高氧
- hyperoxia
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高氧对早产新生大鼠肺组织细胞色素b、三磷酸腺苷合成酶6表达的影响
Effect of Hyperoxia on Expression of Cytochrome b and Adenosine Triphosphate Synthase 6 in Premature Newborn Rats ′ Lung
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Notch信号在新生鼠高氧肺损伤中的表达
Expression of Notch in hyperoxia induced lung injury in newborn rat
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超市生鲜猪肉高氧MAP气调保鲜包装研究
Study on High-Oxygen Modified Atmosphere Packaging for Fresh Pork in Supermarket
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高氧压合成产物La2CuO(4+δ)的相变研究
The phase transition of la_2cuo_ ( 4 + δ) synthesized in high oxygen pressure
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利用浮区法在高氧压下生长ZnO晶须
Growth of ZnO ( Wurtzite ) Whiskers Using the Floating Zone Method at High Oxygen Pressure
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60%~100%高氧处理还可显著抑制果实失重,保持较高的果肉硬度和维生素C含量。
Fruits treated with O_2 concentrations of 60 % or higher also exhibited significantly higher fruit firmness value , vitamin C content and less weight loss .
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方法SD早产鼠80只随机分为对照组和高氧组,于1、71、42、1d行肺组织病理学检查;
Methods Preterm rats were randomly devided into control and hyperoxia group .
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La(2-x)SrxCuO4的高氧压处理与XPS研究
High oxygen pressure treatment and XPS Study for la_ ( 2-x ) sr_xcuo_4
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维甲酸对高氧暴露下原代培养的胎鼠肺泡II型上皮细胞和成纤维细胞增殖与凋亡的影响
Effect of Retinoic Acid on the Primary Rat Embryonic Type II Alveolar Epithelial Cell and Lung Fibroblasts Proliferation and Apoptosis Exposed to Hyperoxia
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结果高氧暴露的SD大鼠肺组织可见水肿、大量炎症细胞浸润等急性炎症反应。
Results After hyperoxia exposure , there were edema and inflammatory cell infiltration in the lung tissues of SD rats .
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前列腺素E1在高氧肺损伤中的保护作用
Lung protective effect of prostaglandin E1 in acute lung injury induced by hyperoxia
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ICAM-1在高氧肺损伤新生大鼠模型肺组织中的表达及意义
Expression of ICAM-1 in the Lung of Premature Neonatal Rats Exposed to Hyperoxia and Its Significance
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结果:高氧组TGF-β1的mRNA水平从第3天开始升高,随暴露时间延长逐渐上升,到第14天仍持续不降;
Results : The level of TGF - β 1mRNA in hyperoxia group increased from the 3rd day , and rose gradually until the 14th day .
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异丙酚对高氧肺损伤大鼠氧自由基、IL-8及肺组织超微结构的影响
Influence of propofol on oxygen-derived free radicals , il-8 and ultrastructure changes of rat lung induced by hyperoxia
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金属性氧化物LaCuO3的高氧压合成及Cu~(3+)态的XPS研究
The high oxygen pressure synthesis of metallic lacuo_3 and the XPS Study on cu ~ ( 3 + ) state
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【结果】高氧液实验组血中MDA含量明显低于对照组(P<0.05);
【 Results 】 The contents of MDA of hyperoxia solution group were lower significantly than those of control group ( P < 0.05 );
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维甲酸下调高氧暴露下胎鼠肺成纤维细胞及Ⅱ型肺泡上皮细胞MMP-2表达
Retinoic acid down-regulates the hyperoxia-induced expression of MMP-2 in fetal rat lung fibroblasts and type ⅱ alveolar epithelial cell
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高氧包装鸭肉制品贮藏期间微生物数量和TBA值的变化
Changes of Microorganism and TBA Values of High Oxygen Packaged Duck Meat Products During Storages
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方法早产新生SD(Sprague-Dawley)大鼠生后1d随机分为空气组、高氧组,高氧组持续暴露于常压氧舱中,氧浓度>85%;
Methods One-day-old preterm Sprague-Dawley ( SD ) rats were randomly divided into air group and hyperoxia group .
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目的研究γ干扰素(IFNγ)对新生大鼠高氧肺损伤的影响,以观察转化生长因子transforminggrowthfactorβ(TGFβ)在新生大鼠高氧肺纤维化中的作用。
Objective To study effect of IFN γ on hyperoxic lung injury in neonatal rats and to investigate the role of transforming growth factor β( TGF β) in hyperoxic lung fibrosis .
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结果高氧暴露72h即可建立肺损伤模型。
Results Hyperoxia-induced lung injury model were established successfully after 72 h by exposure to hyperoxia .
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肺组织TGFβ1表达增强以及肺泡巨噬细胞表达TGFβ1与高氧肺损伤可能有内在联系。
It is possible that stronger TGF β 1 expression in the lung and its expression in alveolar macrophages have relations with lung injury induced by hyperoxia .
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并发ARDS患者的氧缺陷表现为高氧耗型,并可能与TNF介导的系统性炎症反应有关。
Oxygen deficit in patients with ARDS were manifested by high O 2 , which may be related to systemic inflammatory response mediated by TNF .
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高氧暴露7、14d组肺湿/干重比值均明显高于空气组(P0.05);
The lung wet / dry weight ratio of hyperoxia group were much higher than the room-air group at 3,7,14 days .
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结论OPN通过促进TIMP的表达而抑制MMP的释放和激活,从而减轻高氧所致的ALI。
Conclusion OPN can protect against hyperoxia-induced ALI by promoting the expression of TIMP and inhibiting the activation of MMP .
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高氧吸入对剖宫产母婴8-异前列腺素F2α含量的影响
Effect of high inspired oxygen on the concentration of 8-isoprostane F 2 α in maternal and fetal blood during caesarean section
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结论氧化应激与高氧肺损伤密切相关,NAC对高氧肺损伤的氧化应激作用具有明显的干预作用,其作用具有剂量依赖性。
NAC has interventional effect on the pathological process of hyperoxia-induced lung injury and its protection effect was dose dependent .
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高氧致慢性肺疾病早产鼠肺组织CDK4、P21的表达及意义
The Expression and Effect of CDK4 、 P21 in Lung Tissue in Premature Rats with Hyperoxia-induced CLD
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MG-132对高氧肺损伤中细胞凋亡的保护作用及对p38信号通路的影响
Protective effects of MG-132 on p38 signaling pathway and cell apoptosis in lung injury induced by hyperoxia
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目的高氧肺损伤是导致支气管肺发育不良(BPD)的最常见原因。
Objective Hyperoxia-induced lung injury is the most common cause of bronchopulmonary dysplasia ( BPD ) in neonates .