水钠潴留

  • 网络water-sodium retention;retention of water and sodium
水钠潴留水钠潴留
  1. 治疗3wk时两组均未见明显低钾及水钠潴留的发生。

    Neither remarkable hypopotassaemia nor water-sodium retention were found after treatment for 3 weeks .

  2. 坎离煎和开搏通均能明显改善心衰兔水钠潴留状态。

    Both of them obviously improved the status of water-sodium retention .

  3. 慢性肾炎患者水钠潴留与PET相关性研究

    Correlation between PET and Retention of Water and Sodium of Chronic Glomerulonephritis

  4. 为研究慢性肾炎患者水钠潴留与血浆内皮素(PET)的相关性,检测了伴或不伴水钠潴留慢性肾炎患者PET和24h尿蛋白排泄量。

    In order to study the correlation between retention of water and sodium of chronic glomerulonephritis and plasma endotheliolysin ( PET ) .

  5. 与常规蛋白组相比,酮酸组与低蛋白组KT/V、Ccr、尿量均升高(P<0.05),而酮酸组与常规蛋白组的水钠潴留发生率无统计学意义(P>0.05)。

    Levels of KT / V , Ccr and residual urine volume were significantly higher both in ketoacid group and low protein diet group ( P < 0.01 ) .

  6. 人体水钠潴留性疾病心钠素形态学研究

    Morphological study on atrial natriuretic peptide in patients with sodium and water retention

  7. 结论随着肾功能的恶化,低肾素型患者的比例升高提示钠离子介导的水钠潴留成为高血压的主要机理。

    Conclusion Volume expansion mediated by sodium becomes a major mechanism of hypertension in renal insufficiency .

  8. 复方甘草酸苷可迅速降低氨基转移酶水平,并减少水钠潴留和低血钾的发生。

    Compound glycyrrhizin could rapidly lower the aminopherase ( AT ) level and reduce the incidence rates of water-sodium retention and hypopotassemia .

  9. 目的:探讨黄芪对阿霉素肾病大鼠水钠潴留的影响及其可能的作用机制。

    Objective : To observe the effects of astragalus on water sodium retention of adriamycin-induced nephrotic syndrome rats and explore its mechanism . Methods : Nephrotic syndrome was induced by the intravenous injection of ADR .

  10. 并监测患者平均动脉压、腹膜透析超滤量、残余尿量、水钠潴留情况。

    Examinations of RRF , mean arterial pressure ( MAP ), peritoneal ultrafiltration ( UF ), residual urine volume ( RUV ) and the status of water-sodium retention were performed at baseline , median and at the end of the follow-up .

  11. 心力衰竭患者水钠潴留期间,中心静脉压升高,后者传至肾静脉和肾脏,导致肾功能障碍,可能是慢性心衰患者肾功能下降的发病机制之一。

    Heart failure in patients with sodium retention , elevated central venous pressure , which spread to the renal vein and kidneys , causing renal dysfunction may be one of the pathogenesis of chronic heart failure patients with reduced renal function .