膜去极化

ACh的膜去极化作用不依赖于细胞外Ca（2＋），而可被河豚毒（TTX）阻断；

The ACh-induced membrane depolarization was Ca2 + - independent and blocked by TTX .

环腺苷酸（cAMP）抑制凋亡、ROS急剧减少、线粒体膜去极化；

PKC favored , and cAMP inhibited the macrophage apoptosis and the rapid drop in ROS and mitochondrial membrane depolarization .

信使分子影响线粒体生成ROS的变化以及线粒体膜去极化从而影响巨噬细胞凋亡。

The signal molecules affected apoptosis by modulating ROS decline and mitochondria depolarization .

达托霉素能钙依赖性的使细菌细胞膜去极化并导致细胞膜电位的丢失，抑制DNA、RNA和蛋白的合成从而导致细胞死亡。

Daptomycin exhibits Ca-dependent depolarisation of the bacterial membrane resulting in loss of membrane potential leading to inhibition of DNA , RNA and protein synthesis which results in cell death .

前列腺素F（2α）增强膜去极化和升高胞内钙促进NIT-1β细胞胰岛素分泌

Potentiative effect of prostaglandin F_ ( 2 α) on glucose-induced insulin secretion through membrane depolarization and elevation of intracellular calcium in NIT-1 β cells

PGF2α可能通过激活氯通道，增强NIT-1β细胞膜去极化，升高细胞[Ca2+]i，促进高浓度葡萄糖刺激下的胰岛素分泌。

CONCLUSION Potentiation of PGF_ 2 α on the glucose-induced insulin secretion may be mediated by membrane depolarization through activation of chloride channels and elevation of [ Ca ~ ( 2 + ) ] _i .

烟碱样乙酰胆碱受体（nAChR）的表达调控受神经电活动影响，电刺激引起肌细胞膜去极化可抑制nAChR的表达。

It is well documented that electricity regulates the expression of nicotine acetylcholine receptor ( nAChR ) . Electricity induced depolarization inhibits the expression of nAChR , in which calcium , protein kinase C and myogenin play major roles .

它与胰岛β细胞的65-KDa蛋白结合，促使细胞表面敏感的ATP-K~+通道关闭，细胞膜去极化，从而导致胰岛素的释放。

Once GM bonds to 65-KDa protein located on the surface of 3 cell of pancreas islet , that will cause the closing of ATP-K + channel located on the cell membrane and resulting in the membrane 's depolarization ? and consequently insulin is released .

骨骼肌细胞膜去极化对葡萄糖转运子4转位的调节作用

Mechanism of the depolarization-regulated glucose transporter 4 translocation in skeletal muscle cells

神经冲动、激素及器官特异刺激物可以引起细胞膜去极化。

Nerve impulses , hormone and organ-specific irritant can cause cell membrane depolarization .

摸上去非常软，心肌收缩力下降。已经确证，心肌收缩始于动作电位使肌膜去极化。

It felt very flabby , and the myocardium was poorly contractile . It is well established that the cardial contraction is initiated when an action potential depolarizes the sarcolemma .

其反转电位与钾电流相似。急性分离的大鼠皮层神经细胞膜去极化激活的优降糖敏感钾单通道电流

Adenosine suppress spontaneons activity by hyperpolarized membrane potential and induce a time independent potassium current in AV node cell . CURRENTS OF GLIBENCLAMIDE-SENSITIVE k ￣ + SINGLE CHANNEL ACTIVATED BY DEPOLARIZATION IN ACUTELY DISSOCIATED CEREBRAL NEURONS OF RATS

结论甘氨酸对缺氧心肌细胞有保护作用，其可能的机制是甘氨酸与其受体结合后，减轻了心肌细胞膜去极化，从而减少了细胞膜电压依赖性钙通道开放，使钙离子内流减少。

The underlying mechanism may involve attenuation of membrane potential depolarization after hypoxia by conjugation of glycin with its receptor , depleting in turn voltage-dependent calcium channel on the cellular membrane , preventing calcium overload due to influx of calcium ions after hypoxia .

豆科植物感知结瘤因子，改变一系列与结瘤相关基因的表达，引起表皮细胞膜去极化，细胞质钙离子浓度急增，根毛变形和皮层细胞的分裂，最终形成根瘤。

Perception of Nod factors by plant induces genes expression relative to nodulation and leads to depolarization of the plasma membrane , Ca2 + spiking in cytosolic , root hair deformation and cortical cell division that ultimately result in development of the nodule .

NE诱发的膜电位去极化有浓度依赖性且可被酚妥拉明阻断。

NE could induce membrane depolarization in a concentration dependent manner and could be blocked by phentolamine .

这种钾通道在低于阈电位时开始被激活，产生IA抵消了刺激电流，去极化几乎被抑制，延长膜电位去极化。

This potassium channel below the threshold potential is activated when you start to produce export-oriented IA stimulus offset current , the depolarization was almost inhibited , extended membrane potential depolarization .

统计分析提示，糖氧剥夺能损害线粒体膜电位去极化。

Oxygen-glucose deprivation can lead to the mitochondrial membrane potential depolarization .

虎杖甙可引起腹腔巨噬细胞膜部分去极化

Effects of polydatin on membrane potential in intraperitoneal macrophages

线粒体膜电位快速去极化，NADPH氧化酶活性剧降，ROS快速减少，ROS清除剂促进凋亡；

Mitochondria membrane depolarized quickly , the activity of NADPH oxidase declined sharply , and ROS production decreased rapidly .

目的研究梭曼引起神经肌接头突触后膜持续性去极化（SD）的产生机制。

Objective To study the mechanism underlying the sustained depolarization ( SD ) of neuromuscular junction induced by soman in isolated non-uniform stretched muscle preparation of rat diaphragm .

结果表明，HCG对卵母细胞膜电位有去极化作用，酪氨酸可以使膜电位呈现超极化反应，并能抑制HCG的去极化作用。

The results showed that HCG depolarised membrane potential of toad oocytes , while tyrosine directly caused membrane , hyperpolarization , and inhibited the depolarization induced by HCG .

结果：与空白对照组相比，5-FU能明显抑制Tb细胞的增殖活性，并且可使细胞膜电位发生去极化改变，跨膜钾电流显著降低。

RESULTS : Compared with the control group ( CON ), 5-FU inhibited proliferation of the Tb cells . At the same time the membrane was depolarized and the potassium channel current significantly decreased compared with the CON group .

随后，突触后膜开始逐渐去极化，最大去极化27.0mV。62.7min后小终板电位（mEPP）频率逐渐增高，到70.3min达到最高频率，比给药前增加了32倍。

Hereafter postsynaptic membrane potential was depolarized gradually , and the maximum depolarization value was 27 . 0 mV . The frequency of mEPP increased markedly after 70 . 3 min MTX administered , 32 folds compared with control .

梭曼引起大鼠神经肌接头突触后膜持续性去极化的机制

Mechanism underlying the sustained depolarization of neuromuscular junction induced by soman in rats

然而卵母细胞成熟后，膜电位明显去极化。

However , when oocytes had matured , the membrane potential got significantly depolarized .

结论：一定浓度的虎杖甙能引起腹腔巨噬细胞细胞荧光值升高即膜的部分去极化。

It is concluded that Polydatin can lead to partial membrane depolarization of intraperitoneal macrophages .

其动作电位的幅度没有显著变化，然而静息膜电位明显去极化，动作电位时程明显延长。

Action potential amplitude of transplant myocytes was not significantly different , but the resting membrane potential was reduced and action potential duration was prolonged compared to control myocytes . 3 .

结果对12个SON细胞局部压力喷射微量谷氨酸，均诱发伴有膜电阻减小的去极化反应，并呈剂量和膜电位依赖性特征。

Results Local pressure ejection of glutamate induced depolarizing responses with decrease of input resistance , concentration - and membrane potential-dependent properties in 12 SON neurons .

BTA能在金属表面吸附成膜，阻止了去极化物质向金属表面迁移，减缓了金属的腐蚀速率。

Moreover , BTA could form protective film on metal surface which prevented depolarizing substances to transfer toward metal surface , and reduced corrosion rate of metal .

二是紫外线和特殊波长对色素分子影响，使其产生游离基因化合物，而游离基因能干拢细胞膜、线粒体膜、核酸，去极化胶原和玻尿酸。

Effect of pigment molecule of special wavelength and ultraviolet produces free genome compound that interferes with cell membrane , bioblock membrane , nucleic acid , depolarization collagen and atidase .