超极化

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  • hyperpolarization
超极化超极化
  1. 高钾则可取消Ach的内皮依赖性超极化。

    The high K + solution could inhibit the Ach-induced hyperpolarization .

  2. 结果显示三种配体均导致细胞膜电位超极化,胞浆pH降低,溶酶体pH升高,三个生理参量趋于稳定时间稍有不同。

    The results showed all the three kinds of ligands caused macrophage membrane potential hyperpolarization , cytosolic acidification and lysosomal alkalization , but reached their stabilized conditions at different times .

  3. C(36)富勒烯氮杂环衍生物的分子第一超极化率理论研究

    Theoretical Study on the Molecular First Hyperpolarizability of the C_ ( 36 ) Fullerene Derivatives With Nitrogen Heterocycle

  4. 采用荧光定量RT-PCR法检测超极化激活环核苷酸门控阳离子通道

    Qualification of hyperpolarization-activated cyclic nucleotide-gated cation channel mRNA using the fluorogenic quantitative RT-PCR method

  5. 硼氮掺杂富勒烯C(34)BN的分子第一超极化率理论研究

    Theoretical Study on the Molecular First Hyperpolarizability of B and N-Doped Fullerene C_ ( 34 ) BN

  6. 脊神经结扎慢性痛大鼠背根神经节细胞超极化激活电流(Ih)的变化

    Alteration of hyperpolarization-activated current ( I _h ) in dorsal root ganglion neurons in rats with neuropathic pain induced by spinal nerve ligation

  7. 结果:MP使平滑肌细胞显著超极化。

    RESULTS : MP hyperpolarized SMCs significantly .

  8. 不同金属离子交换的FAU型分子筛的超极化~(129)XeNMR研究

    Hyperpolarized ~ ( 129 ) Xe NMR Spectroscopy Investigation of Metal Cation-Exchanged FAU Zeolites

  9. 提示:Ach引起的家兔肺动脉内皮依赖性超极化可能由细胞膜上钾通道开放所致,而和Na/K泵无关。

    The results suggest that the potassium Channels not the Na / K pump might contribute to the Ach-induced hyperpolarization in rabbit pulmonary artery .

  10. 此外,P-物质还抑制去极化激活的慢失活的钾电流、低阈值钙电流和超极化激活的内向整流(H或Q)电流。

    In addition , substance P inhibited the slowly inactivated potassium current , low threshold ( T - ) calcium current and inward rectifier ( H - or Q - ) current .

  11. 除膜电位外,〔K+〕o是此型通道的另一个门控因素,且在膜电位超极化增高的情况下,其作用更加显著。

    In addition to the membrane potential , the [ K + ] o was another channel - gating factor and its effect was more significant especially under greater membrane hyperpolarization .

  12. AVP和OT均可使DRG神经元膜产生超极化,对初级感觉信息的传入具有一定的抑制作用,可能参与感龈息传入的调制。

    AVP and OT might be involved in modulation of sensory input information through inhibiting the DRG neurons .

  13. 结合有限场(FF)方法计算了各体系的极化率和二阶超极化率。

    The polarizability and and second-order hyperpolarizability were calculated by means of finite-field ( FF ) method .

  14. 超极化激活环核苷酸门控阳离子通道(HCN)研究进展

    Research on Hyperpolarization-activated Cyclic Nucleotide-gated Channels

  15. 实验结果表明PNA分子在不同溶液中的一阶超极化率随着溶孔极性的增强而增加。

    Experimental results show that the first hyperpolarizabilities of PNA increase with the increase of the solvent polarity .

  16. 成年豚鼠膀胱ICC上超极化激活环核苷酸门控通道(HCN)电生理的初步研究

    Initial Electrophysiological Study of Hyperpolarization-activated Cyclic Nucleotide-gated ( HCN ) Cation Channel on ICCs in the Adult Guinea Pig Bladder

  17. 目的观察心肺转流(CPB)中吡那地尔超极化停搏液与传统高钾停搏液对犬冠脉流量的影响。

    Objective To evaluate the effects of pinacidil hyperpolarized cardioplegia versus hyperkalemic cardioplegia on coronary blood flow during cardiopulmonary bypass ( CPB ) in dogs .

  18. 超极化停搏对离体兔心肌补体C3a表达的抑制作用

    Hyperpolarized cardioplegia reduces tissue complement protein expression in the rabbit isolated heart

  19. 结果表明,HCG对卵母细胞膜电位有去极化作用,酪氨酸可以使膜电位呈现超极化反应,并能抑制HCG的去极化作用。

    The results showed that HCG depolarised membrane potential of toad oocytes , while tyrosine directly caused membrane , hyperpolarization , and inhibited the depolarization induced by HCG .

  20. UW液保存冠状动脉4小时后,对内皮源性超极化因子?

    The exposure to UW in 4 hours impairs the endothelium-derived hyperpolarizing factor-related endothelial function of coronary artery .

  21. 目的观察离体兔心脏超极化停搏对心肌组织补体C3a蛋白质表达的影响情况。

    Objective To investigate whether hyperpolarized cardioplegia affects complement expression in the isolated heart .

  22. 用基组展开和解析求导的方法精确计算Be原子SCF水平的极化率和超极化率。

    In the present work the polarizability and the hyperpolarizability of Be atom are obtained at the SCF level with basis-set expansion and analytic derivation method .

  23. 细胞外高K~+浓度对HCN(1,2)通道电生理特性的影响超极化激活环核苷酸门控阳离子通道(HCN)研究进展

    Effect of high extracellular K ~ + concentration on electrophysiological properties of HCN ( 1,2 ) Research on Hyperpolarization-activated Cyclic Nucleotide-gated Channels

  24. ACh-敏感性电流的反转电位为(-66±8)mV,提示此电流主要由K+参与形成,其直接作用是使毛细胞超极化。

    The reversal potential of the ACh-sensitive current was ( - 66 ± 8 ) mV , which indicated that potassium ion was the main carrier of this current .

  25. 目的观察超极化停搏对体外循环(CPB)中缺血再灌注心肌细胞膜蛋白生物物理特性变化的影响。

    Objective To elucidate the effects of hyperpolarized arrest on the biophysical property of myocardial cell membrane proteins in ischemia and reperfusion ( IR ) injury during cardiopulmonary bypass ( CPB ) .

  26. 结论ACh增加了OHC电压依赖性外向整流钾通道的电导,使通道的激活电压向超极化方向移位。

    Conclusion ACh increased the conductance of outwardly rectifying K + channel in a voltage dependent manner and caused a hyperpolarizing shift of activation potential .

  27. 新近,一类被命名为超极化激活及环化核苷酸调控的阳离子(HCN)通道被克隆。

    Recently , a family of hyperpolarization-activated and cyclic nucleotide-gated cation ( HCN ) channels comprising at least four members ( HCN1-HCN4 ) has been cloned .

  28. 目的比较超极化停搏和去极化停搏对体外循环(CPB)中心肌细胞膜流动性变化的影响,评价超极化停搏液的心肌保护作用。

    Objective To observe the influences of depolarized arrest and hyperpolarized arrest on alternation of fluidity of myocardial cell membrane during cardiopulmonary bypass ( CPB ) and evaluate the protective effects on myocardium of hyperpolarized arrest .

  29. 明显抑制内向整流钾电流(IK1),尤其在超极化时;

    Suppressed the inward rectifier K + current ( I KI ) significantly , especially at hyperpolarization ;

  30. 在此基础上用TDHF方法计算了分子的线性极化率、一阶和二阶超极化率。

    The linear polarizabilities , the first-and second-order hyperpolarizabilities of these squaraines were calculated using TDHF method .