心肌坏死

新西兰兔实验性高前壁心肌坏死的Wilson和头胸导联的定位诊断

Localization of acute experimental myocardial necrosis in high anterior wall by Wilson and head-chest lead

结论血浆BNP可以反映早发AMI患者心肌坏死的程度和预测近期预后。

Conclusion Plasma levels of BNP can reflect degree of myocardial necrosis and are associated with short-term prognosis in patients with PAMI .

大剂量维生素C对实验性心肌坏死的预防

Prevention of experimental myocardial necroses with large doses of vitamin C

PET高估心肌坏死范围，且不能判断心肌梗死的透壁程度。

PET slightly overestimate extent of necrosis myocardium and can 't distinguish transmural necrosis from subendocardial necrosis .

硒和维生素E与内皮素在克山病心肌坏死发生中的作用

Effect of selenium and Vitamin E on the endothelin concentration and their roles in developing myo - cardial necrosis of Keshan disease

我们研究了多普勒检测微栓塞是否与选择性PCI术后的心肌坏死有关。

We investigated whether the frequency of Doppler-detected microembolism is related to the incidence of myonecrosis during elective PCI .

远端器官缺血性预处理（RPC）可减少缺血再灌注后心肌坏死范围。

Remote organ ischemic preconditioning ( RPC ) may reduce heart infarct size following myocardium ischemia and reperfusion .

左或右室心肌坏死时R波振幅减少和ST段上抬的变化特点二种心电图相似。

S.The similar changes of the ST segment elevation and wave R decrease in the two kinds of ECGs at both of ventricle necrosis rabbits are observeded .

MCA可降低MIR血浆中ET和CK浓度，并减少MIR所致的心肌坏死。

MCA can decrease the contents of ET and CK in plasma and decrease myocardial infarction area in MIR .

结论：非溶栓治疗患者，WBC升高是心肌坏死程度指标。

The conclusion is that increase of WBC is the indicator of degree of myocardial necrosis of patients with unthrombolytic treatment .

实验结果提示，IL-8可以通过抑制白细胞粘附和浸润，抑制内皮素的释放，从而达到防治缺血心肌坏死。

It suggested that IL-8 could prevent and cure ischemic myocardial necrosis by inhibiting the adhesion and infiltration of leukocyte and release of ET.

利用心脏多普勒超声技术，研究小组进一步检测了在择期PCI过程中超声检测的微栓塞的发生率和心肌坏死的发生率之间的关系。

Using intracoronary Doppler ultrasound , the team investigated further by looking at the frequency of Doppler-detected microembolism in relation to the incidence of myonecrosis during elective PCI .

可以选择CK、LDH、FFA、SOD、MDA等生化指标以及心肌坏死面积等来探讨抗心肌缺血药物的作用机理。

Some biochemical indexes such as CK , LDH , FFA , SOD or MDA are also helpful in studying the mechanisms and effects of anti_myocardial ischemia drugs .

目的：比较新西兰兔实验性高前壁心肌坏死Wilson和头胸（head-chest，HC）导联的定位诊断。

Objective : Compare the localization of acute experimental myocardial necrosis in high anterior wall by Wilson and head-chest ( HC ) lead .

目的和方法：观察了碱性成纤维细胞生长因子（bFGF）对异丙肾上腺素（isoproterenol，ISO）引起的大鼠心肌坏死的拮抗作用及其机制。

AIM and METHOD : The effect of basic fibroblast growth factor ( bFGF ) on myocardial necrosis induced by isoproterenol ( ISO ) in rats was observed .

MCA+IP+MIR组为13.69±1.01%，较其他组明显减少（P<0.01）。结论（1）IP可使MIR过程中ET、CK浓度和ANP浓度增高，并减少MIR所致的心肌坏死。

Conclusion ( 1 ) IP can decrease the contents of ET and CK and increase the ANP in MIR contents , and decrease myocardial infarction area induced by MIR .

例4、5（扩张型心肌病。心电图示心肌坏死型）18F-FDG心肌代谢显像示浅淡缺损区，除极小部分放射充填外均未见放射性充填（非存活心肌-坏死或瘢痕形成）。

Case 4 and 5 suffering from dilated cardiomyopathy and myocardial necrosis in ECG showed non-alive myocardium-necrosis in most myocardial areas demonstrated by 18F-FDG myocardial metabolic imaging .

入选标准为患者持续性胸痛并经心电图、心肌坏死标记物检查诊断为NSTEMI。

The inclusion criteria was patients with persistent chest pain and confirmed NSTEMI diagnosed by the ECG and the myocardial necrosis markers .

而TNF-αmAb干预组小鼠死亡率明显减低，TNF-αmAb能减轻VMC的炎细胞浸润和心肌坏死。

Inflammatory cell infiltration and myocardial necrosis in the mice of TNF - α mAb intervention group were milder than simple VMC group apparently , death rate of mice was also lower .

以Se和VE联合缺乏变化最显著。结论Se和VE缺乏可诱导大鼠心脑组织TGF-β1mRNA表达上调，是缺血缺氧性心肌坏死和脑组织损伤的原因之一。

Conclusions Deficiency of Se and VE and increase the expression of TGF - β 1 mRNA , and it may be one of reasons of anoxemic myocardium necrosis and brain tissues lesion related with peripheral characteristic .

结论血浆H-FABP水平可作为心肌坏死或损伤的早期判断指标，可为急性胸痛患者的诊断提供依据。

Conclusion Plasma H-FABP level can be an early diagnostic marker for myocardial necrosis or damage within early stage , and may be applicable for use in the early diagnosis of acute chest pain .

结果大鼠预先给予孟鲁司特钠10或30mg·kg-1可降低血清LDH，CK，MDA含量，缩小心肌坏死面积。

Results Decreased serum level of LDH , CK , MDA and attenuated myocardial necrosis area were found in rats pretreated with Montelukast sodium 10 and 30 mg · kg - 1 . Montelukast sodium 30 mg · kg - 1 also enhanced NO content in myocardium .

结论CVB3m重复感染可导致急性期严重的心肌坏死，慢性期心肌胶原沉积和心肌纤维化。

Conclusion It strongly indicated that repetitive infection of CVB_ ( 3m ) could lead to serious heart injury in acute period and myocardium fibrosis in chronic period .

心肌坏死灶与冠状血管关系的研究

Studies on the Relationship between Myocardial Necrotic Foci and Coronary Vessels

感染中毒性和低血容量性休克病人的心肌坏死研究

Study on myocardial necrosis in patients with septic shock or hypovolemic shock

天冬氨酸钾镁盐对大鼠实验性心肌坏死的保护作用

Protective Effect of K-Mg-Aspartates on Experimental Myocardial Necrosis of Rats

延胡索乙素对异丙肾上腺素所致心肌坏死的保护作用

Protective Effects of dl-tetrahydropalmatine on Experimental Myocardial Necrosis by Isoprenaline in Rat

心肌坏死标记物在检测心肌坏死中的应用与地位

Application and status of cardiac muscle marker in examination of myocardial necrosis

干细胞治疗心肌坏死研究进展

Progress of stem cells therapy in treating myocardial necrosis

前言：目的：观察养心散对心肌坏死模型的疗效。

Objective : To observe the effect of Yangxin San on myocardial necrosis .