肾性高血压

ARB对肾性高血压大鼠脑缺血再灌注后NOS的影响

The effect of ARB to NOS for the renal hypertension rats with injured of focal cerebral ischemia-reperfusion

川芎素对肾性高血压并慢性肾功能不全患者血浆ET-1和NO的影响

Effects of sodium ferulate on plasma levels of endothelin-1 and nitric oxide in patients with renal hypertension and chronic renal insufficiency

肾性高血压大鼠中枢P物质含量的变化

The Changes of Substance P Contents in Central Nervous System of Renal Hypertensive Rats

肾性高血压大鼠组织CaM和cAMP的含量

Changes in Contents of Calmodulin and cAMP in Tissues of Renal Hypertensive Rats

卡托普利和红花萃取物对肾性高血压大鼠血清选择性炎性细胞因子水平及心脏核因子-κB表达的影响

The Effects of Captopril and Honghua Extractives on the Serum Levels of Selective Inflammatory Cytokines and Nuclear Factor - κ B Expression of Heart in Reno-hypertensive Rats

肾性高血压大鼠局灶性脑缺血再灌注后Survivin表达和细胞凋亡及意义

The expression of Survivin and apoptosis after MCAO and reperfusion in renovascular hypertensive rats

结果地龙降压颗粒能明显降低肾性高血压大鼠的血压、降低TG和CHO。

Results Dilong Jiangya Granule markedly lowered renal hypertension , reduced TG and CHO in rats .

结论：肾性高血压主动脉PLC活性升高，AngⅡ可能通过活化的PLC参与高血压的发生和发展。

Conclusion : The activity of phospholipase C in aorta induced by Ang ⅱ may play an important role in renal hypertension .

肾性高血压大鼠离体胸主动脉环Na～+-Ca～（2+）交换活性研究

A Na ~ + - Ca ~( 2 + ) Exchange Process in Isolated Thoracic Aortic Rings from WKY and RH Rats

ACEI治疗肾性高血压和对肾机能的保护作用

The protection of renal function in the ACEI treatment of renal hypertension

结论：原发性高血压与肾性高血压的心肌肥厚均与PP、BVP相关，提示PP、BPV可能是心肌肥厚的预测因子；

Conclusion : PP and BPV may be the predict factors of LVMI in hypertension patients .

目的探讨肾性高血压时血红素加氧酶（HO）/一氧化碳（CO）系统的变化，以明确血红素加氧酶在肾性高血压发病过程中的意义及作用。

Objective To investigate the role of heme oxygenase / carbon monoxide ( HO / CO ) pathway in the development of renal hypertension .

目的：研究肾性高血压大鼠局灶性脑缺血再灌注后脑组织Survivin、Bcl-2表达和细胞凋亡，探讨其意义。

AIM : To study expression of Survivin and Bcl-2 and cellular apoptosis in brain after MCAo and reperfusion in renovascular hypertensive rats .

氯沙坦对肾性高血压左室肥厚大鼠心肌血管紧张素Ⅱ及原癌基因c-junmRNA表达的影响

Effect of losartan on concentration of Ang ⅱ and expression of c-jun mRNA in the left ventricle myocardium of rats with renovascular hypertension

培哚普利可通过降低TGF-β1和MMP-2的表达进而抑制肾性高血压大鼠心肌纤维化的进展。

Perindopril can inhibit progress of RH rat myocardial fibrosis by inhibiting the expression of MMP-2 and TGF - β 1 .

MAPK及MKP-1在肾性高血压大鼠心肌肥大过程中的变化

The Change of MKP-1 and MAPK in Myocardium of Renovascular Hypertensive Rats

芝麻素对肾性高血压伴高血脂大鼠主动脉收缩功能和eNOS蛋白表达的影响

Sesamin on contractile function and eNOS expression in the aorta of renal hypertensive rat with hyperlipidemia

肾性高血压大鼠脑动脉瘤形成过程中NF-kB和MMPs表达的关系

The relationship between the expression of the NF-kB and MMPs in hypertension induced cerebral aneurysm in rats

洛沙坦对肾性高血压大鼠心肌AT（1a）mRNA、AT（1b）mRNA表达的影响

Effects of Losartan on the regulation of the expression of myocardial AT_ ( 1a ) mRNA , AT_ ( 1b ) mRNA from renovascular hypertensive rats

结果表明EO在原发性肾病综合征的发生发展及肾性高血压的产生和维持中起一定作用。

The results indicates that EO play an important role on development of primary nephrotic syndrome and nephrotic hypertension .

目的研究慢性肾性高血压大鼠脑核磁共振成像（MRI）的变化和相应部位超微结构的改变。

Objective To study changes in brain magnetic resonance imaging ( MRI ) and ultrastructures of corresponding area in chronic renal hypertensive rats in order to further recognize the chronic brain damage in hypertension .

结论：天麻钩藤方逆转了肾性高血压大鼠心肌和主动脉肥厚的病理改变，降低了心脏、主动脉组织AngⅡ的升高。

Conclusion : TGR can attenuate myocardial and aorta hypertrophy induced by renovascular hypertension , and suppress the rise of Ang ⅱ in tissue significantly .

提示组织中CGRP的释放减少和循环血中其含量不足可能与肾性高血压发病有关。

If suggested that the decreased release of CGRP from tissues and low plasma levels of CGRP might be related to the pathogenesis of renal hypertension .

结果：网包组11例术后无并发症发生；随访10个月至2年，IVU排泄功能良好，B超检查形态正常，无肾性高血压发生。

Results : no-complication occurred after operation and the patients have been followed up for 10 months to 2 years .

这些结果提示在2K1C肾性高血压中，动脉血压升高是左心室肥厚、左心室肌球蛋白MHC基因表型转换的重要因素；

These results suggested that left ventricular hypertrophy and the MHC isoform transitions may be induced by pressure overload .

结论：肾性高血压早期，MHC基因表达发生改变，肾素血管紧张素系统可能起重要作用。

CONCLUSION : The expression of MHC gene has changed at an early stage of renal hypertensive rat , and renin-angiotensin system may play an important role in this change .

半边莲生物碱抑制肾性高血压大鼠内皮素1mRNA和蛋白表达

Lobelia Chinensis Lour Alkaloids Inhibit Expression of Endothelin-1 mRNA and Protein in Renal Hypertensive Rats

结论：在HD过程中，血小板释放的NPY可能是造成肾性高血压和肾血管痉挛的重要病理因素。

NPY related to NT significantly . Conclusion : NPY , secreted by platelets , may play an important role in the pathogenesis of renal hypertension and renal vasospasm in HD .

大鼠肾性高血压模型稳定建立后，用金属微电极记录室旁核放电活动并检测血中肾素、醛固酮水平和室旁核AngⅡ受体的表达。

After the models were stable , the electrical activity of PVN was recorded with metal microelectrode , and blood Ang II and the expression of Ang II receptor in PVN was detected .

Isorhy（10mg/kg和2mg/kg，iv）亦能分别降低肾性高血压清醒大鼠和麻醉犬的BP。

The BP and HR were also reduced by Isorhy ( 10 mg / kg and 2 mg / kg , iv respectively ) in hypertensive rats and anaesthetized dogs .