缺氧
- hypoxia;anoxia;oxygen deficit;oxygen lack;with meagre oxygen
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[anoxia;hypoxia;with meagre oxygen] 机体组织在得不到充分氧或不能利用氧时所产生的病理现象
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人重组白细胞介素-6可减少缺氧-复氧后大鼠海马培养神经元的DNA损伤
RhIL-6 can decrease DNA damage of cultured rat hippocampal neurons after anoxia and reoxygenation
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结论:脂肪肝CT表现分型与高海拔缺氧明显关系。
The CT findings of steatohepatitis are closely related to anoxia in high altitude area .
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缺氧可能阻碍胎儿的大脑发育。
A lack of oxygen may inhibit brain development in the unborn child .
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人的大脑缺氧4分钟后便会造成永久性损伤。
The human brain needs to be without oxygen for only four minutes before permanent damage occurs .
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缺氧的大脑会迅速受损。
Without oxygen the brain is damaging quickly .
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遮荫树的消失已经造成了世界气候的轻微变化,研究表明,树木缺氧也会导致空气污染和全球变暖。
The loss of shade trees is already causing a slight change in the world 's climate , and studies show that loss of oxygen-giving trees also leads to air pollution and global warming .
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图像对诊断无影响。结论低剂量CT在早期诊断新生儿缺血缺氧性脑病有重要价值
Conclusion low dose CT have important value in the diagnosis of HIE in neonate
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新生儿缺氧缺血性脑病CT表现对预后的评估
CT feature and evaluation hypoxic-ischemic encephalopathy in neonates
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内源性CO在缺氧性肺动脉高压大鼠肺血管重构中的作用
The role of endogenous carbon monoxide in vascular remodeling in hypoxic pulmonary hypertension
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本文主要探讨NF-κB在缺血缺氧性脑损伤中扮演的角色。
We will discuss the role of NF - κ B for brain hypoxia - ischemia in this article .
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低温协迫导致淡水白娼脑部缺氧,SOD活性持续上升。
Low temperatures may resulted in fish hypoxia in brain , showed enhanced SOD activity .
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新生鼠缺氧缺血脑损伤脑组织中巨噬细胞炎性蛋白1-αmRNA的表达及意义
Expression and Significance of mRNA for MIP-1 α in Cerebral Tissue of Newborn Rat with Hypoxic-Ischemic Brain Damage
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慢性缺氧大鼠肺内(肺血管壁)细胞增殖、凋亡及c-myc、p53基因表达研究
The study of cell proliferation apoptosis and expression of c-myc and p53
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ICA能显著抑制缺氧诱导的内皮细胞凋亡。
ICA could significantly inhibit the hypoxia induced apoptosis of VECs .
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方法:分析58例缺氧缺血性脑病患儿的CT表现,参照诊断标准,以病变累及程度将脑损伤程度进行CT分度、临床分度。
Methods : Among 58 cases of HIE , CT findings and clinical manifestations were analysed by the degree of the injury of brain .
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应用ELISA法检测正常和缺氧状态下牛视网膜血管内皮细胞VEGF的表达。
By using ELISA method , we explored the expression of VEGF under normal and hypoxic condition .
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围产期缺氧缺血(Hypoxiaischemia,HI)是造成新生儿脑损伤的首要原因。
Perinatal hypoxia – ischemia ( HI ) is a significant primary cause of brain damage of neonates .
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结果口服GSH可明显提高小鼠耐缺氧能力;
Results The hypoxia - tolerance of mice was increased .
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目的:研究NGF对化学性缺氧神经细胞的保护作用。
Objective : To determine whether NGF has protective effects on chemical hypoxic neurons .
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缺氧通过影响HIF-1蛋白表达,调控下游靶基因VEGF转录。
Hypoxia can up-regulate the expression of HIF-1 protein , which activates the transcription of VEGF .
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PGE1对COPD合并缺氧性肺动脉高压的疗效
The Efficacy of Prostaglandin E1 in the Treatment of COPD with Pulmonary Artery Hypertension
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最佳缺氧处理时间为2h;
The best anoxia processing time is 2 h.
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缺氧与非缺氧环境中肝癌患者炎性标志物CRP的实验研究
An inflammatory marker in patients with liver cancer : comparative study of CRP under hypoxic and non-hypoxic environment
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MID患者CSF中TNFα、NSE含量变化可能客观地反映患者脑细胞缺血缺氧性损害。
The changes of TNF α, NSE concentrations in CSF could reflect the brain cell ischemic lesion objectively in patients with MID .
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目的:观察川芎嗪对慢性缺氧大鼠肺动脉蛋白质激酶C(PKC)表达的影响。
Objective : To explore the effect of tetramethylpyrazine ( TMP ) on protein kinase C ( PKC ) expression of pulmonary arteries in chronic hypoxic rats .
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其机理可能是为DL阻滞肾上腺素能受体及改善能量代谢,从而提高了海马脑片抗缺氧缺糖损伤的能力。
The mechanism may be DL block of α and β receptor and improve of the energy metabolism .
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Western-Blot法检测离体缺氧心肌细胞内COX-1、2不同表达。
The expression of COX-1 , - 2 were analyzed by Western-Blot .
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目的探讨不同葡萄糖水平对缺氧缺血(HI)新生大鼠脑病理改变的影响。
Objective To explore the influence of different blood glucose levels on cerebral pathology under light microscopy in neonatal rats with hypoxic-ischemic ( HI ) .
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目的观察早期干预对防治缺氧缺血性脑病(HIE)病儿脑功能障碍的效果。
Objective To study the effect of early medical intervention on neonates with hypoxic ischemic encephalopathy ( HIE ) .
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背景与目的:急性脑梗死(acutecerebralinfarctionACI)又称缺血性脑卒中(cerebralischemicstroke),是指各种原因引起的脑部血液供应障碍使局部发生不可逆性损伤,导致组织缺血、缺氧性坏死。
Background and object : ACI ( acute cerebral infarction ) or cerebral ischemic stroke refers to irreversible local lesion caused by dysfunction of brain perfusion and successive ischemic-hypoxic necrosis .