RTECS

  • 网络化学物质毒性数据库;化学物质毒性效应登录;登记号;有毒化学物质登录;化学物质毒性作用登记号
RTECSRTECS
  1. METHODS : The culture of primary RTECs was performed by renal tubular segment sticking mass method .

    方法:以肾小管节段贴块法进行原代RTECs的培养。

  2. On-line retrieval of RTECS database on vax / vms

    化学物质毒性效应登录数据库在VAX/VMS上联机检索的实现

  3. AIM : To detect the expressions of the IL-18R α mRNA and IL-18R β mRNA in primary rat renal tubular epithelial cells ( RTECs ) .

    目的:检测培养的大鼠原代肾小管上皮细胞(renaltubularepithelialcells,RTECs)是否表达IL-18Rα和IL-18RβmRNA。

  4. RESULTS : The result of immunocytochemical staining proved that the cultured cells were RTECs , IL-18R α mRNA and IL-18R β mRNA were detected in RTECs .

    结果:免疫细胞化学染色法证实,原代培养的细胞均为RTECs,并表达IL-18RαmRNA和IL18RβmRNA。

  5. The cellular type was identified by immunocytochemical staining . The IL-18R α mRNA and IL-18R β ( mRNA ) expressions in RTECs were detected by RT-PCR .

    用免疫细胞化学染色法鉴定细胞的类型,用RT-PCR检测RTECs中IL-18RαmRNA及IL-18RβmRNA的表达。

  6. But , being one of plant growth regulators , gibberellic acid is a toxic chemical farming drug . According to Sigma , gibberellic acid may cause respiratory and liver cancer , belonging to suspected cancer-causing agents in RTECS standard .

    但是,赤霉酸作为植物生长调节剂属有毒化学农药品,根据Sigma公司毒性资料显示,赤霉酸可能会引起呼吸系统和肝脏发生肿瘤,属于符合RTECS标准的可疑致癌试剂。

  7. In order to know whether or not the renal tubular epithelial cells ( RTECs ) are damaged during the early phase of diabetes mellitus ( DN ), and to understand the pathogenic mechanism of micro protein urine , we designed this experiment .

    为了解糖尿病早期肾小管上皮细胞(renaltubularepithlialcells,简称RTECs)是否有损害,探讨微蛋白尿发生的机制。

  8. CQ was categorized as one of the suspected endocrine disruptors , according to Registry of Toxic Effects of Chemical Substances ( RTECS ), a chemical toxicity database , released by National Institute for Occupational Safety and Health ( NIOSH ) .

    美国国家职业安全和健康研究所发布的化学物质毒性数据库已将矮壮素列为疑似内分泌干扰物质。

  9. Objectives To study the mechanism of cantharidin in protecting F-actin microfilaments from disruption by hypoxic damage by observing the effects of cantharidin on intracellular ATP metabolism in G1 / S-phase renal tubule epithelial cells ( RTECs ) .

    目的通过斑蝥素对G1/S期肾小管上皮细胞(RTECs)低氧损伤后细胞ATP代谢影响的研究,探讨斑蝥素稳定肌动蛋白微丝骨架系统的机制。

  10. Conclusions MCs may contribute to the accumulation of extracellular matrix in AIN and GIN through tryptase secretion , which can activate the PAR-2 in the RTECs or MCs itself , leading to the increase of TGF - β 1 expression and Col I synthesis .

    结论MCs可能参与AIN、CIN细胞外基质蓄积,其机制可能是通过活化PAR-2,增加TGF-β1表达,促进colⅠ合成。