CLCA

Conclusion The goblet cell of asthmatic airway can synthesize GM-CSF , and one of mechanisms is the increased expression of CLCA .

结论哮喘气道上皮杯状细胞能合成GMCSF，特定CLCA表达升高是促进GMCSF合成的机制之一。

Objective To study the modulation alterations of calcium activated potassium channel ( Kca ) and calcium activated chloride channels ( Clca ) on detrusor instability ( DI ) and their roles in DI .

目的研究钙激活钾/氯通道对逼尿肌不稳定的调节作用的变化，探讨其在逼尿肌不稳定（detrusorinstability，DI）发生中的作用。

Objective To evaluate the inhibiting effect of niflumic acid ( NFA ), an inhibitor of calcium-activated chloride channel ( ClCa ) on airway epithelium , on the airway hyperresponsiveness in asthmatic mice .

目的观察钙激活Cl-通道（ClCa）阻断剂尼氟灭酸（NFA）对支气管哮喘（简称哮喘）气道高反应性的抑制作用。

Objective To evaluate the ability of goblet cell in the airway in rat with asthma to synthesize granulocyte-macrophage colony-stimulating factor ( GM-CSF ), and the role of calcium-activated chloride channel ( CLCA ) in the synthesis .

目的观察哮喘小鼠气道上皮杯状细胞合成粒细胞/巨噬细胞集落刺激因子（GMCSF）的能力，探讨钙激活Cl-通道（CLCA）在合成中的调控作用。

Objective To study the modulation of calcium activated potassium channels ( Kca ) and calcium activated chloride channels ( Clca ) on the contraction of detrusor strips , and to investigate the role of these channels play in DI .

目的探讨钙激活钾通道和氯通道对逼尿肌条收缩的调节在逼尿肌不稳定发生中的作用。