内毒素休克

虎杖甙对内毒素休克大鼠肺组织中磷脂酶A2的影响。

Effect of polydatin on phospholipase A2 in lung tissues in rats with endotoxic shock .

不同容量血液滤过对内毒素休克继发MODS早期干预的基础与临床研究

The Basic and Clinical Research of Different Volume Hemofiltration in a Sheep Endotoxic Shock and MODS Model

内毒素休克狗肝脏蛋白激酶C活性的变化

Change of liver protein kinase C activity in canine endotoxic shock

内毒素休克狗血浆6-酮-前列腺素F（1α）含量的动态观察

Changes of Plasma 6-Keto-PGF_1 α Level During Canine Endotoxic Shock

蛋白激酶C对内毒素休克大鼠肝损伤诱导型一氧化氮合成酶表达的调控

Protein kinase-C on the expression of inducible nitric oxide synthase in rats with liver injury induced by endotoxin shock

地塞米松对家兔内毒素休克的防治作用及其与TNF的关系

Therapeutic and preventive effect of dexamethasone on endotoxin shock in rabbits and its relationship with TNF

大肠杆菌内毒素休克狗和暴发型流脑休克型患儿血浆前列腺素E和F（2α）含量的变化

Changes in Plasma Levels of Prostaglandin E and F_ ( 2 α) in E.coli Endotoxin-Shocked Dogs and Children Suffered from Epidemic Meningococcal Meningitis

抗炎药能抑制内毒素休克大鼠血浆CGRP浓度的升高

Antiinflammatory drugs inhibit the elevation of plasma CGRP concentration in endotoxin shock in rats

FK506预处理对内毒素休克小鼠及重症急性胰腺炎大鼠的保护作用

Effect of FK506 Pretreatment on Endotoxin Shock and Severe Acute Pancreatitis

结果：内毒素休克后，心肌组织、线粒体、SR钙含量明显增加。甲腈吡酮治疗后，上述指标进一步升高；

Results : The calcium content of the myocardium , mitochondria and SR was significantly increased in endotoxic rats .

方法：选用清洁级昆明小鼠腹腔注射脂多糖（LPS）制备内毒素休克动物模型，①观察内毒素休克前后Tβ4变化情况。

Methods : LPS ( lipopolysaccharide ) was intraperitoneally injected into clean Kunming mice to establish the animal model .

免疫抑制剂FK506预处理对内毒素休克的干预作用

Effect of FK506 pretreatment and its mechanism on endotoxin shock induced by LPS

清醒大鼠内毒素休克时体内降钙素基因相关肽（CGRP）改变的进一步研究

Further study of changes of calcitonin gene-related peptide ( cgrp ) during endotoxic shock in conscious rats

此外，内毒素休克引起的持续性低血压可导致心肌缺血和LPS对心肌的直接病理损伤。

Moreover , consecutive hypotension caused by endotoxin may lead to myocarditic ischemia and direct pathological damage of LPS to myocardium .

目的探讨内毒素休克发病机制中蛋白激酶G（PKG）的作用。

Objective To study the effect of cGMP-dependent protein kinase ( PKG ) on the pathogenesis of septic shock .

白藜芦醇苷上调Clara细胞分泌蛋白表达缓解大鼠内毒素休克性肺损伤

Polydatin up-regulate Clara cell secretory protein in lung injury of rats with an endotoxic shock

脂质体携带辅酶Q（10）（CoQ（10））对内毒素休克的治疗作用

The Therapeutic Effect of Coenzyme Q_ ( 10 ) Carried by Liposomes on Rat 's Endotoxin Shock

目的：观察多巴胺（DA）及去甲肾上腺素（NE）对内毒素休克犬血流动力学的影响。

Objective : To observe the effects of dopamine ( DA ) and norepinephrine ( NE ) on the hemodynamics in endotoxic shock dog .

CCK-8对内毒素休克大鼠肺脏细胞因子的抑制效应

Inhibitory effect of cholecystokinin-octapeptide on production of cytokines in the lung of endotoxic shock rats

结论FK506预处理可减轻内毒素休克所致的多器官损害，具有下调炎症反应的作用。

Conclusion FK506 pretreatment can decrease the multiple-organ damages caused by endotoxin shock and down-regulate the inflammatory reaction .

内毒素休克大鼠丝裂原活化蛋白激酶p38通路在生物喋呤诱生中的作用及其意义

Role and significance of p38 mitogen-activated protein kinase signal transduction pathway in biopterin induction in rats with endotoxic shock

提示绞股蓝有明显的抗内毒素休克，预防继发性DIC的作用。

The results suggest that GPs have a significant protective effect against endotoxic shock and prevent from subsequent DIC .

本文研究了内毒素休克时狗白细胞糖皮质激素受体（GCR）的变化。

We studied the changes in canine leukocytic glucocorticoid receptors during endotoxin shock .

目的：探讨内毒素休克家兔血浆及肝、脾、肾、心、肺脏等重要器官中内源性一氧化碳（CO）的含量变化以及血红蛋白氧合酶抑制剂&锌原卟啉对其的影响。

Objective : To investigate the changes in carbon monoxide in both plasma and various tissues , including liver , spleen , heart , lungs , and kidneys , in rabbits with septic shock .

结论PDS能够明显改善内毒素休克大鼠的低血压状态，降低NOS活性、NO含量，并对其NO的生成具有抑制作用。

[ Conclusion ] PDS can improve hypotension in septic shock rats , inhibit NOS activity and reduce NO content by inhibiting its production .

以内毒素（4mg·kg-1）复制内毒素休克模型，平均动脉压降至基础血压的2/3为休克状态。

Endotoxin shock models were duplicated by injection of LPS ( 4 mg · kg ~ - 1 ) .

内毒素休克早期，肺代谢TXA2，PGI2的失调是肺解聚功能受损原因之一。

This disturbance may be due to imbalance of the metabolism of TXA2 , PGI2 in the lung .

结论地塞米松对内毒素休克的防治效果与其直接抑制LPS诱导TNF的产生有密切关系。

Conclusion These results indicate that the preventive and therapeutic effect of dexamethasone on endotoxin shock , which may relate to its direct inhibition of the release of TNF induced by LPS .

目的：通过观察内毒素休克大鼠脑皮质中LPO含量、SOD活力和κBαmRNA表达及人参二醇组皂苷（PDS）对其的影响，探讨内毒素引起脑组织损伤的分子机制。

Objective To explore the molecular mechanism of brain tissue injury induced by endotoxin through observation of LPO content , SOD activity , and ⅰκ B α mRNA expression of cortex in endotoxic shock rats .

目的探讨不同剂量氢化可的松（hydrocortisone，HC）对内毒素休克早期大鼠内环境的影响。

Objective To investigate the effects of different dosages of hydrocortisone ( HC ) on the disorder of homeostasis in rats at early stage of endotoxic shock .