腹腔感染
- 网络abdominal infection;intra-abdominal infection;iai
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大黄素对腹腔感染大鼠血浆白蛋白及TNF-α、IL-6影响的实验研究
Effect of Emodin on Serum Albumin and TNF - α、 IL-6 of Abdominal infection Rats
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腹腔感染所致多器官损害与Toll样受体2基因表达的关系
The relationship between gene expression of Toll-like receptor 2 and multiple organ dysfunction in rats after abdominal infection
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口服高渗氯化钠维生素C液对腹腔感染大鼠肠屏障的保护作用
Influences of oral hypertonic saline with vitamin C on gut barrier function of rats with abdominal infection
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腹腔感染后抗氧化能力对血浆维生素A、E、C水平的影响
The influence of antioxidation on plasma vitamin A , E , C following intraabdominal sepsis in rats
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全肠外营养加用维生素A、E、C对严重腹腔感染大鼠肠屏障功能的影响
Influence of vitamin A 、 E 、 C-added TPN on barrier of small bowel mucosa in severely intraabdominal septic rats
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腹腔感染伴多器官功能障碍综合征时抗TNF抗体代谢调理作用的实验研究
An experimental study of metabolic intervention of anti-TNF antibody in intra-abdominal infection complicated by multiple organ dysfunction syndrome
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结论TPN添加维生素A、E、C能改善腹腔感染后机体的抗氧化能力,减轻脂质过氧化损伤。
Conclusion Vitamins A , E , C with TPN may effectively ameliorate the oxidative injuries in peritoneal sepsis .
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抗TNF抗体防治腹腔感染诱发多器官功能障碍综合征的作用
The role of anti-tumor necrosis factor antibody in the treatment of multiple organ dysfunction syndrome induced by intra-abdominal infection
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目的观察完全胃肠外营养(TPN)添加维生素A、E、C对腹腔感染后抗氧化能力的影响。
Objective To study the effects of vitamins A , E , and C with TPN on antioxidation in peritoneal sepsis .
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氟嗪酸(Ofloxacin)治疗复杂性尿路感染和腹膜透析腹腔感染
Ofloxacin Treatment of the Complicated Urinary Tract Infections and CAPD Peritonitis
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病理状态下大鼠腹腔感染前后TNF-α和IL-6基因表达的实验研究
Experimental Study on the TNF - α and IL-6 Gene Expression in Pathological Rat Before and After Celiac Infection
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严重腹腔感染大鼠组织Toll样受体2/4基因表达及其调节机制
Changes in Toll-like receptor 2 and 4 gene expression in vital organs in septic rats and their regulation mechanisms
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结论:TPN加用维生素A、E、C对严重腹腔感染大鼠肠屏障功能有一定的保护作用。
Conclusions : These results suggest that vitamin A 、 E 、 C added TPN can effectively protect the barrier of gut mucosa in infected rats .
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结果:SAP早期手术和延期手术的腹腔感染发生率分别为76.71%和21.82%(P<0.005);
Results : The infection rates of early operations and postponed operations are 76.71 % and 21.82 % respectively ( P < 0.005 ) .
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结论联合使用环氧化酶抑制剂Asp和β-内啡肽受体拮抗剂Nal可以调理腹腔感染大鼠TPN时的蛋白质代谢,减少蛋白质丢失。
Conclusion Asp combined with Nal may intervene protein metabolism in TPN and reduce protein loss in sepsis .
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免疫抑制大鼠腹腔感染致MODS发生机制及氨茶碱治疗的实验研究
Experimental Study on the Mechanisms and the Therapeutic Effect of Aminophylline for MODS Induced by Peritonitis in Rats with Immunosuppression
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本研究旨在探讨重组人生长激素(rhGH)对腹腔感染引起的低白蛋白血症的治疗作用。
The present study was aimed to explore the effect of recombinant human growth hormone ( rh GH ) on hypoalbuminemia in peritoneal sepsis .
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方法采用大鼠盲肠结扎并穿孔(CLP)造成腹腔感染。
Methods The cecal ligation and perforation ( CLP ) was utilized to make the abdominal infection in rats .
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ERCP操作使胆汁内细菌阳性检出率增高,并可能是胰头癌根治术后腹腔感染的潜在危险因素。
ERCP caused positive bile culture and may be potential risk factor of intra-abdominal infection after radical resection of pancreatic adenocarcinoma . 3 .
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结论:大黄素能减轻CLP所致腹腔感染大鼠的炎症反应和肠粘膜的损伤。
Conclusion Emodin can reduce the inflammatory response and extenuate the damage to intestinal mucosa in abdominal infection of rats .
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结论:1.不同年龄段腹腔感染大鼠脾切除前后TNF-α、IL-10的变化水平基本一致。
For celiac infection rats of different ages , the changes in their TNF-a and IL-10 level before and after splenectomy are very similar . 2 .
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脾切除导致腹腔感染晚期大鼠体内TNF-α降低及IL-10的升高,使其更易发生免疫抑制。
The reduction of TNF-a and increase of IL-10 level in celiac infection rats of late stage are more likely to induce immunosuppression after splenectomy . 3 .
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结论一定剂量的KET(50mg/kg)可显著降低腹腔感染脓毒症大鼠死亡率。
Conclusions Ketamine at a given dose can decrease significantly the mortality of septic rats .
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目的探讨Toll样受体2(TLR2)基因表达与严重腹腔感染所致多器官损害的关系。
Objective To investigate the relationship between gene expression of Toll-like receptor 2 ( TLR2 ) and multiple organs dysfunction after severe abdominal infection .
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应用盲肠结扎穿孔法(CLP)制备腹腔感染大鼠模型,同时给予外源性生长激素;
The intra_abdominal infection model was made by cecal ligation and puncture ( CLP ) and the infected rats were treated with exogenous growth hormone .
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腹腔感染致呼吸窘迫综合征的动物模型Gq蛋白及RGS在急性呼吸窘迫综合征中的作用机制研究
Respiratory distress syndrome due to intraperitonealinfection : Study in animal models The Mechanism of Gq Protein and RGS on ARDS
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所有患者由ICU医生行重要器官功能支持治疗;对有明确腹腔感染病灶,经非手术治疗效果不理想或病情加重的患者进行外科干预。
All patients by doctor of ICU support for life and organ function ; the lesions of abdominal infection , surgical intervention by non operation treatment effect is not ideal or sicker patients .
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目的了解广州地区腹腔感染产ESBLs大肠埃希菌和肺炎克雷伯菌的耐药性及ESBLs基因型分布。
Objective To investigate the drug resistance of Escherichia coli and Klebsiella pneumoniae producing extended-spectrum β - lactamases ( ESBLs ) in intraabdominal infections and analyze the genotypes of ESBLs .
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我们应用腹腔感染诱发多器官衰竭(MOF)的动物模型观察不同热量全肠外营养(TPN)对MOF兔的器官功能、氮平衡和蛋白质分解代谢的影响。
Rabbits with MOF induced by intraab-dominal sepsis were used to observe the effect of TPN with different amount of calorie on organ function , nitrogen balance and protein catabolism .
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目的:观察抗TNF抗体对腹腔感染(IAI)伴多器官功能障碍综合征(MODS)时的代谢调理作用。
Objectives : To observe the effect of metabolic intervention of anti TNF antibody on the hypermetabolism occurred in intra abdominal infection ( IAI ) complicated by multiple organ dysfunction syndrome ( MODS ) .