebral edema with unstable hemodynamics following cardio-pulmonary resuscitation .