淋巴因子
- 名lymphokine
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本文结果对淋巴因子激活的NK细胞是LAK的效应细胞提供了直接证明。
This study provided direct evidence that lymphokine activated NK cells are effectors of LAK cells .
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IL-2和淋巴因子激活杀伤细胞与手术、化疗联合应用治疗中晚期原发性肝癌31例
A report on the treatment of patients with advanced primary liver cancer using interleukin-2 and lymphokine activated killer cells combined with surgery or chemotherapy
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植物血凝素及淋巴因子激活的杀伤细胞体外抗肿瘤作用的MTT比色法分析
The detection of phytohemagglutinin-lymphokine activated killer cells ' in vitro antitumor action with MTT colorimetry
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目的:证实激活骨髓(Activatedbonemarrow,ABM)具有与淋巴因子激活的杀伤(LAK)细胞不同的生物学效应。
Objective : To prove that activated bone marrow ( ABM ) has different biological effect different from LAK cell .
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树突状细胞诱导的淋巴因子激活的杀伤细胞对肺癌细胞系A549的体外杀伤作用
Cytotoxic Activity of LAK Cells Induced by Dendritic Cells against Lung Cancer Cell Line A549 in Vitro
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促进T细胞产生淋巴因子,增强诱杀性细胞NK(自然杀伤性细胞)的分化。
The promotion of T cells to produce lymphokines , and enhance cell Traps NK ( natural killer cell ) differentiation .
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IL-2、IL-4和IL-7体外诱导人外周血淋巴因子激活的杀伤细胞效应的比较
Evaluation of the effects of il-2 , IL-4 and IL-7 on the induction of human lymphokine-activated killer cells
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然而,在感染10周和12周小鼠,无论ConA或SEA都不能诱导IFN-γ或IL-4mRNA转录,显示这两种淋巴因子表达受到抑制。
However , neither ConA nor SEA could induce IFN γ or IL 4 mRNA transcription in 10 and 12 week infected mice indicating the modulated expression of mRNAs of these two lymphokines .
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粘附性淋巴因子活化的杀伤细胞抗自体白血病细胞作用的研究冷冻保存对肝癌患者粘附性LAK细胞生物学特性的影响
Study on the Cytotoxicity of Adherent LAK Cells against Autologous Leukemic Blasts Effects of Cryopreservation on Biological Character of Adherent LAK Cells from Patients with Hepatocellular Carcinoma
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对可溶性CD23(sCD23)的淋巴因子效应进行了初步研究。
Cytokine-effects of soluble CD23 ( sCD23 ) were investigated in our laboratory .
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CD23在B淋巴瘤的发生发展中起作用和该淋巴因子可能对治疗(药理学调节)有重要意义。
CD23 plays a role in the development and progress of B - NHL and that this lymphokine might have great significance ( pharmacological modulation ) in the treatment of B - NHL .
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比较了淋巴因子IL-2、IL-4和IL-7在体外诱导健康人外周血淋巴因子激活的杀伤(LAK)细胞活性的效应。
Had investigated and compared the effect of IL-2 , IL-4 and IL-7 on the generation of lymphokine-activated killer ( LAK ) cell activity in human peripheral blood mononuclear cells of healthy individuals .
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作者从单核、淋巴因子的角度探讨了Tamm-Horsfall蛋白(THP)与免疫系统的关系,以及THP通过自身免疫反应诱发肾小管间质性肾炎(TIN)的机制。
He relationship between Tamm-Horsfall protein ( THP ) and cytokines and the immuno - logical mechanism was studied , by which THP triggers tubulointerstitial nephritis ( TIN ) .
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目的:研究三物降压汤对血压、淋巴因子激活的杀伤(LAK)细胞的影响及其可能机制。
Objective : To investigate the effect of Sanwu hypotensive decoction ( SWHD ) on blood pressure ( BP ) and lymphokine activated killer cell ( LAK ) and possible mechanism .
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提示评价新生儿的免疫状态应结合淋巴因子水平的测定,临床上对严重感染性疾病的治疗试用IL-2和IFNγ有潜在的重要作用。
The results suggest that the evaluation of neonatal immune function must be performed in combination with the assay of lymphokines . Consequently , IL-2 and IFN γ are a new potent form of therapeutic intervention in neonatal infections .
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目的观察淋巴因子激活杀伤细胞(LAK细胞)治疗慢性活动型乙型肝炎(CAH)的疗效,探讨治疗机理。
Objective To observe the therapeutic effect of lymphokine activated killer cells ( LAK cell ) on chronic active hepatitis B ( CAH ) and study the mechanism of the therapy .
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许多体内外实验证实,淋巴因子激活的杀伤细胞(LAK)和肿瘤浸润的淋巴细胞(TIL)有较强的细胞毒活性,但LAK和TIL在体外增殖率较低。
Many experiments have proved that lymphokine-activated killer cells and tumor infiltrating lymphocytes have stronger cytotoxicity and lower proliferation in vivo and vitro . Recently a new type immunocompetent cell has been discovered .
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目前已知除抗病毒活性外,IFN还具有免疫调节、抑制细胞增殖、抗肿瘤生长、与多种淋巴因子协同作用等多种重要的生物功能。
Now it became clear that in addition to inhibit virus replication , IFNs have many important roles , such as regulating immune response , inhibiting tumor growth , resisting cell differentiation and showing a apparent synergic effect with several lymphokines .
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目的探讨前列腺素E2(PGE2)在膀胱癌患者淋巴因子激活的杀伤细胞(LAK)增殖及细胞毒的作用。
Objective To investigate the combined effects of interleukin-2 ( IL-2 ) with prostaglandin E_2 ( PGE_2 ) on the proliferation and cytolysis of bladder tumor cells by lymphokine activated killer ( LAK ) cells in patients with bladder cancer .
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KC可以被PAF活化,同时受活化的T细胞释放的淋巴因子调节,如IFN-γ可以诱导KC产生多种细胞因子、表达ICAM-1、启动Ⅱ类抗原表达等。
KC can be activated by PAF and lymphoid factors released from activated T cell . For example , cytokine , as well as ICAM-1 and antigen to express can be induced by IFN - γ .
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精氨酸与机体免疫功能关系密切,为淋巴细胞增殖、分化及合成细胞因子所必需,还能维持巨噬细胞、中性粒细胞、单核细胞及淋巴因子激活的杀伤细胞(LAK)的活性。
Arginine is required to support the proliferation and differentiation of lymphocytes and the production of cytokine . It also participates in maintaining the activities of monocytes , neutrophils , and lymphokine-activated killer cells ( LAK ) .
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目的:为提高中、晚期恶性肿瘤的治疗效果,根据恶性肿瘤在细胞代谢、分化程度、细胞周期之特点,以化疗与淋巴因子活化性杀伤细胞LAK,/IL2联合应用新方法,观察其治疗效果。
Objective : To explore the therapeutic effect of Combined Chemical therapy and LAK / IL 2 therapy on late malignant tumor according to the feature of its cellular metabolism . differentiating degrees . and cellular cycle .
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目的:探讨雌二醇(E2)和他莫昔芬(TAM)对淋巴因子激活的杀伤细胞(LAK细胞)杀伤卵巢癌细胞3AO作用的影响。
Objective : To investigate the effect of estradiol ( E 2 ) and tamoxifen ( TAM ) on cytotoxicity of lymphokine activated killer cells ( LAK cell ) to lysis human ovarian tumors .
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本文测定和比较了34例消化道癌症、23例非癌症患者和14名健康人外周血淋巴细胞转化和淋巴因子激活杀伤细胞(LAK)诱导生成能力。
The capacities of lymphocyte transformation and the induction of lymphokine-activated killer ( LAK ) cells were investigated and compared between subjects including 34 cases with established cancer in the digestive system , 23 cases of non-cancer patients and 14 healthy volunteers .
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TNF-α是一种具有广泛生物学效应的淋巴因子。
TNF - α is an abroad biological effect lymph factor .
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脐血淋巴因子激活杀伤细胞的实验研究
Study on lymphokine - activated killer cell in-duced from cord blood
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淋巴因子相应靶细胞的小剂量辐射效应
Effect of low dose radiation on the target cells to lymphokines related
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淋巴因子激活的LAK/TIL细胞的表型
Lymphokine activated LAK / TIL cell and its phenotype
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匹多替莫的免疫增强作用及对淋巴因子表达的影响
The effect of immune - potentiating action and expressing lymphocyte factor of pidotimod
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小鼠胸腺和牌细胞对淋巴因子反应的辐射剂量效应关系
Dose-effect relationship of reactivity of thymocytes and splenocytes to lymphokines in mice following X-irradiation