四氯化碳中毒

1起急性四氯化碳中毒事故的调查分析

Investigation and analysis on a case of acute carbon tetrachloride poisoning

结论养肝利胆颗粒加维生素C能显著改善四氯化碳中毒导致的小鼠慢性肝损伤。

Conclusion NLCG plus vitamin C can significantly improve chronic hepatic injury caused by carbon tetrachloride poisonining .

目的探讨养肝利胆颗粒与维生素C对小鼠慢性四氯化碳中毒引起肝损伤的保护作用。

Objective To observe the protection of nourishing liver and cholaneresis grains ( NLCG ) and vitamin C against mice chronic hepatic injury caused by carbon tetrachloride ( CCL ) poisoning .

建立四氯化碳中毒性大鼠肝纤维化模型，采用半定量Western免疫印迹方法，对肝纤维化不同阶段肝内MMP-3的酶原蛋白的表达水平进行了检测。

The carbon tetrachloride-induced hepatic fibrosis rat models were used . MMP-3 proenzyme protein expression in the different stages of liver fibrosis was measured by half quantitative analysis of Western blot .

四氯化碳中毒肝损害大鼠血清谷草转氨酶同工酶的变化

Changes of serum GOT isoenzymes in rats with subchronic CCl_4-induced hepatopathy

四氯化碳中毒性肝硬化肝胶原纤维的定量分析

A quantitative analysis on the collagenous fiber in liver with toxic cirrhosis induced by carbon tetrachloride

本文报导大白鼠经四氯化碳中毒及用3′-甲基-4-二甲基氨基偶氮苯诱发肝癌过程中肝脏鸟氨酸氨基甲酰移换酶活力的变化。

The change in activity of ornithine carbamyl transferase ( OCT ) in the liver of rats administered with carbon tetrachloride and 3 ' - methyl-4-dimethyl-aminoazobenzene ( 3 ' - Me-DAB ) was investigated .

大白鼠实验性肝病变的组织化学和生物化学研究Ⅰ.四氯化碳中毒后肝内脂肪、脂肪酶、核糖核酸和碱性磷酸酶的观察及血、肝内转氨酶活力的变化

HISTO-AND BIOCHEMICAL STUDIES ON EXPERIMENTAL LIVER DISEASE OF RAT I. Effect of CCl_4 Poisoning on the Fat , Lipase , RNA and Alkaline Phosphatase of Rat Liver with Special Reference to the Change of Transaminase Activity of Blood and Liver

DL-半胱氨酸对四氯化碳肝中毒小鼠超氧化物歧化酶和乳酸脱氢酶同功酶的影响

Effects of DL-cysteine on the SOD and LDH of Carbon Tetrachloride Hepatotoxicity in Mice

肝细胞生长素对四氯化碳慢性中毒性肝损伤保护作用的研究

Investigation On Protective Effect of Hepatocyte Growth Factor from CCl_4-Induced Chronic Toxic Liver Injury

京尼平甙和藏红花素对四氯化碳急性肝中毒小鼠的保护作用研究

The Hepatic Protective Effects of Geniposide and Crocin on Liver-injured Mice Induced by CCl_4

目的：探讨高血糖本身及其对四氯化碳（CCl4）中毒大鼠肝纤维化的影响。

AIMS : To explore the injury of hyperglycemia to liver and its influence on the progress of liver fibrosis in rats induced by carbon tetrachloride ( CCl4 ) .