fyn

These results suggest that Fyn may participate in the process of the enhanced learning and memory by chronic multiple stress .

提示Fyn参与了慢性复合性应激增强大鼠学习记忆能力的过程。

On the basis of these results it may be concluded that Fyn deficiency impairs associative learning .

根据以上实验结果可以认为，Fyn-激酶缺损可导致小鼠的联合型学习能力下降。

However , no deficit of 2 week memory retention of homozygous Fyn kinase deficient mice was found .

然而，纯合子Fyn-激酶缺损小鼠的两周记忆保持与其他两组相比没有显著性差别。

Proliferation and expression of hippocampal cells and change of postsynaptic density Fyn in the hippocampus of the rat whose learning and memory was enhanced by chronic multiple stress

慢性复合应激性学习记忆增强大鼠海马神经细胞增殖和突触后酪氨酸激酶表达的变化

The lack of Fyn in testis can lead to delayed formatio n of seminiferous tubule and degeneration of cells .

研究证明：Fyn蛋白存在于支持细胞的细胞骨架，Fyn缺损可导致生精小管形成延缓，并出现细胞变性。

Proto-oncogene fyn protein tyrosine kinase ( PTK ) activity and its tyrosine phosphorylation were explored in granulocyte-macrophage colony-stimulating factor ( GM-CSF ) receptor inducing growth signal transduction in murine macrophages .

本实验研究了小鼠巨噬细胞中原癌基因fyn酪氨酸蛋白激酶（PTK）的活性和酪氨酸磷酸化过程在粒-巨噬细胞集落刺激因子（GM-CSF）受体诱导的信号转导中的作用。