氯化锂

在清醒自由活动状态的大鼠上，肌醇磷酸酶抑制剂氯化锂、M受体激动剂氧化震颤素和N受体激动剂烟碱均可剂量依赖性地降低体温；

In conscious rats , inositol phosphatase inhibitor lithium chloride , muscarinic receptor agonist oxotremorine and nicotinic receptor agonist nicotine produced hypothermia in a dose dependent manner .

目的观察氯化锂对沙土鼠全脑缺血时海马CA1区神经细胞凋亡及相关基因蛋白表达的影响。

Objective To observe the effect of lithium chloride on neuronal apoptosis and expression of apoptosis-related gene in the CA1 region of the hippocampus after global ischemia in gerbils .

建立氯化锂-匹罗卡品大鼠SE模型。

The lithium-pilocarpine induced SE were used as the SE model .

氯化锂抑制K562白血病细胞增殖及诱导凋亡机理的研究

Mechanism of Lithium Chloride-Induced Proliferation Inhibition and Apoptosis of K562 Leukemic Cells

结论：水合氯醛是氯化锂鄄匹罗卡品所致SC模型的有效止惊剂；

Conclusions : SC induced by lithium and pilocarpine is responsive to chloral hydrate .

建立氯化锂-匹罗卡品SE模型，设立生理盐水对照组。

The Lithium chloride-pilocarpine SE model was established , and the control group were injected with Sodium Chloride .

方法给SD大鼠先后腹腔注射氯化锂、匹罗卡品，制成癫疒间动物模型；

Methods Models of epilepsy of SD rats were established by intraperitoneal injection with lithium chloride and pilocarpine .

ICP-AES法同时测定氯化锂和氢氧化锂中七种杂质元素

Simultaneous Determination of Seven Impurity Elements in Lithium Chloride and Lithium Hydroxide by ICP AES

方法以氯化锂、匹罗卡品对雄性成年SD大鼠先后腹腔注射，制成癫痫持续状态动物模型；

Methods Established epilepsy continuing status model of male adult SD rats by intraperitoneal injection with chloride lithium and pilocarpine .

结论预先给予姜黄素能抑制氯化锂-匹罗卡品诱导的SE，但无明显神经元保护作用，其机制和潜在的临床应用价值值得深入研究。

Conclusion Pretreatment of curcumin can prevent the SE induced by lithium chloride-pilocarpine and the pretreatment can not protect the neuron .

氯化锂对HL-60细胞增殖和分化与c-myc表达的影响

Effects of Lithium Chloride on the Proliferation and Differentiation and c-myc Proto-Oncogene Expression in HL-60 Cells

阿托品、氯化锂和nimodipine均可拮抗Ach对神经细胞的毒性作用。

Atropine , lithium chloride and nimodipine decreased the toxicity of Ach on neurons .

下调促凋亡基因p53蛋白及NFκB蛋白的表达可能是氯化锂产生脑保护作用的机制之一。

The down-regulation of expression of p53 protein or NF - κ B protein is one of the mechanisms of the neuroprotective effect by lithium chloride .

应用NBT还原试验发现氯化锂能诱导HL-60细胞分化。

It was discovered that there was induction of the differentiation of HL-60 cells by lithium .

结果1.氯化锂-匹罗卡品SE模型：12只SD大鼠，91.7%诱发出SE，死亡率为9.3%，总成功率为83.3%。

Lithium-pilocarpine SE model : 12 SD rats were induced into SE , one was dead , the overall successful rate was 83.3 % . 2 .

氯化锂抑制A549细胞增殖及其对Polo样激酶1转录活性的影响

Effect of Glycogen Synthase Kinase 3 Inhibitor Lithium on Proliferation and Correlation with Transcription of Polo-like Kinase 1 in A549 Cells

结论氯化锂预处理能显著减轻沙土鼠短暂全脑缺血后海马CA1区神经元凋亡；

Conclusion Lithium chloride can significantly suppress neuronal apoptosis after global ischemia in gerbils .

结果氯化锂染毒后体外培养人外周血淋巴细胞SCE率增加（P<001）；

Results ① The frequency of SCE in peripheral lymphocyte of human blood was increased ( P < 0 01 ) .

目的探讨氯化锂（lithiumchloride，LiCl）对人肺泡Ⅱ型上皮细胞来源的A549细胞在细胞增殖和细胞周期方面的影响。

Objective To investigate the effect of lithium chloride ( LiCl ) on cell proliferation and cell cycle progression in alveolar epithelial type ⅱ cell-derived A549 cells .

氯化锂腹腔注射后10～10h后给予匹罗卡品。

Pilocarpine was injected intraperitoneally ( ip ) after the 10 ~ 18 hours of the lithium chloride ip .

在苯酚法和氯化锂沉淀法的基础上，改进并提出了一种适合小麦叶片总RNA的快速提取方法，消除了蛋白质、DNA、多糖、多酚等污染。

A method of quickly extracting total RNA from wheat leaves were put forward on the basis of phenol extraction method and lithium chloride precipitation extraction method , and it could avoid protein , DNA , polysaccharide and polyphenol contaminations .

与染铅组比较，铅+氯化锂（3、30、300和3000mg/kg）组皮层NOS阳性细胞数均显著性增多（P<0.05）。

The number of NOS positive neurons of lead + LiCl ( 3,30 and ( 3 000 mg / kg )) groups were higher than lead group ( P < 0.05 ) .

结论：氯化锂在大鼠脊髓损伤后能促进内源性神经干细胞的增殖，其机制与Wnt信号通路有关。

Conclusions : Lithium could activate the endogenous stem cells after SCI , the mechanism may be relate with the Wnt signal pathway . Conclusion : 1 .

基于以氯化锂水溶液为除湿剂的除湿冷却系统（DECS），对其溶液的再生过程进行实验研究。

Regeneration performance of liquid desiccant was studied experimentally for the LiCl-H2O desiccant cooling system ( DECS ) .

方法用氯化锂-匹罗卡品腹腔注射法制备小鼠癫痫模型，将成模后的大鼠随机分为生理盐水对照组、NGF组、致痫组。

Methods First we establish the lithium-pilocarpine induced rat model of acute epilepsy . All of the seizure-induced rats will divide into the normal saline control group , NGF group and seizure-induced group randomly .

目的探讨在氯化锂-匹罗卡品诱导大鼠急性痫性发作模型中N-myc表达及其意义。

Objective To explore the N-myc expression and its significance in rat model of acute seizures induced by lithium-pilocarpine .

10十一碳烯氧肟酸（UHA）氯化锂体系中，在示坡极谱仪上，钛有灵敏的阴极波，峰电位-1.66V（vs·SCE.）

In 10 undecene hydroxamic acid lithium chloride system , with oscillopolarography , titanium produced a sensitive cathode wave . The peak potential is-1.66V ( vs. SCE ) .

以金霉素产生菌H-502作为出发菌株，采用紫外线加氯化锂作为复合诱变剂，通过生物技术手段获得了F-303变株。

The Aureomycin producing strain F-303 was derived from H-502 by Using UV and mutagen lithium chloride .

目的：观察氯化锂对短暂局灶性脑缺血再灌注大鼠海马CA1区神经元凋亡及GSK-3β的影响。

Objective : To observe the effect of lithium chloride on neuronal apoptosis and GSK-3 β of hippocampal CA 1 area after transient focal cerebral ischemia / reperfusion in rats .

因此种物貭在水中的溶解度很低，故采用80%乙醇中的0.5M氯化锂溶液（加有0.01%明胶）为底液。

Owing to the low solubility of psoralen in water , a 0.5M solution of lithium chloride in 80 % ethanol containing 0.01 % gelatin was used for the base electrolyte .