糖原合成

肝再生修复过程中雌性鼠肝糖原合成能力比雄性鼠强。

The glycogen synthesis capacity in female mice is stronger than that of male mice during liver regeneration .

探讨格列美脲（GM）在促进正常大鼠骨骼肌糖原合成中对钙调素（CaM）信号的作用。

To investigate the effect of calmodulin ( CaM ) signal system on the glycogen synthesis in skeletal muscle cells from glimepiride ( GM ) - treated normal rats .

蛋白激酶B与糖原合成激酶3

Protein Kinase B and Glycogen Synthase Kinase 3

本文以大鼠为实验对象，主要从机体运动后的糖原合成与运动中葡萄糖摄取方面来探讨人参皂甙Rg1的作用机制。

This paper mostly probed into the mechanism of ginseng Rg1 from glycogen synthesize after exercise and glucose ingestion during exercise .

此外，GM还能增加周围组织对Ins的敏感性，使周围组织对葡萄糖摄取、利用增加，脂肪和糖原合成增加。

In addition . GM may make the peripheral tissues to improve the sensitivity to insulin , to incease the intape and using of glucose and increase the synthesis of fat and glycogen .

转染脂联素基因对C2C12肌细胞葡萄糖氧化和糖原合成无显著性影响。

Transfection with adiponectin cDNA had no significant effect on the glucose oxidation and glycogen synthesis of C2C12 myotubes .

结论运动可下调胰岛素抵抗大鼠脂肪组织GSK-3β的表达，促进葡萄糖的摄取及糖原合成，县有改善胰岛素抵抗功能。

Conclusion ET can ameliorate IR by decreasing GSK 3 β protein expression in adipose tissues and enhancing the ingestion of glucose and the synthesis of glycogen .

钙调素信号系统在格列美脲促进糖原合成中的作用

Effect of calmodulin signal system on glycogen synthesis promoted by glimepiride

低氧和/或运动对肌糖原合成的影响及其机制探究

Effects of Hypoxia and Exercise on Muscle Glycogen Synthesis and Its Regulating Mechanism

钙调素在格列美脲促进正常大鼠骨骼肌细胞糖原合成中的作用

The Effect of Calmodulin on Glycogen Synthesis in Glimepiride-Treated Skeletal Muscle in Normal Rats

补糖与运动后糖原合成

Carbohydrate feedings and glycogen synthesis after exercise

富铬酵母降糖作用机制可能是通过促进肝组织和周围组织的糖原合成、葡萄糖利用等类胰岛素样作用来实现的；

May be its mechanism of decreasing blood sugar was the insulin kind function of promoting synthesis of hepatic glycogen and utilization of glucose etc ;

研究表明，无机钒化合物和有机钒化合物均可改善糖尿病大鼠的高血糖症状，促进葡萄糖转运和糖原合成。

The results show that both inorganic vanadium complexes and organic vanadium complexes can increase glucose level of diabetic rats , promote glucose transport and glycogen synthesis .

肌酸补充后骨骼肌糖代谢能力增强，主要表现为血糖降低、骨骼肌糖原合成增强、骨骼肌葡萄糖转运蛋白4的表达上调，以及柠檬酸合酶活性升高。

The improvement of glucose metabolism after creatine supplement is associated with lower level of blood glucose , glycogen synthesis improvement , up-regulation of glucose transporter 4 and increased citrate synthase activity .

结果化合物G003显著降低正常小鼠空腹血糖水平，刺激离体胰岛分泌胰岛素，显著促进脂肪细胞转运和氧化利用葡萄糖，对离体肝细胞的糖原合成无明显影响。

Results Compound G-003 markedly decreased the levels of fasting plasma glucose of normal mouse , stimulated isolated pancreatic rat islets to release insulin and increased glucose transport and utilization in isolated rat adipocytes . Compound G-003 has no effect on glycogen synthesis in isolated mouse hepatocytes .

ELISA法检测培养上清白蛋白浓度，PAS染色法检测糖原的合成。

Detecting the level of albumin in supernatant and the synthesis of glycogen by the methods of ELISA and PAS-dying respectively .

胶原蛋白多肽-铬（Ⅲ）螯合物可以显著地降低血糖，增加肝糖原的合成及葡萄糖激酶的活性。

CPCC could markedly decrease blood glucose , increase liver glycogen contents and improve liver glucokinase activity .

实验结果提示葡萄糖酸铬可能通过促进肝糖原的合成和周围组织中葡萄糖的利用起到抗糖尿病作用。

The experimental result indicated that chromium gluconate had effect of anti-diabetic , maybe by facilitating synthesis of hepatic glucogen and glucose utilization of peripheral tissue .

研究资料表明，胰岛素作为体内最重要的合成激素之一，具有促进肝糖原的合成和葡萄糖的转运、增加蛋白质合成和脂肪细胞的脂质贮存的作用。

Studies indicate that insulin , one of the most important anabolism hormone , can promote the synthesis of the hephormone and transportation of the glucose , and increase the synthesis of protein and storage of the lipid in the adipocyte .

当机体进入代谢起涨期后，糖原分解殆尽，糖原合成不足，此时血糖升高主要来源于糖异生，糖摄取利用障碍也可能是原因之一。

When it gradually comes to theflowphase ( a period of high metabolism reactions ), as glycogenolysis is almost exhausted and glycogenesis becomes insufficient , the rising blood glucose mainly results from glyconeogenesis , and may also be accompanied by the inhibition of glucose utilization .

其降血糖的胰内机制是促进胰岛β细胞分泌胰岛素，抑制胰岛α细胞分泌胰高血糖素，胰外机制可能是抑制肝糖原分解和促进肝糖原合成。

Its mechanisms are stimulating the secretion of insulin from β cells and inhibiting the secretion of glucagons from α cells , and it can probably decrease the decomposition of liver glucogen and increase the synthesis of liver glucogen .

糖原合成酶激酶（GSK3）是一种高度保守的丝氨酸苏氨酸蛋白激酶，在动物中参与诸如糖原合成、胰岛素调节、多种蛋白的转录激活和发育调控等许多生命活动的信号转导。

Glycogen synthase kinase 3 ( GSK 3 ) / SHAGGY is a highly conserved serine / threonine protein kinase implicated in many signaling pathways in animals .