多巴

结论：培养的大量细胞可用于RPE的体外实验研究，多巴染色是鉴定RPE细胞一种较好的方法。

Conclusion : We can produce a large quantity of RPE cells for in vitro experiments and DOPA staining proves effective in identification of RPE cells .

治疗组32例，运用眼针疗法，再结合常规剂量的美多巴（madopar）治疗。

Treatment group of 32 patients using eye-acupuncture therapy , in combination with conventional doses of the United States dopa treatment .

美多巴B族维生素浓度对帕金森患者同型半胱氨酸的影响

The effect of Madopar and vitamin B on plasma homocysteine in patients with Parkinson diease

中国人常染色体隐性遗传性多巴反应性肌张力障碍TH基因突变分析

TH gene mutation in Chinese patients with autosomal recessive dopa-responsive dystonia

方法将6羟基多巴（6OHDA）注入大鼠纹状体内制作PD动物模型。

Methods Rat models of PD were made by injection 6-hydroxydopamine ( 6-OHDA ) into striatum .

美多巴治疗后P1波潜伏时缩短、振幅增高（P<0.05）。

Through detecting PVEP during the treatment of amblyopia with Madopar , we found P1 latency shortened and amplitude increased ( P < 0 . 05 ) .

未用多巴类药物的患者针刺前DA含量均低于健康人。

The DA content in urine of patients before acupuncture was less than healthy ones .

多巴资本（TobaCapital）风险投资合伙人帕特里克•马西森的回答

Answer by Patrick Mathieson , VC Associate at Toba Capital

结论低蛋白饮食方法有助于PD患者长期服用美多巴治疗。

Conclusion Low - protein diet could help parkinson 's patients , to take a long - term treatment of Madopar .

方法：将40例肝肾阴虚型PD患者随机分为治疗组和对照组，治疗组联合服用美多巴和熄风定颤丸；

Methods : Forty cases of PD with kidney deficiency type were selected and divided into the treatment group and the control group .

结论：吡贝地尔单用或联合复方旋多巴应用，吡贝地尔可以较好地改善早期PD患者的精神、情绪和睡眠等非运动症状；

Conclusion : Both mono-use and combined use of piribedil can improve the mental status , depression emotion and sleep quality of early PD patients .

头部电针透穴疗法组与体针组能减少美多巴的副作用，与美多巴药物组比较（P＜0.01），同时治疗组、体针组能减少美多巴的用量。

Sometime , electroacupuncture Scalp Point-through-Point therapy group and acupuncture group could decreased side effect , contrasted acupuncture group ( P < 0.01 ), electroacupuncture Scalp Point-through-Point therapy group and acupuncture group could decreased medicine dose .

目的探讨立体定向注射6-羟基多巴（6-OHDA）建立帕金森病（PD）大鼠模型的方法及其稳定性。

Objective To study the methods of establishing Parkinson 's disease ( PD ) rat model by stereotactic injection of6-hydroxydopamine ( 6-OHDA ) .

目的明确6-羟基多巴（6-OHDA）致多巴胺能神经元凋亡与氧化应激的关系，以及外源性超氧化物歧化酶（SOD）对多巴胺能神经元凋亡的保护作用。

Objective To define oxidative stress and antagonistic action of the exogenous SOD in the apoptosis of the rat midbrain dopaminergic neuronal induced by 6-OHDA .

结论：抗帕颗粒可改善PD大鼠黑质纹状体TH阳性神经元的病理改变，抗帕颗粒联合美多巴治疗效果明显。

Conclusion : Anti-Parkinson potion can decrease the lesions of the TH-positive neurons in substantia nigra and striatum in PD rats , and the group treated with both anti-Parkinson potion and Madopar is better .

Fisher博士不相信大脑可以忽略对某人的厌恶&即使对婚姻危机中的夫妇大量注射的多巴氨，也于事无补。

Dr Fisher does not believe that the brain could overlook distaste for someone & even if a couple in trouble could inject themselves with huge amounts of dopamine .

方法对16例家族性患者和10例散发性多巴反应性肌张力障碍患者进行病史采集、神经系统体格检查和CT或MRI检查，所得数据应用SPSS13.0软件进行统计分析。

Methods Sixteen familiar DRD patients and 10 patients with sporadic DRD were studied by history taking , physical examination of nervous system and CT / MR scaning . The obtained data were statistical analyzed with SPSS 13.0 software in computer .

成功培养了人色素斑皮肤的黑色素细胞，细胞呈双极或者多级树突样，多巴染色和S-100蛋白免疫组化染色结果阳性。

The human melanocytes in melasma skin were successfully cultured , and the cells were multi-dendritic . S-100 protein immunohistochemical staining and Dopa staining were positive .

长期服用多巴制剂的6例中晚期PD两侧积聚、BG/OC和BG/FC比值无明显差别，但均低于正常对照组（P<0.01）。

In the moderate or advanced PD group , no significant differences were observed bilaterally , and the BG / OC and the BG / FC rates in this group was lower than those of the control .

帕金森病组予美多巴治疗1个月后复查，结果纹状体区NAA，NAA/肌酸有上升趋势（P<0.05）。

The NAA and NAA / creatine of striatum in the PD group showed a tendency of increase after treated with Medopa for a month ( P < 0.05 ) .

正常雌鼠及6-羟基多巴型帕金森病雌鼠黑质听觉P50诱发电位的实验研究

P50 auditory evoked potential in substantia nigra of normal female rats and female rat models of Parkinson disease induced by 6-hydroxydopamine

目的研究溴隐停及美多巴联合治疗创伤性脑外伤（TBI）后迁延性植物生存状态（PVS）的临床价值及使用范围。

Objective To study the clinic application and value of the combination use of bromocriptine and madopa in the treatment of the posttraumatic persistent vegetative state ( PVS ) .

工程细胞可抵抗N甲基苯基吡啶（MPP+）对多巴脂能神经元的毒性损伤作用，与对照组比较，细胞数目增加了83.6%（为76倍，P<0.01）。

Results The number of primary dopaminergic neurons protected by double-gene engineered cells increased at least by 2.8 times in comparison with the control cells ( P < 0.01 ), and those cells against MPP + toxicity increased by 83.6 % ( 7.6 times , P < 0.01 ) .

结论AR-JP具有发病年龄早、症状轻、进展缓慢、多巴制剂反应好等特征。

Conclusions AR-JP is characterized by early age of onset , mild symptoms , slow progression and good response to levodopa .

目的：观察6-羟基多巴（6-OHDA）诱导PC12细胞凋亡的作用，探讨神经生长因子（NGF）对PC12细胞凋亡的保护作用。

AIM : To investigate the protective effect of nerve growth factors ( NGF ) against PC12 cell apoptosis by observing the role of6-hydroxydopamine ( 6-OHDA ) in inducing apoptosis of PC12 cells .

目的本研究拟探讨慢性双侧丘脑底核脑深部电刺激器（STN-DBS）对患者多巴反应性以及所需多巴胺能药物剂量的影响。

Objective To investigate the effect of bilateral subthalamic nucleus stimulation ( STN-DBS ) on the levodopa responsiveness and the change of levodopa-equivalent dose .

方法选择YahrⅡ期PD患者24例，随机分为两组，观察组12例给予低蛋白饮食加美多巴治疗，对照组12例单纯美多巴治疗。

Method 24 cases with parkinson 's disease as Yahr ⅱ divided into two groups randomly . The experimental group were treated low - protein diet and taking madopar . The control group took Madopar .

方法：帕金森病（PD）大鼠80只，随机分为4组：对照组、美多巴组、抗帕颗粒组、抗帕颗粒联合美多巴组。

Methods : 80 Parkinson disease ( PD ) rats were divided into 4 groups randomly , the control group ( treated with normal saline ), the group treated with Madopar , the group treated with anti-Parkinson potion , and the group treated with both anti-Parkinson potion and Madopar .

PDD发生的相关因素按标准回归系数依次为性别、疾病严重程度、运动障碍严重程度、文化程度、是否应用多巴受体激动剂5项。

The regression analysis showed that factors accounted for the occurrence of PDD were gender , the severity of PD , stage of patient 's motor impairment , education level and the use of dopamine agonists .

多巴一种胺基酸，C9H11NO4，由酪胺酸在肝脏中生成并在大脑中变为多巴胺。

An amino acid , C9H11NO4 , formed in the liver from tyrosine and converted to dopamine in the brain .