纤维斑块

方法：将不同组成（钙化斑块、纤维斑块和全血凝块）和不同大小（75～105μm和55～74μm）的微栓子混悬液注入SD大鼠右侧颈内动脉。

Methods : Sprague-Dawley rats were injected with saline containing different composition ( calcified plaque , fibrous plaque and whole blood clot ) and size ( 75 ~ 105 μ m and 55 ~ 74 μ m ) or saline alone ( sham operation ) into right internal cerebral artery .

镜下没有脂质条纹没有纤维斑块，没有动脉粥样硬化斑块。

The is no Fatty streak , no Fibrous plaque , and no Atheromatous plaque .

MRI检出纤维斑块11层，检出脂质斑块29层，MR对斑块检测总的阳性率为88%，准确率为80%。

MRI detected 11 slices that contained fiber plaque , detected 29 slices that contained fatty plaque , with a total positive rate of 88 % and accuracy rate of 80 % .

通过OCT成像清晰地显示8处纤维斑块、3处钙化斑块、9处富含脂质斑块、2处血栓形成、斑块破裂3处及血管壁上夹层、粥样硬化斑块微小裂口和夹层等。

Two thromboses , 8 fibrous , 9 lipid-rich and 3 calcium plaques as well as 3 plaque ruptures were visualized by OCT.

分析了VH-IVUS的组成及工作原理，高分辨率的VH&IVUS将冠脉斑块的组成成分分为纤维斑块、纤维脂质斑块、坏死核心和钙化斑块4种。

Four major plaque components of the coronary plaque , including fibrous , fibro-fatty , necrotic core and dense calcified tissue , can be identified after the analysis and discussion of VH-IVUS .

参考段的病变中纤维斑块及钙化斑块较少而脂质斑块较多。

Reference segments contained less fibrotic and calcific plaque and proportionately more soft plaque elements .

科学家关于阿尔茨海默病的研究重心已从病理学的神经纤维斑块和纤维缠结转向研究更好的治疗措施。

Scientists have moved away from plaque and tangles in the brain toward developing better treatments for the disease .

两者的表达显著大于纤维斑块期（P<0.01）。

The expression of CD68 , CD43 in fatty streak plaques were remarkably higher than that in fibrous plaques ( P < 0.01 ) .

陈旧脑梗死中纤维斑块、钙化斑块多见；脑梗死的发生亦与斑块的易损性有关，易损斑块与新发脑梗死有关，稳定斑块与陈旧脑梗死有关。

Fibrous plaque and calcified plaque are found in the old cerebral infarction . The occurrence of cerebral infarction is related with the vulnerability of the plaque . Vulnerable plaque is related with the new cerebral infarction .

纤维期斑块中Actin阳性物质主要分布在斑块的基底部和纤维帽中，其表达显著高于脂纹期（P<0.01）。

Actin in fibrous plaques were significantly higher than that in fatty streak plaques ( P < 0.01 ) .

病理状态下潜在型MMP-9被激活，参与动脉粥样硬化的形成、发展，并削弱斑块纤维帽，使斑块破裂，从而诱发急性冠脉综合征。

It is associated with atherosclerosis , and plays an important role in weakened fibre cap , disruption of plaque , and acute coronary syndrome .

纤维平滑面的斑块状结构消失。

The plaquelike structure on the smooth surface of the fibers disappeared .